Bone morphogenetic protein receptor II is a novel mediator of endothelial nitric-oxide synthase activation. - Wikidata - wikimedia - TriplyDB

Bone morphogenetic protein receptor II is a novel mediator of endothelial nitric-oxide synthase activation.

Bone morphogenetic protein receptor II is a novel mediator of endothelial nitric-oxide synthase activation.

http://www.wikidata.org/entity/Q35311990

about

Post-translational regulation of endothelial nitric oxide synthase in vascular endothelium Functional changes in pulmonary arterial endothelial cells associated with BMPR2 mutations Human primary lung endothelial cells in culture Endothelial follistatin-like-1 regulates the postnatal development of the pulmonary vasculature by modulating BMP/Smad signaling.Meta-analysis of genetic programs between idiopathic pulmonary fibrosis and sarcoidosis Chronic hypoxia induces right heart failure in caveolin-1-/- mice.Vascular remodeling in pulmonary hypertension.Nitric oxide deficiency in pulmonary hypertension: Pathobiology and implications for therapy.BMP regulates regional gene expression in the dorsal otocyst through canonical and non-canonical intracellular pathways BMPR2-pSMAD1/5 signaling pathway regulates RUNX2 expression and impacts the progression of dedifferentiated chondrosarcoma.Genotype-phenotype effects of Bmpr2 mutations on disease severity in mouse models of pulmonary hypertension.Review on Hypothetical Implementing TGF-β Family Members in Glaucoma Therapy.TGFβ signaling and cardiovascular diseases.A process-based review of mouse models of pulmonary hypertension.Nitric oxide deficiency and endothelial dysfunction in pulmonary arterial hypertension.The role of BMPs in endothelial cell function and dysfunction.Regulators and effectors of bone morphogenetic protein signalling in the cardiovascular system.Lung Circulation.The eNOS signalosome and its link to endothelial dysfunction.BMPR2 gene delivery reduces mutation-related PAH and counteracts TGF-β-mediated pulmonary cell signalling.Redox Mechanisms Influencing cGMP Signaling in Pulmonary Vascular Physiology and Pathophysiology.Intrinsic BMP Antagonist Gremlin-1 as a Novel Circulating Marker in Pulmonary Arterial Hypertension.AMPK Phosphorylation of ACE2 in Endothelium Mitigates Pulmonary Hypertension.Consequences of BMPR2 Deficiency in the Pulmonary Vasculature and Beyond: Contributions to Pulmonary Arterial Hypertension

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P2860

Bone morphogenetic protein receptor II is a novel mediator of endothelial nitric-oxide synthase activation.

http://www.wikidata.org/entity/Q35311990

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Bone morphogenetic protein rec ...... ric-oxide synthase activation.

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Bone morphogenetic protein rec ...... ric-oxide synthase activation.

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type

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Bone morphogenetic protein rec ...... ric-oxide synthase activation.

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Bone morphogenetic protein rec ...... ric-oxide synthase activation.

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Bone morphogenetic protein rec ...... ric-oxide synthase activation.

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Bone morphogenetic protein rec ...... ric-oxide synthase activation.

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JBC.M111.274100

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Journal of Biological Chemistry

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Bone morphogenetic protein rec ...... ric-oxide synthase activation.

@en

P2093

Archana Gangopahyay

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Eileen M Bauer

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Jeffrey W Wertz

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Max Oran

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Philip M Bauer

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Serpil C Erzurum

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Suzy A Comhair

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P2860

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Dynamics and interaction of caveolin-1 isoforms with BMP-receptors

Endothelial nitric oxide synthase is regulated by tyrosine phosphorylation and interacts with caveolin-1

The BMP type II receptor is located in lipid rafts, including caveolae, of pulmonary endothelium in vivo and in vitro

Bone morphogenetic protein 2 induces pulmonary angiogenesis via Wnt-beta-catenin and Wnt-RhoA-Rac1 pathways.

Familial primary pulmonary hypertension (gene PPH1) is caused by mutations in the bone morphogenetic protein receptor-II gene.

An antiproliferative BMP-2/PPARgamma/apoE axis in human and murine SMCs and its role in pulmonary hypertension.

Sporadic primary pulmonary hypertension is associated with germline mutations of the gene encoding BMPR-II, a receptor member of the TGF-beta family.

Alterations of cellular bioenergetics in pulmonary artery endothelial cells

The Akt1-eNOS axis illustrates the specificity of kinase-substrate relationships in vivo.

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33134-33140

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scholarly article

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10.1074/JBC.M111.274100

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38

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