trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency. - Wikidata - wikimedia - TriplyDB

trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.

trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.

http://www.wikidata.org/entity/Q35363296

about

Epigenetic regulation of EBV persistence and oncogenesis.Arsenic trioxide inhibits EBV reactivation and promotes cell death in EBV-positive lymphoma cells.Cordycepin is a novel chemical suppressor of Epstein-Barr virus replication.Quercetin-induced apoptosis prevents EBV infection.Genipin as a novel chemical activator of EBV lytic cycle.Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases.Contributions of CTCF and DNA methyltransferases DNMT1 and DNMT3B to Epstein-Barr virus restricted latency.The Epstein Barr virus circRNAome

P2860

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P2860

trans-Repression of protein expression dependent on the Epstein-Barr virus promoter Wp during latency.

http://www.wikidata.org/entity/Q35363296

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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trans-Repression of protein ex ...... us promoter Wp during latency.

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P1433

Journal of Virology

P1476

trans-Repression of protein ex ...... us promoter Wp during latency.

@en

P2093

Carol A Dickerson

http://www.w3.org/2001/XMLSchema#string

Clare E Sample

http://www.w3.org/2001/XMLSchema#string

Jeffery T Sample

http://www.w3.org/2001/XMLSchema#string

Marie S Shaner

http://www.w3.org/2001/XMLSchema#string

P2860

An Epstein-Barr virus anti-apoptotic protein constitutively expressed in transformed cells and implicated in burkitt lymphomagenesis: the Wp/BHRF1 link

Epstein-Barr virus nuclear antigen 1 does not cause lymphoma in C57BL/6J mice

CTCF prevents the epigenetic drift of EBV latency promoter Qp

Nuclear location of an endogenously expressed antigen, EBNA1, restricts access to macroautophagy and the range of CD4 epitope display.

Epstein-Barr virus nuclear antigen 1 does not induce lymphoma in transgenic FVB mice.

Redefining the Epstein-Barr virus-encoded nuclear antigen EBNA-1 gene promoter and transcription initiation site in group I Burkitt lymphoma cell lines

Epstein-barr virus nuclear antigen 3C activates the latent membrane protein 1 promoter in the presence of Epstein-Barr virus nuclear antigen 2 through sequences encompassing an spi-1/Spi-B binding site.

The Epstein-Barr virus promoter initiating B-cell transformation is activated by RFX proteins and the B-cell-specific activator protein BSAP/Pax5

Repression of Epstein-Barr virus EBNA-1 gene transcription by pRb during restricted latency

Epstein-Barr virus nuclear antigen 2 (EBNA2) gene deletion is consistently linked with EBNA3A, -3B, and -3C expression in Burkitt's lymphoma cells and with increased resistance to apoptosis.

P304

11435-11447

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scholarly article

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10.1128/JVI.05158-11

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21

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85

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2011-08-24T00:00:00Z

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51593763

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21865378

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P921

Epstein–Barr virus

P932

3194970

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