Effects of nicotine administration in a mouse model of familial hypertrophic cardiomyopathy, α-tropomyosin D175N.
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Potential contribution of aromatase inhibition to the effects of nicotine and related compounds on the brainDesensitization of myofilaments to Ca2+ as a therapeutic target for hypertrophic cardiomyopathy with mutations in thin filament proteins.Peroxisome proliferator-activated receptor-α expression induces alterations in cardiac myofilaments in a pressure-overload model of hypertrophy.
P2860
Effects of nicotine administration in a mouse model of familial hypertrophic cardiomyopathy, α-tropomyosin D175N.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@ast
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@en
type
label
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@ast
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@en
prefLabel
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@ast
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@en
P2093
P2860
P1476
Effects of nicotine administra ...... myopathy, α-tropomyosin D175N.
@en
P2093
Beata M Wolska
Cibele T D Ribeiro
David F Wieczorek
Fernando A L Dias
Marco S L Alves
Robert D Gaffin
Rosalvo T H Fogaca
Shamim A K Chowdhury
P2860
P304
P356
10.1152/AJPHEART.00277.2010
P577
2011-07-08T00:00:00Z