Interleukin-10 downregulates protective immunity to Brucella abortus.
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Brucella abortus uses a stealthy strategy to avoid activation of the innate immune system during the onset of infection.Brucella control of dendritic cell maturation is dependent on the TIR-containing protein Btp1.Quantification of parasite load in clinical samples of leishmaniasis patients: IL-10 level correlates with parasite load in visceral leishmaniasisImpaired control of Brucella melitensis infection in Rag1-deficient miceTumour necrosis factor gene polymorphisms as severity markers in rheumatoid arthritisVaccination with Brucella abortus recombinant in vivo-induced antigens reduces bacterial load and promotes clearance in a mouse model for infectionHuman brucellosis is characterized by an intense Th1 profile associated with a defective monocyte function.Protection of mice against brucellosis by vaccination with Brucella melitensis WR201(16MDeltapurEK).Interleukin-10 has different effects on proliferation of Listeria monocytogenes in livers and spleens of mice.Mouse cytokine profiles associated with Brucella abortus RB51 vaccination or B. abortus 2308 infectionCourse of infection with the emergent pathogen Brucella microti in immunocompromised miceImmunization of mice with gamma-irradiated Brucella neotomae and its recombinant strains induces protection against virulent B. abortus, B. melitensis, and B. suis challengeComparative analysis of the early transcriptome of Brucella abortus--infected monocyte-derived macrophages from cattle naturally resistant or susceptible to brucellosisEvaluation of protection afforded by Brucella abortus and Brucella melitensis unmarked deletion mutants exhibiting different rates of clearance in BALB/c mice.CD4+ T cell-derived IL-10 promotes Brucella abortus persistence via modulation of macrophage function.Ex vivo innate immune cytokine signature of enhanced risk of relapsing brucellosisLack of endogenous IL-10 enhances production of proinflammatory cytokines and leads to Brucella abortus clearance in mice.Immune responses of elk to initial and booster vaccinations with Brucella abortus strain RB51 or 19.CC chemokine receptor 7 expression by effector/memory CD4+ T cells depends on antigen specificity and tissue localization during influenza A virus infection.A B lymphocyte mitogen is a Brucella abortus virulence factor required for persistent infection.Lack of a role for natural killer cells in early control of Brucella abortus 2308 infections in mice.Expression of cytokines and inducible nitric oxide synthase mRNA in the lungs of mice infected with Cryptococcus neoformans: effects of interleukin-12.Gamma interferon loaded onto albumin nanoparticles: in vitro and in vivo activities against Brucella abortus.Immunopathology and ehrlichial propagation are regulated by interferon-gamma and interleukin-10 in a murine model of human granulocytic ehrlichiosis.Improved immunogenicity of a vaccination regimen combining a DNA vaccine encoding Brucella melitensis outer membrane protein 31 (Omp31) and recombinant Omp31 boosting.Brucella lumazine synthase elicits a mixed Th1-Th2 immune response and reduces infection in mice challenged with Brucella abortus 544 independently of the adjuvant formulation usedWhat have we learned from brucellosis in the mouse model?Crucial role of gamma interferon-producing CD4+ Th1 cells but dispensable function of CD8+ T cell, B cell, Th2, and Th17 responses in the control of Brucella melitensis infection in mice.Inactivation of the type IV secretion system reduces the Th1 polarization of the immune response to Brucella abortus infection.Chronic Bacterial Pathogens: Mechanisms of Persistence.Host susceptibility to Brucella abortus infection is more pronounced in IFN-γ knockout than IL-12/β2-microglobulin double-deficient mice.Interleukin-12 in infectious diseases.Resolution-phase macrophages possess a unique inflammatory phenotype that is controlled by cAMP.Protective or deleterious role of scavenger receptors SR-A and CD36 on host resistance to Staphylococcus aureus depends on the site of infectionSubcellular fractions of Brucella ovis distinctively induce the production of interleukin-2, interleukin-4, and interferon-gamma in mice.Identification of Brucella abortus virulence proteins that modulate the host immune response.Activation of NF-kB-Mediated TNF-Induced Antimicrobial Immunity Is Required for the Efficient Brucella abortus Clearance in RAW 264.7 Cells.Interleukin 10 suppresses lysosome-mediated killing of Brucella abortus in cultured macrophages.Interferon-gamma is crucial for surviving a Brucella abortus infection in both resistant C57BL/6 and susceptible BALB/c mice.miR-181a-5p Regulates TNF-α and miR-21a-5p Influences Gualynate-Binding Protein 5 and IL-10 Expression in Macrophages Affecting Host Control of Brucella abortus Infection.
P2860
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P2860
Interleukin-10 downregulates protective immunity to Brucella abortus.
description
1995 nî lūn-bûn
@nan
1995 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մարտին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Interleukin-10 downregulates protective immunity to Brucella abortus.
@ast
Interleukin-10 downregulates protective immunity to Brucella abortus.
@en
type
label
Interleukin-10 downregulates protective immunity to Brucella abortus.
@ast
Interleukin-10 downregulates protective immunity to Brucella abortus.
@en
prefLabel
Interleukin-10 downregulates protective immunity to Brucella abortus.
@ast
Interleukin-10 downregulates protective immunity to Brucella abortus.
@en
P2860
P1476
Interleukin-10 downregulates protective immunity to Brucella abortus
@en
P2093
Baldwin CL
Fernandes DM
P2860
P304
P407
P577
1995-03-01T00:00:00Z