KIM-1-mediated phagocytosis reduces acute injury to the kidney. - Wikidata - wikimedia - TriplyDB

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

http://www.wikidata.org/entity/Q35409103

about

Immunogenic versus tolerogenic phagocytosis during anticancer therapy: mechanisms and clinical translation Mining kidney toxicogenomic data by using gene co-expression modules Pediatric acute kidney injury: prevalence, impact and management challenges.The Dynamics of Apoptotic Cell Clearance Engulfment signals and the phagocytic machinery for apoptotic cell clearance.KIM-1-/TIM-1-mediated phagocytosis links ATG5-/ULK1-dependent clearance of apoptotic cells to antigen presentation.Epigenetic regulation in acute kidney injury: new light in a dark area A potential adjuvant chemotherapeutics, 18β-glycyrrhetinic acid, inhibits renal tubular epithelial cells apoptosis via enhancing BMP-7 epigenetically through targeting HDAC2.Kidney injury molecule-1 (KIM-1) mediates renal epithelial cell repair via ERK MAPK signaling pathway Mammalian Target of Rapamycin Mediates Kidney Injury Molecule 1-Dependent Tubule Injury in a Surrogate Model.G protein α12 (Gα12) is a negative regulator of kidney injury molecule-1-mediated efferocytosis.Detection of Drug-Induced Acute Kidney Injury in Humans Using Urinary KIM-1, miR-21, -200c, and -423 ADAM17 substrate release in proximal tubule drives kidney fibrosis A proteolytic modification of AIM promotes its renal excretion.Alisol A 24-Acetate and Alisol B 23-Acetate Induced Autophagy Mediates Apoptosis and Nephrotoxicity in Human Renal Proximal Tubular Cells.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.Extracellular vesicles as immune mediators in response to kidney injury.New answers to old conundrums: what antibodies, exosomes and inflammasomes bring to the conversation. Canadian National Transplant Research Program international summit report.Kidney Injury Molecule-1 is Elevated in Nephropathy and Mediates Macrophage Activation via the Mapk Signalling Pathway.Downregulation of kidney protective factors by inflammation: role of transcription factors and epigenetic mechanisms.Reno-Cerebral Reflex Activates the Renin-Angiotensin System, Promoting Oxidative Stress and Renal Damage After Ischemia-Reperfusion Injury.Efferocytosis Signaling in the Regulation of Macrophage Inflammatory Responses.The Tubulointerstitial Pathophysiology of Progressive Kidney Disease.A kidney injury molecule-1 (Kim-1) gene reporter in a mouse artificial chromosome: the responsiveness to cisplatin toxicity in immortalized mouse kidney S3 cells.Relationship of proximal tubular injury to chronic kidney disease as assessed by urinary kidney injury molecule-1 in five cohort studies Apoptosis inhibitor of macrophage protein enhances intraluminal debris clearance and ameliorates acute kidney injury in mice.Acute kidney injury and chronic kidney disease: From the laboratory to the clinic Apoptosis inhibitor of macrophage ameliorates fungus-induced peritoneal injury model in mice Intracellular pH Regulates TRAIL-Induced Apoptosis and Necroptosis in Endothelial Cells.Early detection of acute kidney injury after pediatric cardiac surgery.KIM-1/TIM-1 in proximal tubular cell immune response.Expression of Kidney Injury Molecule-1 in Healthy and Diseased Feline Kidney Tissue.Urinary biomarkers of cell cycle arrest are delayed predictors of acute kidney injury after pediatric cardiopulmonary bypass.Cisplatin-induced renal inflammation is ameliorated by cilastatin nephroprotection.A novel approach to off-clamp partial nephrectomy demonstrates significant improvements in renal injury in an experimental porcine model.In vitro study of putative genomic biomarkers of nephrotoxicity through differential gene expression using gentamicin.Lymphocyte-specific deletion of IKK2 or NEMO mediates an increase in intrarenal Th17 cells and accelerates renal damage in an ischemia-reperfusion injury mouse model.Tctex-1, a novel interaction partner of Kidney Injury Molecule-1, is required for efferocytosis.[Autophagy in the kidney].Acute Kidney Injury and Progression of Diabetic Kidney Disease.

P2860

Q28386858-848C1497-4BE6-4B75-AF58-F974DB7BE40B Q28822270-0BACC560-D857-4D7A-8376-5D2071698164 Q30234692-D3597B0B-02FB-4680-8937-F6573E4BD8EB Q30276273-D293D83D-D7F9-41B2-8B3D-B34D9B21136B Q33754060-FF5EE9F4-93E2-4420-A1A5-89FFCFA6166B Q36148385-9CB31FA5-D3DC-465B-B0E2-8086D3F5AA89 Q36525433-1EC4972C-FD24-4D72-AFF5-8045D871D6A4 Q36870776-ED932037-050D-41C5-A6EC-08A868B7A161 Q36942409-AD97C217-6209-4B54-B401-C0071A8900D7 Q37052029-69BC0E90-3CD0-4129-8434-82979280B63D Q37148892-AC4656DA-8AD3-4B5A-8000-A63C49E1BAFE Q37228202-A49FF946-D7F6-416B-B06D-335F85E93543 Q37262336-2CAE2E7B-92C6-4F9B-B68C-9DA426D4B2E3 Q37483757-8CC9C746-A3DA-4CB3-8D55-CC57E2682E16 Q37731040-36CEEF8F-43B2-4A48-A89D-879D1E38944E Q38341214-547BE76B-D085-49D1-9FCC-6F7F912BC89A Q38601736-3070C86D-B8D8-4F7E-8721-6A915D7BB4DF Q38668025-FDDA5CBB-367F-47D0-ACCA-BE2F9B87E6F1 Q38715872-20D71C41-73C0-45DF-8E1B-1C2226500EBE Q38985548-63A426FE-DDC6-45F5-8AC0-853B7207BC46 Q39057673-8A0A3F7E-AFE5-4E94-B1D6-76C4BA83290B Q39123271-CED593A7-D8E1-449B-9A78-2BED59991764 Q39173994-85E6BA07-6912-4C2F-8DBB-2AA84FD99FF1 Q40505688-A7BC2952-85A9-41E3-BBFE-1C0E8E78C544 Q40715066-86CD41C7-4CAE-4E1C-98FF-345746180209 Q41003256-47227382-C6E8-425C-A178-09C78535309F Q41102152-B91714FD-8AC6-4B23-AACC-7FD8388E8AEC Q41125520-C7E59059-6A68-45F4-90AC-ABA4487308B2 Q41528739-66E56460-9E51-4426-A398-ED1CBA930DDF Q41855054-3DFB4AE5-94F9-4288-89C7-20740384289C Q42923626-6946807E-30FC-4268-B980-19359ED4A1EB Q47911737-41AEB4E9-E70A-4463-BF7B-60489F46AEE6 Q48014477-2BEF3879-32A4-4D8A-BB8D-9A44915BE1D9 Q48807960-95AAFBCF-FABC-490C-9192-09DF447A1BD5 Q49561257-956549A1-28A0-4A58-9D00-AF3DFC119AFD Q50181110-1FF8C523-9A93-4A8C-B035-198E41356E53 Q51539455-1B328E8F-CC62-4EE7-95D0-88101F748BEA Q52564082-70330D5A-1DFD-474D-854A-37C235101C94 Q52693141-D2E0E552-F2A3-4905-9344-CDC1F8E98406 Q52724019-89F5CAFA-55C6-4AF4-85A5-195600F2609A

P2860

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

http://www.wikidata.org/entity/Q35409103

description

2015 nî lūn-bûn

@nan

2015 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի մարտին հրատարակված գիտական հոդված

@hy

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

name

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@ast

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@en

type

label

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@ast

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@en

prefLabel

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@ast

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@en

P1433

Q35409103-8D23DF16-19CD-43F9-A306-74B119BB4DDC

P1476

Q35409103-62DE755F-8225-4B62-9DFB-3B1F38D44581

P2093

Q35409103-1A58D96B-11A7-46ED-9B1F-5F5F1DE26E27

Q35409103-2270FF90-1574-4A70-AA40-818F4C9D481A

Q35409103-2A084BCB-F9AB-4175-A486-8F8F3A405337

Q35409103-57179280-DC0B-459A-AD1A-6155ED01B279

Q35409103-82293168-1674-45F0-8528-73FF22B92EA1

Q35409103-B1DD8582-7A8E-435B-93CC-903661E4DEEA

Q35409103-DD9CE4C7-3331-40BC-A430-1870113F0470

Q35409103-E58A0BDA-B80D-49F4-8FEE-16277E32D423

Q35409103-EB3838E8-F9E7-4F7A-8D22-6F6B73496B2C

P2860

Q35409103-0DB16BA9-ABEA-46D5-A5C4-32B3665B49CF

Q35409103-0FE06ECA-EE7B-48F9-99DF-0E0C62EA2465

Q35409103-1209CA30-A5E6-4D34-A48B-23628FC88198

Q35409103-181413C7-07ED-4627-A2FC-34D32373AE9D

Q35409103-18E8C9DB-A630-4A20-899D-D536BCEF23D2

Q35409103-19F2D4A2-E012-4E79-8371-12D1610CF4D7

Q35409103-1FDE5922-8A8D-4697-BAE9-EDBDE3346A84

Q35409103-21F4F396-93D9-4735-AF6E-8D9B3845065E

Q35409103-3AF99370-F68B-4E2B-AB4B-4DBE93E68CF6

Q35409103-3B4DDFFA-A4A1-4954-971F-17A2447F8844

P304

Q35409103-1A5A175F-1ED7-49E6-988A-448BB1448A07

P31

Q35409103-68A7DCA2-105B-4A7A-B26E-56FEDD419851

P356

Q35409103-0E72DF7A-E406-417F-B8FF-EB9D1418BF8A

P407

Q35409103-94F8DB73-7198-4B54-9318-0D0878E53530

P433

Q35409103-106FEA13-776B-4B09-8806-7FB277B31FA4

P478

Q35409103-E9D773AC-2A91-4301-BDFB-487D18025C37

P577

Q35409103-871A28BF-9B1E-40B7-9826-95A6249133EB

P698

Q35409103-70E8D1DE-A392-4769-8AA1-63B46DA55E52

P921

Q35409103-0C0C35DD-5624-4575-89A3-5EDD83BE6C90

P932

Q35409103-9B6862AF-0816-42F5-9B01-30477F789DF6

P356

P1433

Journal of Clinical Investigation

P1476

KIM-1-mediated phagocytosis reduces acute injury to the kidney.

@en

P2093

Craig R Brooks

http://www.w3.org/2001/XMLSchema#string

Joseph V Bonventre

http://www.w3.org/2001/XMLSchema#string

Li Yang

http://www.w3.org/2001/XMLSchema#string

Li-Li Hsiao

http://www.w3.org/2001/XMLSchema#string

Melissa Y Yeung

http://www.w3.org/2001/XMLSchema#string

Sheng Xiao

http://www.w3.org/2001/XMLSchema#string

Takaharu Ichimura

http://www.w3.org/2001/XMLSchema#string

Venkata Sabbisetti

http://www.w3.org/2001/XMLSchema#string

Vijay Kuchroo

http://www.w3.org/2001/XMLSchema#string

P2860

Direct binding of p85 to sst2 somatostatin receptor reveals a novel mechanism for inhibiting PI3K pathway

TRIP-Br: a novel family of PHD zinc finger- and bromodomain-interacting proteins that regulate the transcriptional activity of E2F-1/DP-1

Toll-like receptors in the induction of the innate immune response

CSF-1 signaling mediates recovery from acute kidney injury.

Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells

T cell Ig and mucin domain-1-mediated T cell activation requires recruitment and activation of phosphoinositide 3-kinase

NKT cell activation mediates neutrophil IFN-gamma production and renal ischemia-reperfusion injury.

Toll-like receptor-4 coordinates the innate immune response of the kidney to renal ischemia/reperfusion injury.

B cells limit repair after ischemic acute kidney injury.

Scavenger receptors and modified lipoproteins: fatal attractions?

P304

1620-1636

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1172/JCI75417

http://www.w3.org/2001/XMLSchema#string

P407

P433

4

http://www.w3.org/2001/XMLSchema#string

P478

125

http://www.w3.org/2001/XMLSchema#string

P577

2015-03-09T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

25751064

http://www.w3.org/2001/XMLSchema#string

P921

P932

4396492

http://www.w3.org/2001/XMLSchema#string