Toxoplasma gondii effectors are master regulators of the inflammatory response. - Wikidata - wikimedia - TriplyDB

Toxoplasma gondii effectors are master regulators of the inflammatory response.

Toxoplasma gondii effectors are master regulators of the inflammatory response.

http://www.wikidata.org/entity/Q35434828

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Recombinant vaccines against T. gondii: comparison between homologous and heterologous vaccination protocols using two viral vectors expressing SAG1 A nuclear factor of high mobility group box protein in Toxoplasma gondii Structure of the Toxoplasma gondii ROP18 Kinase Domain Reveals a Second Ligand Binding Pocket Required for Acute Virulence Secreted effectors in Toxoplasma gondii and related species: determinants of host range and pathogenesis?Neospora caninum Activates p38 MAPK as an Evasion Mechanism against Innate Immunity Is Toxoplasma gondii type related to clinical outcome in human congenital infection? Systematic and critical review.Cell death of gamma interferon-stimulated human fibroblasts upon Toxoplasma gondii infection induces early parasite egress and limits parasite replication East Coast Fever Caused by Theileria parva Is Characterized by Macrophage Activation Associated with Vasculitis and Respiratory Failure.Toxoplasma gondii Infection Induces High Mobility Group Box 1 Released from Mouse Macrophages.Metabolic reconstruction identifies strain-specific regulation of virulence in Toxoplasma gondii.Neurophysiological Changes Induced by Chronic Toxoplasma gondii Infection Selective and strain-specific NFAT4 activation by the Toxoplasma gondii polymorphic dense granule protein GRA6.The rhoptry proteins ROP18 and ROP5 mediate Toxoplasma gondii evasion of the murine, but not the human, interferon-gamma response.Toxoplasma gondii clonal strains all inhibit STAT1 transcriptional activity but polymorphic effectors differentially modulate IFNγ induced gene expression and STAT1 phosphorylation Evaluation of immuno-efficacy of a novel DNA vaccine encoding Toxoplasma gondii rhoptry protein 38 (TgROP38) against chronic toxoplasmosis in a murine model Genetic basis for phenotypic differences between different Toxoplasma gondii type I strains The genome of Eimeria falciformis--reduction and specialization in a single host apicomplexan parasite.Severe South American ocular toxoplasmosis is associated with decreased Ifn-γ/Il-17a and increased Il-6/Il-13 intraocular levels.Transcriptional analysis of murine macrophages infected with different Toxoplasma strains identifies novel regulation of host signaling pathways Beta-catenin signaling drives differentiation and proinflammatory function of IRF8-dependent dendritic cells Inflammasome sensor NLRP1 controls rat macrophage susceptibility to Toxoplasma gondii.Border maneuvers: deployment of mucosal immune defenses against Toxoplasma gondii.Calomys callosus chronically infected by Toxoplasma gondii clonal type II strain and reinfected by Brazilian strains is not able to prevent vertical transmission.Strain hypothesis of Toxoplasma gondii infection on the outcome of human diseases.Genome-wide expression patterns of calcium-dependent protein kinases in Toxoplasma gondii.Transcriptional and Linkage Analyses Identify Loci that Mediate the Differential Macrophage Response to Inflammatory Stimuli and Infection.Variation detection based on next-generation sequencing of type Chinese 1 strains of Toxoplasma gondii with different virulence from China.Genetic Diversity of Toxoplasma gondii Strains from Different Hosts and Geographical Regions by Sequence Analysis of GRA20 Gene Toxoplasma gondii GRA7-Induced TRAF6 Activation Contributes to Host Protective Immunity Association of a NOD2 gene polymorphism and T-helper 17 cells with presumed ocular toxoplasmosis.Chronic Toxoplasmosis Modulates the Induction of Contact Hypersensitivity by TNCB in Mouse Model.Behavioral Abnormalities in a Mouse Model of Chronic Toxoplasmosis Are Associated with MAG1 Antibody Levels and Cyst Burden.Toxoplasma gondii rhoptry 16 kinase promotes host resistance to oral infection and intestinal inflammation only in the context of the dense granule protein GRA15.The tandemly repeated NTPase (NTPDase) from Neospora caninum is a canonical dense granule protein whose RNA expression, protein secretion and phosphorylation coincides with the tachyzoite egress.Secretion of Rhoptry and Dense Granule Effector Proteins by Nonreplicating Toxoplasma gondii Uracil Auxotrophs Controls the Development of Antitumor Immunity.A Toxoplasma dense granule protein, GRA24, modulates the early immune response to infection by promoting a direct and sustained host p38 MAPK activation.Toxoplasma gondii TgIST co-opts host chromatin repressors dampening STAT1-dependent gene regulation and IFN-γ-mediated host defenses.Toxoplasma gondii serostatus is not associated with impaired long-term survival after heart transplantation.Virulent and avirulent strains of Toxoplasma gondii which differ in their glycosylphosphatidylinositol content induce similar biological functions in macrophages.Functional Characterization of Rhoptry Kinome in the Virulent Toxoplasma gondii RH Strain.

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

http://www.wikidata.org/entity/Q35434828

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2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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J.PT.2011.08.001

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Trends in Parasitology

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Toxoplasma gondii effectors are master regulators of the inflammatory response.

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Jeroen P J Saeij

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Kirk D C Jensen

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Mariane B Melo

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P2860

Disruption of the Toxoplasma gondii parasitophorous vacuole by IFNgamma-inducible immunity-related GTPases (IRG proteins) triggers necrotic cell death

The inflammasomes

Structures of Toxoplasma gondii tachyzoites, bradyzoites, and sporozoites and biology and development of tissue cysts.

Phosphorylation of immunity-related GTPases by a Toxoplasma gondii-secreted kinase promotes macrophage survival and virulence

The activation mechanism of Irga6, an interferon-inducible GTPase contributing to mouse resistance against Toxoplasma gondii

Human IRGM regulates autophagy and cell-autonomous immunity functions through mitochondria

Inflammasomes: too big to miss

UNC93B1 mediates host resistance to infection with Toxoplasma gondii

A Conserved Non-canonical Motif in the Pseudoactive Site of the ROP5 Pseudokinase Domain Mediates Its Effect on Toxoplasma Virulence

Pattern recognition receptors and inflammation

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487-495

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scholarly article

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10.1016/J.PT.2011.08.001

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11

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27

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2011-09-03T00:00:00Z

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Toxoplasma gondii

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3200456

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