Upregulated expression of STIM2, TRPC6, and Orai2 contributes to the transition of pulmonary arterial smooth muscle cells from a contractile to proliferative phenotype.
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Molecular Mechanisms of Pulmonary Vascular Remodeling in Pulmonary Arterial HypertensionStore-operated calcium entry and diabetic complicationsObesity Induces Artery-Specific Alterations: Evaluation of Vascular Function and Inflammatory and Smooth Muscle Phenotypic Markers.The Role of Transient Receptor Potential Channel 6 Channels in the Pulmonary VasculatureOrai channel-mediated Ca2+ signals in vascular and airway smooth muscle.Pathogenic role of calcium-sensing receptors in the development and progression of pulmonary hypertension.PDGF induces SphK1 expression via Egr-1 to promote pulmonary artery smooth muscle cell proliferation.Enhanced store-operated Ca2+ influx and ORAI1 expression in ventricular fibroblasts from human failing heart.Molecular pathogenesis and current pathology of pulmonary hypertension.From contraction to gene expression: nanojunctions of the sarco/endoplasmic reticulum deliver site- and function-specific calcium signals.Nicotinamide Phosphoribosyltransferase Promotes Pulmonary Vascular Remodeling and Is a Therapeutic Target in Pulmonary Arterial Hypertension.Role of STIM2 in cell function and physiopathology.Transient Receptor Potential Channel 4 Encodes a Vascular Permeability Defect and High-Frequency Ca(2+) Transients in Severe Pulmonary Arterial Hypertension.The STIM-Orai Pathway: Regulation of STIM and Orai by Thiol Modifications.Translating Research into Improved Patient Care in Pulmonary Arterial Hypertension.STIM2 regulates both intracellular Ca2+ distribution and Ca2+ movement in skeletal myotubes.Hypoxia Selectively Upregulates Cation Channels and Increases Cytosolic [Ca2+] in Pulmonary, but not Coronary, Arterial Smooth Muscle Cells.Is p38 MAPK a Dark Force in Right Ventricular Hypertrophy and Failure in Pulmonary Arterial Hypertension?Cardiovascular and Hemostatic Disorders: Role of STIM and Orai Proteins in Vascular Disorders.Aspirin attenuates monocrotaline-induced pulmonary arterial hypertension in rats by suppressing the ERK/MAPK pathway.Reduced membrane cholesterol after chronic hypoxia limits Orai1-mediated pulmonary endothelial Ca2+ entry.STIM2 (Stromal Interaction Molecule 2)-Mediated Increase in Resting Cytosolic Free Ca2+ Concentration Stimulates PASMC Proliferation in Pulmonary Arterial Hypertension.The Complex Role of Store Operated Calcium Entry Pathways and Related Proteins in the Function of Cardiac, Skeletal and Vascular Smooth Muscle Cells.Revisiting the Role of TRP, Orai, and ASIC Channels in the Pulmonary Arterial Response to Hypoxia.Bone Marrow-Derived Endothelial Progenitor Cells Contribute to Monocrotaline-Induced Pulmonary Arterial Hypertension in Rats via Inhibition of Store-Operated Ca Channels
P2860
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P2860
Upregulated expression of STIM2, TRPC6, and Orai2 contributes to the transition of pulmonary arterial smooth muscle cells from a contractile to proliferative phenotype.
description
2015 nî lūn-bûn
@nan
2015 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Upregulated expression of STIM ...... le to proliferative phenotype.
@ast
Upregulated expression of STIM ...... le to proliferative phenotype.
@en
type
label
Upregulated expression of STIM ...... le to proliferative phenotype.
@ast
Upregulated expression of STIM ...... le to proliferative phenotype.
@en
prefLabel
Upregulated expression of STIM ...... le to proliferative phenotype.
@ast
Upregulated expression of STIM ...... le to proliferative phenotype.
@en
P2093
P2860
P1476
Upregulated expression of STIM ...... le to proliferative phenotype.
@en
P2093
Ayako Makino
Dolly Mehta
Haiyang Tang
Jason X-J Yuan
Ruby A Fernandez
Shanshan Song
P2860
P304
P356
10.1152/AJPCELL.00202.2014
P577
2015-02-11T00:00:00Z