Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
about
Dopaminergic Neurons Exhibit an Age-Dependent Decline in Electrophysiological Parameters in the MitoPark Mouse Model of Parkinson's Disease.Reward processing in autism: a thematic series.Preclinical research in Rett syndrome: setting the foundation for translational success.The neural circuit basis of Rett syndromeNeuroanatomy in mouse models of Rett syndrome is related to the severity of Mecp2 mutation and behavioral phenotypes.Desensitized D2 autoreceptors are resistant to trafficking.Is X-linked methyl-CpG binding protein 2 a new target for the treatment of Parkinson's diseasePrenatal maternal immune activation causes epigenetic differences in adolescent mouse brain.Identification of neurodegenerative factors using translatome-regulatory network analysis.Distinct regulation of dopamine D2S and D2L autoreceptor signaling by calcium.Applying the ethoexperimental approach to neurodevelopmental syndrome research reveals exaggerated defensive behavior in Mecp2 mutant mice.Stem Cell-Induced Biobridges as Possible Tools to Aid Neuroreconstruction after CNS Injury.Spontaneous inhibitory synaptic currents mediated by a G protein-coupled receptorThe first mecp2-null zebrafish model shows altered motor behaviorsHyperexcitability of Mesencephalic Trigeminal Neurons and Reorganization of Ion Channel Expression in a Rett Syndrome Model.Dopaminergic dysfunction in neurodevelopmental disorders: recent advances and synergistic technologies to aid basic research.Engrailed Homeoprotein Protects Mesencephalic Dopaminergic Neurons from Oxidative StressModification of spectral features by nonhuman primatesMeCP2-mediated alterations of striatal features accompany psychomotor deficits in a mouse model of Rett syndrome.Characteristics of allelic gene expression in human brain cells from single-cell RNA-seq data analysis.Chronic exposure to ethanol of male mice before mating produces attention deficit hyperactivity disorder-like phenotype along with epigenetic dysregulation of dopamine transporter expression in mouse offspring.Selective preservation of MeCP2 in catecholaminergic cells is sufficient to improve the behavioral phenotype of male and female Mecp2-deficient mice.En route to disentangle the impact and neurobiological substrates of early vocalizations: learning from Rett syndrome.Rescue of hyperexcitability in hippocampal CA1 neurons from Mecp2 (-/y) mouse through surface potential neutralization.Reward circuitry dysfunction in psychiatric and neurodevelopmental disorders and genetic syndromes: animal models and clinical findings
P2860
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P2860
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
@ast
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
@en
type
label
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
@ast
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
@en
prefLabel
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
@ast
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway.
@en
P2093
P2860
P1476
Loss of Mecp2 in substantia nigra dopamine neurons compromises the nigrostriatal pathway
@en
P2093
Christopher P Ford
John T Williams
Kim A Neve
P2860
P304
12629-12637
P356
10.1523/JNEUROSCI.0684-11.2011
P407
P577
2011-08-01T00:00:00Z