CRAC channel inhibition produces greater anti-inflammatory effects than glucocorticoids in CD8 cells from COPD patients - Wikidata - wikimedia - TriplyDB

CRAC channel inhibition produces greater anti-inflammatory effects than glucocorticoids in CD8 cells from COPD patients

CRAC channel inhibition produces greater anti-inflammatory effects than glucocorticoids in CD8 cells from COPD patients

http://www.wikidata.org/entity/Q35457347

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Silver Nanoparticle-Directed Mast Cell Degranulation Is Mediated through Calcium and PI3K Signaling Independent of the High Affinity IgE Receptor STIM1 controls T cell-mediated immune regulation and inflammation in chronic infection.NFATc3 and VIP in Idiopathic Pulmonary Fibrosis and Chronic Obstructive Pulmonary Disease Additive anti-inflammatory effects of corticosteroids and phosphodiesterase-4 inhibitors in COPD CD8 cells Ion channels in innate and adaptive immunity Tailoring of corticosteroids in COPD management.Ca2+-Dependent Regulation of NFATc1 via KCa3.1 in Inflammatory Osteoclastogenesis.

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P2860

CRAC channel inhibition produces greater anti-inflammatory effects than glucocorticoids in CD8 cells from COPD patients

http://www.wikidata.org/entity/Q35457347

description

2014 nî lūn-bûn

@nan

2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

CRAC channel inhibition produc ...... n CD8 cells from COPD patients

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CRAC channel inhibition produc ...... n CD8 cells from COPD patients

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type

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CRAC channel inhibition produc ...... n CD8 cells from COPD patients

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CRAC channel inhibition produc ...... n CD8 cells from COPD patients

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CRAC channel inhibition produc ...... n CD8 cells from COPD patients

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CRAC channel inhibition produc ...... n CD8 cells from COPD patients

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Clinical Science

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CRAC channel inhibition produc ...... n CD8 cells from COPD patients

@en

P2093

Dave Singh

http://www.w3.org/2001/XMLSchema#string

David House

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Manminder Kaur

http://www.w3.org/2001/XMLSchema#string

Seamus Grundy

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Simon Hall

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Sophie Reynolds

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P2860

Inhaled corticosteroids for stable chronic obstructive pulmonary disease

Physical association and functional antagonism between the p65 subunit of transcription factor NF-kappa B and the glucocorticoid receptor

Transcription factors of the NFAT family: regulation and function

NFAT proteins: key regulators of T-cell development and function

The nature of small-airway obstruction in chronic obstructive pulmonary disease

Cytotoxic potential of lung CD8(+) T cells increases with chronic obstructive pulmonary disease severity and with in vitro stimulation by IL-18 or IL-15.

Antielastin autoimmunity in tobacco smoking-induced emphysema.

The effects of inhaled corticosteroids in chronic obstructive pulmonary disease: a systematic review of randomized placebo-controlled trials.

The interplay between the glucocorticoid receptor and nuclear factor-kappaB or activator protein-1: molecular mechanisms for gene repression.

T lymphocyte insensitivity to corticosteroids in chronic obstructive pulmonary disease

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223-232

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scholarly article

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10.1042/CS20130152

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3

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126

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23905758

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4401013

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