Glial-conditional deletion of aquaporin-4 (Aqp4) reduces blood-brain water uptake and confers barrier function on perivascular astrocyte endfeet. - Wikidata - wikimedia - TriplyDB

Glial-conditional deletion of aquaporin-4 (Aqp4) reduces blood-brain water uptake and confers barrier function on perivascular astrocyte endfeet.

Glial-conditional deletion of aquaporin-4 (Aqp4) reduces blood-brain water uptake and confers barrier function on perivascular astrocyte endfeet.

http://www.wikidata.org/entity/Q35471502

about

A new look at cerebrospinal fluid circulation A paravascular pathway facilitates CSF flow through the brain parenchyma and the clearance of interstitial solutes, including amyloid β.Aquaporin-4 and Cerebrovascular Diseases Molecular pathophysiology of cerebral edema Dynamic regulation of aquaporin-4 water channels in neurological disorders Transplantation of stem cell-derived astrocytes for the treatment of amyotrophic lateral sclerosis and spinal cord injury Development, maintenance and disruption of the blood-brain barrier Osmotic homeostasis Physiological roles of aquaporin-4 in brain Novel molecular biomarkers at the blood-brain barrier in ALS Angiopoietin-2-induced blood-brain barrier compromise and increased stroke size are rescued by VE-PTP-dependent restoration of Tie2 signaling.Fluid and ion transfer across the blood-brain and blood-cerebrospinal fluid barriers; a comparative account of mechanisms and roles Aquaporin-4: A Potential Therapeutic Target for Cerebral Edema The impact of alpha-syntrophin deletion on the changes in tissue structure and extracellular diffusion associated with cell swelling under physiological and pathological conditions Regional registration of [6-(14)C]glucose metabolism during brain activation of α-syntrophin knockout mice.Aquaporin-4-dependent K(+) and water transport modeled in brain extracellular space following neuroexcitation Ammonia triggers neuronal disinhibition and seizures by impairing astrocyte potassium buffering.Human and mouse cortical astrocytes differ in aquaporin-4 polarization toward microvessels.Aquaporins: important but elusive drug targets.Gliotransmitters travel in time and space The blood-brain barrier in health and disease.Changes in the astrocytic aquaporin-4 and inwardly rectifying potassium channel expression in the brain of the amyotrophic lateral sclerosis SOD1(G93A) rat model.Evidence that pericytes regulate aquaporin-4 polarization in mouse cortical astrocytes.Selective vasopressin-1a receptor antagonist prevents brain edema, reduces astrocytic cell swelling and GFAP, V1aR and AQP4 expression after focal traumatic brain injury.Drowning stars: reassessing the role of astrocytes in brain edema.Mechanisms of astrocyte-mediated cerebral edema.Biomarkers of traumatic injury are transported from brain to blood via the glymphatic system Reduced brain edema and infarct volume in aquaporin-4 deficient mice after transient focal cerebral ischemia Changes in expression of aquaporin-4 and aquaporin-9 in optic nerve after crushing in rats.Group B streptococcal infection and activation of human astrocytes.APOE Stabilization by Exercise Prevents Aging Neurovascular Dysfunction and Complement Induction.Nanoagonist-mediated endothelial tight junction opening: A strategy for safely increasing brain drug delivery in mice.Heterogeneity of aquaporin-4 localization and expression after focal cerebral ischemia underlies differences in white versus grey matter swelling How astrocyte networks may contribute to cerebral metabolite clearance.Perivascular Accumulation of β-Sheet-Rich Proteins in Offspring Brain following Maternal Exposure to Carbon Black Nanoparticles.Real-time analysis of microglial activation and motility in hepatic and hyperammonemic encephalopathy.Aquaporin water channels in the nervous system.Dose-dependent induction of astrocyte activation and reactive astrogliosis in mouse brain following maternal exposure to carbon black nanoparticle.Ventriculomegaly associated with ependymal gliosis and declines in barrier integrity in the aging human and mouse brain.Glial K⁺ clearance and cell swelling: key roles for cotransporters and pumps.

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P2860

Glial-conditional deletion of aquaporin-4 (Aqp4) reduces blood-brain water uptake and confers barrier function on perivascular astrocyte endfeet.

http://www.wikidata.org/entity/Q35471502

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Glial-conditional deletion of ...... erivascular astrocyte endfeet.

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Glial-conditional deletion of ...... erivascular astrocyte endfeet.

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Glial-conditional deletion of ...... erivascular astrocyte endfeet.

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Proceedings of the National Academy of Sciences of the United States of America

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Glial-conditional deletion of ...... erivascular astrocyte endfeet.

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Anna Elisabeth Thorén

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Erlend Arnulf Nagelhus

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Georg Andreas Gundersen

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Gry Fluge Vindedal

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John Michael Burkhardt

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Martine Eilert-Olsen

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Nadia Nabil Haj-Yasein

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Petter Laake

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P2860

Astrocyte-endothelial interactions at the blood-brain barrier

Longitudinal data analysis (repeated measures) in clinical trials.

Sensitive blood-retinal barrier breakdown quantitation using Evans blue.

Alpha-syntrophin deletion removes the perivascular but not endothelial pool of aquaporin-4 at the blood-brain barrier and delays the development of brain edema in an experimental model of acute hyponatremia.

In vivo measurement of brain extracellular space diffusion by cortical surface photobleaching.

Loss of perivascular aquaporin 4 may underlie deficient water and K+ homeostasis in the human epileptogenic hippocampus.

Induction of aquaporin 1 but not aquaporin 4 messenger RNA in rat primary brain microvessel endothelial cells in culture.

Fine structural localization of a blood-brain barrier to exogenous peroxidase

The molecular basis of water transport in the brain.

Critical role of aquaporin-4 (AQP4) in astrocytic Ca2+ signaling events elicited by cerebral edema.

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17815-17820

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Ole Petter Ottersen

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51709918

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