mdx(⁵cv) mice manifest more severe muscle dysfunction and diaphragm force deficits than do mdx Mice - Wikidata - wikimedia - TriplyDB

mdx(⁵cv) mice manifest more severe muscle dysfunction and diaphragm force deficits than do mdx Mice

mdx(⁵cv) mice manifest more severe muscle dysfunction and diaphragm force deficits than do mdx Mice

http://www.wikidata.org/entity/Q35474357

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Homologous recombination mediates functional recovery of dysferlin deficiency following AAV5 gene transfer Animal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapy MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms Inflammatory monocytes promote progression of Duchenne muscular dystrophy and can be therapeutically targeted via CCR2.Muscle side population cells from dystrophic or injured muscle adopt a fibro-adipogenic fate Dystrophic muscle improvement in zebrafish via increased heme oxygenase signaling.Prednisolone attenuates improvement of cardiac and skeletal contractile function and histopathology by lisinopril and spironolactone in the mdx mouse model of Duchenne muscular dystrophy.AAV.Dysferlin Overlap Vectors Restore Function in Dysferlinopathy Animal Models Toll-like receptor 4 ablation in mdx mice reveals innate immunity as a therapeutic target in Duchenne muscular dystrophy Defective membrane fusion and repair in Anoctamin5-deficient muscular dystrophy.Quantitative evaluation of the beneficial effects in the mdx mouse of epigallocatechin gallate, an antioxidant polyphenol from green tea.In vivo assessment of contractile strength distinguishes differential gene function in skeletal muscle of zebrafish larvae.The force-temperature relationship in healthy and dystrophic mouse diaphragm; implications for translational study design.What do mouse models of muscular dystrophy tell us about the DAPC and its components?Systemic AAV-Mediated β-Sarcoglycan Delivery Targeting Cardiac and Skeletal Muscle Ameliorates Histological and Functional Deficits in LGMD2E Mice.Identification and Function of Fibrocytes in Skeletal Muscle Injury Repair and Muscular Dystrophy.α-smooth muscle actin is not a marker of fibrogenic cell activity in skeletal muscle fibrosis.CCR2 deficiency does not provide sustained improvement of muscular dystrophy in mdx5cv mice.Necroptosis mediates myofibre death in dystrophin-deficient mice

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P2860

mdx(⁵cv) mice manifest more severe muscle dysfunction and diaphragm force deficits than do mdx Mice

http://www.wikidata.org/entity/Q35474357

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

@ast

mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

@en

type

label

mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

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mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

@en

prefLabel

mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

@ast

mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

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P356

J.AJPATH.2011.07.009

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The American Journal of Pathology

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mdx(⁵cv) mice manifest more se ...... orce deficits than do mdx Mice

@en

P2093

Allison Macke

http://www.w3.org/2001/XMLSchema#string

Benjamin D Canan

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Brian K Kaspar

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Christopher M Penton

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Eric K Johnson

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Federica Montanaro

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Haiyan Lu

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Lan Zhou

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Louise R Rodino-Klapac

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Nicholas Beastrom

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P2860

SPP1 genotype is a determinant of disease severity in Duchenne muscular dystrophy

Short telomeres and stem cell exhaustion model Duchenne muscular dystrophy in mdx/mTR mice

Rescue of enzyme deficiency in embryonic diaphragm in a mouse model of metabolic myopathy: Pompe disease

X chromosome-linked muscular dystrophy (mdx) in the mouse

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

Persistent expression of FLAG-tagged micro dystrophin in nonhuman primates following intramuscular and vascular delivery.

Emerging genetic therapies to treat Duchenne muscular dystrophy

Genetic background affects properties of satellite cells and mdx phenotypes.

Recovery of induced mutations for X chromosome-linked muscular dystrophy in mice.

Characterization of progressive motor deficits in mice transgenic for the human Huntington's disease mutation.

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2464-2474

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scholarly article

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10.1016/J.AJPATH.2011.07.009

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5

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2011-09-03T00:00:00Z

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51619373

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21893021

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