Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
about
Role of Transcription Factors in Steatohepatitis and Hypertension after Ethanol: The Epicenter of MetabolismZebrafish: an important tool for liver disease researchDefining hepatic dysfunction parameters in two models of fatty liver disease in zebrafish larvaeActivating transcription factor 6 is necessary and sufficient for alcoholic fatty liver disease in zebrafishAlcohol decreases organic dust-stimulated airway epithelial TNF-Alpha through a nitric oxide and protein kinase-mediated inhibition of TACENew Insights into the Pathogenesis of Alcohol-Induced ER Stress and Liver DiseasesMolecularly defined unfolded protein response subclasses have distinct correlations with fatty liver disease in zebrafish.Zebrafish models of human liver development and diseaseThe expanding role of fish models in understanding non-alcoholic fatty liver disease.Ethanol metabolism and oxidative stress are required for unfolded protein response activation and steatosis in zebrafish with alcoholic liver disease.Effect of BI-1 on insulin resistance through regulation of CYP2E1.Chronic exposure to ethanol causes steatosis and inflammation in zebrafish liver.Key Events Participating in the Pathogenesis of Alcoholic Liver Disease.Autophagy in ethanol-exposed liver disease.Targeting endoplasmic reticulum stress in liver disease.Zebrafish dives into food research: effectiveness assessment of bioactive compounds.Hepatokines: linking nonalcoholic fatty liver disease and insulin resistance.Endoplasmic reticulum involvement in yeast cell death.Human immunodeficiency virus protease inhibitors modulate Ca2+ homeostasis and potentiate alcoholic stress and injury in mice and primary mouse and human hepatocytes.Alcohol Induces Cellular Senescence and Impairs Osteogenic Potential in Bone Marrow-Derived Mesenchymal Stem Cells.Steric contribution of macromolecular crowding to the time and activation energy for preprotein translocation across the endoplasmic reticulum membrane.Inorganic arsenic causes fatty liver and interacts with ethanol to cause alcoholic liver disease in zebrafish.Protective Effects of Diallyl Sulfide Against Ethanol-Induced Injury in Rat Adipose Tissue and Primary Human Adipocytes.Evaluating the effect of diallyl sulfide on regulation of inflammatory mRNA expression in 3T3L1 adipocytes and RAW 264.7 macrophages during ethanol treatment.
P2860
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P2860
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
name
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@ast
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@en
type
label
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@ast
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@en
prefLabel
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@ast
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@en
P2093
P2860
P1476
Alcohol disrupts endoplasmic reticulum function and protein secretion in hepatocytes.
@en
P2093
Ana M Vacaru
Deanna L Howarth
Elisabetta Mormone
Kirsten C Sadler
Lindsey M Costantini
Natalia Nieto
Orkhontuya Tsedensodnom
P2860
P356
10.1111/J.1530-0277.2011.01602.X
P577
2011-07-25T00:00:00Z