Th2 signals induce epithelial injury in mice and are compatible with the biliary atresia phenotype.
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Interleukin-1 Family Cytokines in Liver DiseasesPathogenesis of biliary atresia: defining biology to understand clinical phenotypesIdentification of a plant isoflavonoid that causes biliary atresiaInteractions between Th1 cells and Tregs affect regulation of hepatic fibrosis in biliary atresia through the IFN-γ/STAT1 pathway.Preoperative Serum IL-12p40 Is a Potential Predictor of Kasai Portoenterostomy Outcome in Infants with Biliary Atresia.Biliary repair and carcinogenesis are mediated by IL-33-dependent cholangiocyte proliferation.Gene expression signature for biliary atresia and a role for interleukin-8 in pathogenesis of experimental diseaseInterleukin-13 Activates Distinct Cellular Pathways Leading to Ductular Reaction, Steatosis, and FibrosisHMGB1-promoted and TLR2/4-dependent NK cell maturation and activation take part in rotavirus-induced murine biliary atresia.Natural killer cells promote long-term hepatobiliary inflammation in a low-dose rotavirus model of experimental biliary atresiaWhat Causes Biliary Atresia? Unique Aspects of the Neonatal Immune System Provide Clues to Disease PathogenesisDendritic Cells Regulate Treg-Th17 Axis in Obstructive Phase of Bile Duct Injury in Murine Biliary Atresia.Aetiology of biliary atresia: what is actually known?Clues to the etiology of bile duct injury in biliary atresiaReplication of a GWAS signal in a Caucasian population implicates ADD3 in susceptibility to biliary atresia.Recent Advances in Etiology of Biliary Atresia.Innate lymphoid cells involve in tumorigenesis.Interleukin-8: A critical chemokine in biliary atresia.Pivotal roles of ILCs in hepatic diseases.Hepatic MDR3 expression impacts lipid homeostasis and susceptibility to inflammatory bile duct obstruction in neonates.Type 2 immunity in tissue repair and fibrosis.Foxp3 promoter methylation impairs suppressive function of regulatory T cells in biliary atresia.The dendritic cell-T helper 17-macrophage axis controls cholangiocyte injury and disease progression in murine and human biliary atresia.Glutathione antioxidant pathway activity and reserve determine toxicity and specificity of the biliary toxin biliatresone in zebrafish.Perforin and granzymes work in synergy to mediate cholangiocyte injury in experimental biliary atresia.Ambiguous roles of innate lymphoid cells in chronic development of liver diseases.
P2860
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P2860
Th2 signals induce epithelial injury in mice and are compatible with the biliary atresia phenotype.
description
2011 nî lūn-bûn
@nan
2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@ast
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@en
type
label
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@ast
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@en
prefLabel
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@ast
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@en
P2093
P2860
P356
P1476
Th2 signals induce epithelial ...... the biliary atresia phenotype.
@en
P2093
Gilda Porta
Irene K Miura
Jorge A Bezerra
Jorge L Dos Santos
Kazuhiko Bessho
Pranavkumar Shivakumar
Reena Mourya
Sujit Kumar Mohanty
P2860
P304
P356
10.1172/JCI57728
P407
P577
2011-10-17T00:00:00Z