ARID1A, a factor that promotes formation of SWI/SNF-mediated chromatin remodeling, is a tumor suppressor in gynecologic cancers - Wikidata - wikimedia - TriplyDB

ARID1A, a factor that promotes formation of SWI/SNF-mediated chromatin remodeling, is a tumor suppressor in gynecologic cancers

ARID1A, a factor that promotes formation of SWI/SNF-mediated chromatin remodeling, is a tumor suppressor in gynecologic cancers

http://www.wikidata.org/entity/Q35501591

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The spectrum of SWI/SNF mutations, ubiquitous in human cancers Residual complexes containing SMARCA2 (BRM) underlie the oncogenic drive of SMARCA4 (BRG1) mutation New and emerging factors in tumorigenesis: an overview Molecular landscape of pancreatic cancer: implications for current clinical trials Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.ATP-dependent chromatin remodeling complexes as novel targets for cancer therapy Recurrent SMARCA4 mutations in small cell carcinoma of the ovary.Synovial sarcoma: recent discoveries as a roadmap to new avenues for therapy Synthetic lethality by targeting EZH2 methyltransferase activity in ARID1A-mutated cancers ARID1A mutations in cancer: another epigenetic tumor suppressor?The SWI/SNF genetic blockade: effects in cell differentiation, cancer and developmental diseases ARID1A gene silencing reduces the sensitivity of ovarian clear cell carcinoma to cisplatin Deletion of Arid1a in Reproductive Tract Mesenchymal Cells Reduces Fertility in Female Mice.ARID1A Is Essential for Endometrial Function during Early Pregnancy Integrative network analysis of TCGA data for ovarian cancer.Bromodomain Protein BRD4 Is a Transcriptional Repressor of Autophagy and Lysosomal Function.Roles of deletion of Arid1a, a tumor suppressor, in mouse ovarian tumorigenesis Identification of drivers from cancer genome diversity in hepatocellular carcinoma.Analysis of Tp53 codon 72 polymorphisms, Tp53 mutations, and HPV infection in cutaneous squamous cell carcinomas.Flavonoids from each of the six structural groups reactivate BRM, a possible cofactor for the anticancer effects of flavonoids The target therapy of ovarian clear cell carcinoma.Decreased expression of the ARID1A gene is associated with poor prognosis in primary gastric cancer.Improving accuracy for cancer classification with a new algorithm for genes selection.Mutant p53 cooperates with the SWI/SNF chromatin remodeling complex to regulate VEGFR2 in breast cancer cells Adenocarcinoma of Mullerian origin: review of pathogenesis, molecular biology, and emerging treatment paradigms.ARID1A alterations are associated with FGFR3-wild type, poor-prognosis, urothelial bladder tumors.ARID1A expression in gastric adenocarcinoma: clinicopathological significance and correlation with DNA mismatch repair status.A functional proteogenomic analysis of endometrioid and clear cell carcinomas using reverse phase protein array and mutation analysis: protein expression is histotype-specific and loss of ARID1A/BAF250a is associated with AKT phosphorylation New routes to targeted therapy of intrahepatic cholangiocarcinomas revealed by next-generation sequencing.Ovarian cancer: new developments in clear cell carcinoma and hopes for targeted therapy.ARID1A Deficiency Impairs the DNA Damage Checkpoint and Sensitizes Cells to PARP Inhibitors Special Agents Hunting Down Women Silent Killer: The Emerging Role of the p38α Kinase.Ovarian Cancer Is an Imported Disease: Fact or Fiction?Identification of multiple pathways involved in the malignant transformation of endometriosis (Review)The genomics and genetics of endometrial cancer.ARID1A loss correlates with mismatch repair deficiency and intact p53 expression in high-grade endometrial carcinomas SWI/SNF regulates a transcriptional program that induces senescence to prevent liver cancer.Gliotoxin Inhibits Proliferation and Induces Apoptosis in Colorectal Cancer Cells.Loss of ARID1A expression is an early molecular event in tumor progression from ovarian endometriotic cyst to clear cell and endometrioid carcinoma Functional analysis of in-frame indel ARID1A mutations reveals new regulatory mechanisms of its tumor suppressor functions.

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ARID1A, a factor that promotes formation of SWI/SNF-mediated chromatin remodeling, is a tumor suppressor in gynecologic cancers

http://www.wikidata.org/entity/Q35501591

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Cancer Research

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Ie-Ming Shih

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Tian-Li Wang

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P2860

The human SWI-SNF complex protein p270 is an ARID family member with non-sequence-specific DNA binding activity

SWI/SNF complex interacts with tumor suppressor p53 and is necessary for the activation of p53-mediated transcription

Cloning and characterization of hELD/OSA1, a novel BRG1 interacting protein

A novel E1A-like inhibitor of differentiation (EID) family member, EID-2, suppresses transforming growth factor (TGF)-beta signaling by blocking TGF-beta-induced formation of Smad3-Smad4 complexes

Largest subunits of the human SWI/SNF chromatin-remodeling complex promote transcriptional activation by steroid hormone receptors

HIC1 interacts with a specific subunit of SWI/SNF complexes, ARID1A/BAF250A

Purification and biochemical heterogeneity of the mammalian SWI-SNF complex

BAF60a interacts with p53 to recruit the SWI/SNF complex

Exome sequencing identifies frequent mutation of the SWI/SNF complex gene PBRM1 in renal carcinoma

Two related ARID family proteins are alternative subunits of human SWI/SNF complexes

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6718-6727

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