Hepatitis C virus infection: molecular pathways to insulin resistance. - Wikidata - wikimedia - TriplyDB

Hepatitis C virus infection: molecular pathways to insulin resistance.

Hepatitis C virus infection: molecular pathways to insulin resistance.

http://www.wikidata.org/entity/Q35505394

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Metabolic factors and chronic hepatitis C: a complex interplay Trypanosoma cruzi infection is a potent risk factor for non-alcoholic steatohepatitis enhancing local and systemic inflammation associated with strong oxidative stress and metabolic disorders Hepatitis C virus NS5A promotes insulin resistance through IRS-1 serine phosphorylation and increased gluconeogenesis.Regulation of FOXO3 by phosphorylation and methylation in hepatitis C virus infection and alcohol exposure.The central question of type 2 diabetes.Lack of Association between Hepatitis C Virus core Gene Variation 70/91aa and Insulin Resistance Hepatitis C virus infection induces elevation of CXCL10 in human brain microvascular endothelial cells.Factors Associated with Improved Glycemic Control by Direct-Acting Antiviral Agent Treatment in Egyptian Type 2 Diabetes Mellitus Patients with Chronic Hepatitis C Genotype 4.Hepatitis C virus NS5A protein enhances gluconeogenesis through upregulation of Akt-/JNK-PEPCK signalling pathways.Effect of branched-chain amino acid supplementation on insulin resistance and quality of life in chronic hepatitis C patients.Development of experimental fibrotic liver diseases animal model by Carbon Tetracholoride.

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Hepatitis C virus infection: molecular pathways to insulin resistance.

http://www.wikidata.org/entity/Q35505394

description

2011 nî lūn-bûn

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2011 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Hepatitis C virus infection: molecular pathways to insulin resistance.

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Fahed Parvaiz

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Farakh Javed

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Huma Tariq

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Kaneez Fatima

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Creative Commons Attribution 2.0 Generic

P2860

Hepatitis C virus subgenomic replicons induce endoplasmic reticulum stress activating an intracellular signaling pathway

Control of PKR protein kinase by hepatitis C virus nonstructural 5A protein: molecular mechanisms of kinase regulation.

Hepatitis C Virus Down-Regulates Insulin Receptor Substrates 1 and 2 through Up-Regulation of Suppressor of Cytokine Signaling 3

Involvement of the PA28 -Dependent Pathway in Insulin Resistance Induced by Hepatitis C Virus Core Protein

Worldwide variation in the relative importance of hepatitis B and hepatitis C viruses in hepatocellular carcinoma: a systematic review

HCV Induces Oxidative and ER Stress, and Sensitizes Infected Cells to Apoptosis in SCID/Alb-uPA Mice

A study of best positive predictors for sustained virologic response to interferon alpha plus ribavirin therapy in naive chronic hepatitis C patients

Isolation of a cDNA clone derived from a blood-borne non-A, non-B viral hepatitis genome

SOCS-1 and SOCS-3 block insulin signaling by ubiquitin-mediated degradation of IRS1 and IRS2

Insulin resistance, insulin insensitivity, and insulin unresponsiveness: a necessary distinction

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10.1186/1743-422X-8-474

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type-2 diabetes

regulation of gene expression

maturity-onset diabetes of the young

physiological phenomena

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