Transforming growth factor beta is protective in host resistance against Listeria monocytogenes infection in mice - Wikidata - wikimedia - TriplyDB

Transforming growth factor beta is protective in host resistance against Listeria monocytogenes infection in mice

Transforming growth factor beta is protective in host resistance against Listeria monocytogenes infection in mice

http://www.wikidata.org/entity/Q35520360

about

Ambiguous role of interleukin-12 in Yersinia enterocolitica infection in susceptible and resistant mouse strains.Identification of tgh-2, a filarial nematode homolog of Caenorhabditis elegans daf-7 and human transforming growth factor beta, expressed in microfilarial and adult stages of Brugia malayi.Effect of transforming growth factor beta on experimental Salmonella typhimurium infection in mice Listeria monocytogenes impairs SUMOylation for efficient infection.A physical/psychological and biological stress combine to enhance endoplasmic reticulum stress.

P2860

Q33753916-D6485542-ADB6-4D08-AA8A-171DBE88D6AA Q34005039-149C09B3-4C4A-4C08-9F6B-5CD2CA6D9A0E Q39510169-552D7074-470D-46F5-B48A-0F98B7328A1A Q39711579-D186C3C5-2B57-4D50-A725-167820C32592 Q40989468-66E47C28-8D93-4739-9FA8-0D28AD8A6F03

P2860

Transforming growth factor beta is protective in host resistance against Listeria monocytogenes infection in mice

http://www.wikidata.org/entity/Q35520360

description

1996 nî lūn-bûn

@nan

1996 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

1996 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

1996年の論文

@ja

1996年論文

@yue

1996年論文

@zh-hant

1996年論文

@zh-hk

1996年論文

@zh-mo

1996年論文

@zh-tw

1996年论文

@wuu

name

Transforming growth factor bet ...... onocytogenes infection in mice

@ast

Transforming growth factor bet ...... onocytogenes infection in mice

@en

type

label

Transforming growth factor bet ...... onocytogenes infection in mice

@ast

Transforming growth factor bet ...... onocytogenes infection in mice

@en

prefLabel

Transforming growth factor bet ...... onocytogenes infection in mice

@ast

Transforming growth factor bet ...... onocytogenes infection in mice

@en

P1433

Q35520360-6B3156C8-6283-440B-9B78-4BE8805A9AC8

P1476

Q35520360-92A930FD-DFAD-45C5-BCE2-5CD0ED3A7D66

P2093

Q35520360-07108887-1D1B-4B88-8DB9-3853EB175CBA

Q35520360-161E48FF-A0EE-415C-97D2-D94A3992C5F7

Q35520360-1EA457E4-FD7A-49A4-9902-0708EDA1B07A

Q35520360-3F01B691-293D-4979-9096-97D031368A32

Q35520360-B091B038-03FA-43EF-9F96-1F04D35F81D8

Q35520360-EE57384D-4B6B-44AF-8B92-955A08BA5A73

Q35520360-F72D357E-DAC4-4F9D-A5E6-68B72644B178

P2860

Q35520360-0BFB1B1F-38ED-4411-B566-D5F37122DCD2

Q35520360-190EF293-06FE-496A-BFDE-3CE6A6CCC4AD

Q35520360-224DDEC3-E166-4510-AA82-CB77A36DD9E0

Q35520360-242D8F11-896F-44D6-92B1-71A2C7896D56

Q35520360-2B0EBFF7-A3E2-4F18-8C62-D34175B5B574

Q35520360-2DECD99A-B95A-4303-B80B-53310BB85CB4

Q35520360-2E91F0C9-A2D1-44DC-B950-9370E6E289B8

Q35520360-3E242526-AE74-4375-9459-500337BCC9C2

Q35520360-584CF7CE-5167-4DFD-AB84-210950CFB233

Q35520360-66FF21EB-E627-41B5-9D5D-AB0445112590

P304

Q35520360-E90714FA-5F1E-4762-8534-4F23E1D5CE8F

P31

Q35520360-05EEAB27-4262-480F-B1D4-0533F0E686BE

P407

Q35520360-8310C2E9-BB43-48BA-9B7B-31CAB5D89AB8

P433

Q35520360-46D0C415-AF07-48D6-9E3F-098920AB5CFC

P478

Q35520360-2A316C8C-4A21-4668-9A4B-3388D0BC4B90

P577

Q35520360-BBE819DF-1B08-485D-AC3E-3543F36C1C30

P698

Q35520360-260AFE9A-DDB3-48D8-9BB1-8776CABD51B7

P921

Q35520360-79B7A3F2-5F7F-48F8-9319-D27F0D25E184

P932

Q35520360-63E1C595-4247-4A13-A626-E2465CDD24E5

P1433

Infection and Immunity

P1476

Transforming growth factor bet ...... onocytogenes infection in mice

@en

P2093

Asano M

http://www.w3.org/2001/XMLSchema#string

Kohanawa M

http://www.w3.org/2001/XMLSchema#string

Minagawa T

http://www.w3.org/2001/XMLSchema#string

Miura T

http://www.w3.org/2001/XMLSchema#string

Nakane A

http://www.w3.org/2001/XMLSchema#string

Nishikawa S

http://www.w3.org/2001/XMLSchema#string

Sasaki S

http://www.w3.org/2001/XMLSchema#string

P2860

Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

Endogenous interleukin-4, but not interleukin-10, is involved in suppression of host resistance against Listeria monocytogenes infection in interferon-depleted mice

Transforming growth factor beta as a virulence mechanism for Leishmania braziliensis

Production of transforming growth factor beta by human T lymphocytes and its potential role in the regulation of T cell growth.

Inhibition of cytotoxic T cell development by transforming growth factor beta and reversal by recombinant tumor necrosis factor alpha.

Inhibition of cytokine production by cyclosporin A and transforming growth factor beta.

Endogenous tumor necrosis factor, interleukin-6, and gamma interferon levels during Listeria monocytogenes infection in mice.

Endogenous tumor necrosis factor (cachectin) is essential to host resistance against Listeria monocytogenes infection.

Requirement of endogenous interferon-gamma production for resolution of Listeria monocytogenes infection.

Transforming growth factor-beta and IL-4 cause helper T cell precursors to develop into distinct effector helper cells that differ in lymphokine secretion pattern and cell surface phenotype.

P304

3901-3904

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P407

P433

9

http://www.w3.org/2001/XMLSchema#string

P478

64

http://www.w3.org/2001/XMLSchema#string

P577

1996-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

8751946

http://www.w3.org/2001/XMLSchema#string

P921

Listeria monocytogenes

P932

174310

http://www.w3.org/2001/XMLSchema#string