Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
about
Endothelin-1 and its role in the pathogenesis of infectious diseasesBoth functional LTbeta receptor and TNF receptor 2 are required for the development of experimental cerebral malariaMolecular basis of human cerebral malaria developmentParasite burden and CD36-mediated sequestration are determinants of acute lung injury in an experimental malaria model.Malaria: a haematological disease.Serum angiopoietin-1 and -2 levels discriminate cerebral malaria from uncomplicated malaria and predict clinical outcome in African childrenDiagnosis and management of the neurological complications of falciparum malariaEllagitannins of the fruit rind of pomegranate (Punica granatum) antagonize in vitro the host inflammatory response mechanisms involved in the onset of malariaRole of the aryl hydrocarbon receptor in the immune response profile and development of pathology during Plasmodium berghei Anka infectionStudy on association between genetic polymorphisms of haem oxygenase-1, tumour necrosis factor, cadmium exposure and malaria pathogenicity and severity.Loss of Toll-like receptor 7 alters cytokine production and protects against experimental cerebral malariaCommon and divergent immune response signaling pathways discovered in peripheral blood mononuclear cell gene expression patterns in presymptomatic and clinically apparent malaria.IL-33-mediated protection against experimental cerebral malaria is linked to induction of type 2 innate lymphoid cells, M2 macrophages and regulatory T cells.Tumour necrosis factor allele variants and their association with the occurrence and severity of malaria in African children: a longitudinal study.Review Article: blood-brain barrier in falciparum malaria.Polymorphisms of TNF-enhancer and gene for FcgammaRIIa correlate with the severity of falciparum malaria in the ethnically diverse Indian population.Similar cytokine responses and degrees of anemia in patients with Plasmodium falciparum and Plasmodium vivax infections in the Brazilian Amazon region.C5 deficiency and C5a or C5aR blockade protects against cerebral malariaEarly prediction of cerebral malaria by (1)H NMR based metabolomicsCerebral Malaria: An Unusual Cause of Central Diabetes Insipidus.Platelet factor 4 mediates inflammation in experimental cerebral malaria.Decreased prevalence of sepsis but not mild or severe P. falciparum malaria is associated with pre-existing filarial infection.Genome-wide association studies of immune, disease and production traits in indigenous chicken ecotypes.Malaria-induced murine pregnancy failure: distinct roles for IFN-gamma and TNF.Exploring experimental cerebral malaria pathogenesis through the characterisation of host-derived plasma microparticle protein contentTumor necrosis factor superfamily molecules in acute coronary syndromes.Peroxisome proliferator activating receptor (PPAR) in cerebral malaria (CM): a novel target for an additional therapy.Host matrix metalloproteinases in cerebral malaria: new kids on the block against blood-brain barrier integrity?Evaluation of leptin, interleukin-1 beta and tumor necrosis factor alpha in serum of malaria patients as prognostic markers of treatment outcome.Severe malaria in children in areas with low, moderate and high transmission intensity in Uganda.Associations between frequencies of a susceptible TNF-alpha promoter allele and protective alpha-thalassaemias and malaria parasite incidence in Vanuatu.Regulatory multitasking of tolerogenic dendritic cells - lessons taken from vitamin d3-treated tolerogenic dendritic cells.TRPV1 antagonism by capsazepine modulates innate immune response in mice infected with Plasmodium berghei ANKA.Involvement of inflammatory chemokines in survival of human monocytes fed with malarial pigment.Plasmodium falciparum-infected erythrocytes increase intercellular adhesion molecule 1 expression on brain endothelium through NF-kappaB.Phagocytosis of haemozoin (malarial pigment) enhances metalloproteinase-9 activity in human adherent monocytes: role of IL-1beta and 15-HETE.Tumor necrosis factor alpha antibody prevents brain damage of rats with acute necrotizing pancreatitis.Matrix metalloproteinases and their tissue inhibitors (TIMPs) in Plasmodium falciparum malaria: serum levels of TIMP-1 are associated with disease severity.Understanding host-parasite relationship: the immune central nervous system microenvironment and its effect on brain infections.Matrix metalloproteinases, tissue inhibitors of MMPs and TACE in experimental cerebral malaria.
P2860
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P2860
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@ast
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@en
type
label
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@ast
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@en
prefLabel
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@ast
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@en
P2093
P1476
Tumor necrosis factor alpha in the pathogenesis of cerebral malaria.
@en
P2093
Barraud de Lagerie S
Fernandez C
P2888
P304
P356
10.1007/S00018-003-2347-X
P577
2003-08-01T00:00:00Z