Augmented central nitric oxide production inhibits vasopressin release during hemorrhage in acute alcohol-intoxicated rodents
about
Alcohol abuse: critical pathophysiological processes and contribution to disease burdenAlcohol exposure after mild focal traumatic brain injury impairs neurological recovery and exacerbates localized neuroinflammation.Hypertonic saline resuscitation enhances blood pressure recovery and decreases organ injury following hemorrhage in acute alcohol intoxicated rodentsAlcohol abuse and the injured host: dysregulation of counterregulatory mechanisms review.Angiotensin (1-7) contributes to nitric oxide tonic inhibition of vasopressin release during hemorrhagic shock in acute ethanol intoxicated rodents.Acute alcohol intoxication prolongs neuroinflammation without exacerbating neurobehavioral dysfunction following mild traumatic brain injury.
P2860
Augmented central nitric oxide production inhibits vasopressin release during hemorrhage in acute alcohol-intoxicated rodents
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@ast
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@en
type
label
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@ast
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@en
prefLabel
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@ast
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@en
P2093
P2860
P1476
Augmented central nitric oxide ...... te alcohol-intoxicated rodents
@en
P2093
Annie M Whitaker
Jesse K Sulzer
Patricia E Molina
P2860
P304
P356
10.1152/AJPREGU.00035.2011
P577
2011-08-17T00:00:00Z