Impaired Rho GTPase activation abrogates cell polarization and migration in macrophages with defective lipolysis.
about
Vascular growth responses to chronic arterial occlusion are unaffected by myeloid specific focal adhesion kinase (FAK) deletion.Maternal Plane of Nutrition during Late Gestation and Weaning Age Alter Angus × Simmental Offspring Longissimus Muscle Transcriptome and Intramuscular Fat.PEDF increases the tumoricidal activity of macrophages towards prostate cancer cells in vitro.Macrophage CGI-58 deficiency activates ROS-inflammasome pathway to promote insulin resistance in miceMacrophage models of Gaucher disease for evaluating disease pathogenesis and candidate drugs.Estrogen-related receptor α decreases RHOA stability to induce orientated cell migration.Deletion of CGI-58 or adipose triglyceride lipase differently affects macrophage function and atherosclerosis.Recombinant Treponema pallidum protein Tp0965 activates endothelial cells and increases the permeability of endothelial cell monolayer.Active autophagy but not lipophagy in macrophages with defective lipolysis.Impact of Reduced ATGL-Mediated Adipocyte Lipolysis on Obesity-Associated Insulin Resistance and Inflammation in Male Mice.ACE2/Ang-(1-7)/Mas axis stimulates vascular repair-relevant functions of CD34+ cells.Hydrogen Sulfide Recruits Macrophage Migration by Integrin β1-Src-FAK/Pyk2-Rac Pathway in Myocardial InfarctionCell motility is decreased in macrophages activated by cancer cell-conditioned medium.Regulation of Hepatic Triacylglycerol Metabolism by CGI-58 Does Not Require ATGL Co-activation.Adipose triglyceride lipase in immune response, inflammation, and atherosclerosis.Opposing effects of in vitro differentiated macrophages sub-type on epithelial wound healing.Integrated regulation of the cellular metabolism and function of immune cells in adipose tissue.Pigment epithelium-derived factor regulates microvascular permeability through adipose triglyceride lipase in sepsis.Muscle cells challenged with saturated fatty acids mount an autonomous inflammatory response that activates macrophages.Identification of pigment epithelium-derived factor as an adipocyte-derived inflammatory factor.Macrophage CGI-58 deficiency promotes IL-1β transcription by activating the SOCS3-FOXO1 pathway.Identification of Key Pathways and Genes in Advanced Coronary Atherosclerosis Using Bioinformatics Analysis.Critical roles for α/β hydrolase domain 5 (ABHD5)/comparative gene identification-58 (CGI-58) at the lipid droplet interface and beyond.Hints on ATGL implications in cancer: beyond bioenergetic clues.
P2860
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P2860
Impaired Rho GTPase activation abrogates cell polarization and migration in macrophages with defective lipolysis.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@ast
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@en
type
label
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@ast
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@en
prefLabel
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@ast
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@en
P2093
P2860
P50
P1476
Impaired Rho GTPase activation ...... ages with defective lipolysis.
@en
P2093
Elma Aflaki
Nariman A B Balenga
Petra Luschnig-Schratl
Prakash G Chandak
Sandra Eder
Sepp-Dieter Kohlwein
Silvia Povoden
P2860
P2888
P304
P356
10.1007/S00018-011-0688-4
P577
2011-05-02T00:00:00Z