Gastroenteritis in NF-kappaB-deficient mice is produced with wild-type Camplyobacter jejuni but not with C. jejuni lacking cytolethal distending toxin despite persistent colonization with both strains.
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Impact of CDT Toxin on Human DiseasesMolecular Mechanisms and Potential Clinical Applications of Campylobacter jejuni Cytolethal Distending ToxinRole of regulatory T cell in the pathogenesis of inflammatory bowel diseaseBacterial Genotoxins: Merging the DNA Damage Response into Infection BiologyContribution of amino acid catabolism to the tissue specific persistence of Campylobacter jejuni in a murine colonization modelThe Cytolethal Distending Toxin Contributes to Microbial Virulence and Disease Pathogenesis by Acting As a Tri-Perditious ToxinOutcome of infection of C57BL/6 IL-10(-/-) mice with Campylobacter jejuni strains is correlated with genome content of open reading frames up- and down-regulated in vivoThe NF-kappaB p50 subunit is protective during intestinal Entamoeba histolytica infection of 129 and C57BL/6 mice.Multiple factors interact to produce responses resembling spectrum of human disease in Campylobacter jejuni infected C57BL/6 IL-10-/- mice.Gnotobiotic IL-10; NF-kappaB mice develop rapid and severe colitis following Campylobacter jejuni infection.Use of genome-wide expression profiling and mutagenesis to study the intestinal lifestyle of Campylobacter jejuni.Delineation of the innate and adaptive T-cell immune outcome in the human host in response to Campylobacter jejuni infection.Cytolethal distending toxin is essential for Helicobacter hepaticus colonization in outbred Swiss Webster mice.Characterization of cytolethal distending toxin of campylobacter species isolated from captive macaque monkeys.Dendritic cells from C57BL/6 mice undergo activation and induce Th1-effector cell responses against Campylobacter jejuni.Genetic background of IL-10(-/-) mice alters host-pathogen interactions with Campylobacter jejuni and influences disease phenotype.Cytolethal distending toxin: a conserved bacterial genotoxin that blocks cell cycle progression, leading to apoptosis of a broad range of mammalian cell lineagesMicrobial imbalance and intestinal pathologies: connections and contributionsModulation of host immune responses by the cytolethal distending toxin of Helicobacter hepaticus.The microbiome and cancer.Use of a rabbit soft tissue chamber model to investigate campylobacter jejuni-host interactions.Prevalence and distribution of Aggregatibacter actinomycetemcomitans and its cdtB gene in subgingival plaque of Chinese periodontitis patients.Carcinogenic bacterial pathogen Helicobacter pylori triggers DNA double-strand breaks and a DNA damage response in its host cellsCytolethal distending toxins require components of the ER-associated degradation pathway for host cell entry.Functional impacts of the intestinal microbiome in the pathogenesis of inflammatory bowel disease.C57BL/6 and congenic interleukin-10-deficient mice can serve as models of Campylobacter jejuni colonization and enteritis.A MyD88-deficient mouse model reveals a role for Nramp1 in Campylobacter jejuni infection.Streptococcus pneumoniae secretes hydrogen peroxide leading to DNA damage and apoptosis in lung cells.Distinct Roles for CdtA and CdtC during Intoxication by Cytolethal Distending ToxinsMUC1 cell surface mucin is a critical element of the mucosal barrier to infection.Inhibitory effect of enterohepatic Helicobacter hepaticus on innate immune responses of mouse intestinal epithelial cells.The Typhoid Toxin Promotes Host Survival and the Establishment of a Persistent Asymptomatic Infection.Campylobacter jejuni outer membrane vesicles play an important role in bacterial interactions with human intestinal epithelial cellsImmune response to cytolethal distending toxin of Aggregatibacter actinomycetemcomitans in periodontitis patients.The contribution of cytolethal distending toxin to bacterial pathogenesis.Genotoxicity of Cytolethal Distending Toxin (CDT) on Isogenic Human Colorectal Cell Lines: Potential Promoting Effects for Colorectal Carcinogenesis.Oxymatrine prevents NF-κB nuclear translocation and ameliorates acute intestinal inflammation.Host-pathogen interactions in Campylobacter infections: the host perspective.NF-kappaB and the intestine: friend or foe?Correlation of proinflammatory and anti-inflammatory cytokine levels with histopathological changes in an adult mouse lung model of Campylobacter jejuni infection.
P2860
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P2860
Gastroenteritis in NF-kappaB-deficient mice is produced with wild-type Camplyobacter jejuni but not with C. jejuni lacking cytolethal distending toxin despite persistent colonization with both strains.
description
2004 nî lūn-bûn
@nan
2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@ast
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@en
type
label
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@ast
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@en
prefLabel
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@ast
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@en
P2093
P2860
P1476
Gastroenteritis in NF-kappaB-d ...... olonization with both strains.
@en
P2093
Arlin B Rogers
Bruce H Horwitz
Mark T Whary
Nancy S Taylor
Susan E Erdman
Zhongming Ge
P2860
P304
P356
10.1128/IAI.72.2.1116-1125.2004
P407
P50
P577
2004-02-01T00:00:00Z