Chronic exposure to neonicotinoids increases neuronal vulnerability to mitochondrial dysfunction in the bumblebee (Bombus terrestris).
about
A restatement of recent advances in the natural science evidence base concerning neonicotinoid insecticides and insect pollinatorsNeonicotinoid Insecticides and Their Impacts on Bees: A Systematic Review of Research Approaches and Identification of Knowledge GapsImproving Mitochondrial Function Protects Bumblebees from Neonicotinoid PesticidesLarval exposure to field-realistic concentrations of clothianidin has no effect on development rate, over-winter survival or adult metabolic rate in a solitary bee, Osmia bicornisBehavioral and metabolic effects of sublethal doses of two insecticides, chlorpyrifos and methomyl, in the Egyptian cotton leafworm, Spodoptera littoralis (Boisduval) (Lepidoptera: Noctuidae).No effect of low-level chronic neonicotinoid exposure on bumblebee learning and fecundity.Neonicotinoids target distinct nicotinic acetylcholine receptors and neurons, leading to differential risks to bumblebees.Chronic exposure to a neonicotinoid pesticide alters the interactions between bumblebees and wild plants.Large-scale monitoring of effects of clothianidin-dressed OSR seeds on pollinating insects in Northern Germany: effects on large earth bumble bees (Bombus terrestris).Effect of acute pesticide exposure on bee spatial working memory using an analogue of the radial-arm mazeInvestigating the impacts of field-realistic exposure to a neonicotinoid pesticide on bumblebee foraging, homing ability and colony growth.Bumblebee colony development following chronic exposure to field-realistic levels of the neonicotinoid pesticide thiamethoxam under laboratory conditionsAgrochemical-induced stress in stingless bees: peculiarities, underlying basis, and challenges.Weight of evidence evaluation of a network of adverse outcome pathways linking activation of the nicotinic acetylcholine receptor in honey bees to colony death.An update of the Worldwide Integrated Assessment (WIA) on systemic insecticides. Part 1: new molecules, metabolism, fate, and transport.Insect models of central nervous system energy metabolism and its links to behavior.Organic honey supplementation reverses pesticide-induced genotoxicity by modulating DNA damage response.Imidacloprid slows the development of preference for rewarding food sources in bumblebees (Bombus impatiens).Nerve agents in honey.Chronic neonicotinoid pesticide exposure and parasite stress differentially affects learning in honeybees and bumblebees.The Challenge: Assessment of risks posed by systemic insecticides to hymenopteran pollinators: New perception when we move from laboratory via (semi-)field to landscape scale testing?
P2860
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P2860
Chronic exposure to neonicotinoids increases neuronal vulnerability to mitochondrial dysfunction in the bumblebee (Bombus terrestris).
description
2015 nî lūn-bûn
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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris).
@ast
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris).
@en
type
label
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris).
@ast
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris).
@en
prefLabel
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris).
@ast
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris).
@en
P2093
P2860
P50
P356
P1433
P1476
Chronic exposure to neonicotin ...... bumblebee (Bombus terrestris)
@en
P2093
Christopher Moffat
Jeffrey Huang
Karen A Bollan
Sheila Sharp
Stephen T Buckland
P2860
P304
P356
10.1096/FJ.14-267179
P407
P577
2015-01-29T00:00:00Z