Fluid shear stress induces endothelial transforming growth factor beta-1 transcription and production. Modulation by potassium channel blockade. - Wikidata - wikimedia - TriplyDB

Fluid shear stress induces endothelial transforming growth factor beta-1 transcription and production. Modulation by potassium channel blockade.

Fluid shear stress induces endothelial transforming growth factor beta-1 transcription and production. Modulation by potassium channel blockade.

http://www.wikidata.org/entity/Q35554195

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Vascular MADs: two novel MAD-related genes selectively inducible by flow in human vascular endothelium Expression of TGF-beta1 and beta3 but not apoptosis factors relates to flow-induced aortic enlargement.Acute development of collateral circulation and therapeutic prospects in ischemic stroke Stop the flow: a paradigm for cell signaling mediated by reactive oxygen species in the pulmonary endothelium Emerging techniques for evaluation of the hemodynamics of intracranial vascular pathology TGF-beta: a crucial component of the pathogenesis of diabetic nephropathy The National NeuroAIDS Tissue Consortium brain gene array: two types of HIV-associated neurocognitive impairment.Mechanisms underlying renoprotection during renin-angiotensin system blockade.Vascular endothelium, hemodynamic forces, and atherogenesis.Hereditary haemorrhagic telangiectasia and pulmonary arteriovenous malformations: issues in clinical management and review of pathogenic mechanisms Matrix rigidity induces osteolytic gene expression of metastatic breast cancer cells.Autocrine and paracrine mechanisms in the early stages of diabetic nephropathy.DNA microarray reveals changes in gene expression of shear stressed human umbilical vein endothelial cells.Shear stress regulates the endothelial Kir2.1 ion channel.The effects of hemodynamic force on embryonic development.A novel flow bioreactor for in vitro microvascularization.Flow, NO, and atherogenesis The key role of the transforming growth factor-beta system in the pathogenesis of diabetic nephropathy.Endothelial cellular response to altered shear stress.Genetic aspects of pulmonary arterial hypertension.Ventilation-induced lung injury and mechanotransduction: stretching it too far?Flow-mediated endothelial mechanotransduction.Transforming growth factor-β in graft vessels: histology and immunohistochemistry.Induction and activation of latent transforming growth factor-β1 are carried out by two distinct domains of pregnancy-specific glycoprotein 1 (PSG1).Transforming growth factor-beta 1 hyperexpression in African-American hypertensives: A novel mediator of hypertension and/or target organ damage.Physiological impact of continuous flow on end-organ function: clinical implications in the current era of left ventricular assist devices.Angiogenic factors in the central nervous system.Effects of Smad decoy ODN on shear stress-induced atherosclerotic ApoE-/-mouse.Pulmonary hemodynamics modify the rat pulmonary artery response to injury. A neointimal model of pulmonary hypertension Varicose veins: role of mechanotransduction of venous hypertension.Collateral growth in the peripheral circulation: a review.Fetal wound healing: implications for minimal scar formation.Valve-Related Hemodynamics Mediate Human Bicuspid Aortopathy: Insights From Wall Shear Stress Mapping.Effect of dietary salt on regulation of TGF-β in the kidney.Extracellular matrix and the mechanics of large artery development.Injury-activated transforming growth factor β controls mobilization of mesenchymal stem cells for tissue remodeling.Endothelial Cell Dysfunction and the Pathobiology of Atherosclerosis Molecular cloning and expression of a bovine endothelial inward rectifier potassium channel.Vascular consequences of dietary salt intake.Shear stress induction of the tissue factor gene.

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P2860

Fluid shear stress induces endothelial transforming growth factor beta-1 transcription and production. Modulation by potassium channel blockade.

http://www.wikidata.org/entity/Q35554195

description

1995 nî lūn-bûn

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1995 թուականի Մարտին հրատարակուած գիտական յօդուած

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1995 թվականի մարտին հրատարակված գիտական հոդված

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1995年の論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年論文

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1995年论文

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Fluid shear stress induces end ...... by potassium channel blockade.

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Fluid shear stress induces end ...... by potassium channel blockade.

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Journal of Clinical Investigation

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Fluid shear stress induces end ...... by potassium channel blockade.

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P2093

G H Gibbons

http://www.w3.org/2001/XMLSchema#string

J P Cooke

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M Ohno

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V J Dzau

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P2860

Transforming growth factor-beta-induced growth inhibition and cellular hypertrophy in cultured vascular smooth muscle cells.

Hydrodynamic hyperpolarization of endothelial cells.

An activated form of transforming growth factor beta is produced by cocultures of endothelial cells and pericytes.

Fluid shear stress differentially modulates expression of genes encoding basic fibroblast growth factor and platelet-derived growth factor B chain in vascular endothelium

Hemodynamics and atherosclerosis. Insights and perspectives gained from studies of human arteries.

Flow activates an endothelial potassium channel to release an endogenous nitrovasodilator

Vascular smooth muscle cell hypertrophy vs. hyperplasia. Autocrine transforming growth factor-beta 1 expression determines growth response to angiotensin II.

Transforming growth factor-beta activity is potentiated by heparin via dissociation of the transforming growth factor-beta/alpha 2-macroglobulin inactive complex.

Inhibition of endothelial cell movement by pericytes and smooth muscle cells: activation of a latent transforming growth factor-beta 1-like molecule by plasmin during co-culture

Platelet-derived growth factor B chain promoter contains a cis-acting fluid shear-stress-responsive element.

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1363-1369

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10.1172/JCI117787

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3

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95

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7883983

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441476

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