HSV-1 gene expression from reactivated ganglia is disordered and concurrent with suppression of latency-associated transcript and miRNAs
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The role of the CoREST/REST repressor complex in herpes simplex virus 1 productive infection and in latencyHerpes simplex virus type 1 latency and reactivation: an updateA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationA cultured affair: HSV latency and reactivation in neuronsLocal CD4 and CD8 T-cell reactivity to HSV-1 antigens documents broad viral protein expression and immune competence in latently infected human trigeminal gangliaHerpes simplex virus and the lexicon of latency and reactivation: a call for defining terms and building an integrated collective frameworkTransient reversal of episome silencing precedes VP16-dependent transcription during reactivation of latent HSV-1 in neuronsRestarting Lytic Gene Transcription at the Onset of Herpes Simplex Virus ReactivationNeuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation.Immune Escape via a Transient Gene Expression Program Enables Productive Replication of a Latent Pathogen.The stability of herpes simplex virus 1 ICP0 early after infection is defined by the RING finger and the UL13 protein kinase.Transcription of the herpes simplex virus 1 genome during productive and quiescent infection of neuronal and nonneuronal cells.Induction of varicella zoster virus DNA replication in dissociated human trigeminal gangliaLytic gene expression is frequent in HSV-1 latent infection and correlates with the engagement of a cell-intrinsic transcriptional responseHerpes keratitis.Cells infected with herpes simplex virus 1 export to uninfected cells exosomes containing STING, viral mRNAs, and microRNAs.Three-dimensional normal human neural progenitor tissue-like assemblies: a model of persistent varicella-zoster virus infectionPatterns of accumulation of miRNAs encoded by herpes simplex virus during productive infection, latency, and on reactivation.A herpes simplex virus type 1 mutant disrupted for microRNA H2 with increased neurovirulence and rate of reactivation.Characterization of herpes simplex virus 2 primary microRNA Transcript regulationAn in vitro model of latency and reactivation of varicella zoster virus in human stem cell-derived neuronsApoptosis-induced activation of HIV-1 in latently infected cell linesHSV-1 miR-H6 inhibits HSV-1 replication and IL-6 expression in human corneal epithelial cells in vitro.Lytic Promoters Express Protein during Herpes Simplex Virus Latency.Control of viral latency in neurons by axonal mTOR signaling and the 4E-BP translation repressor.Role of activating transcription factor 3 in the synthesis of latency-associated transcript and maintenance of herpes simplex virus 1 in latent state in ganglia.Induction of apoptosis accelerates reactivation of latent HSV-1 in ganglionic organ cultures and replication in cell cultures.Kinetics of facultative heterochromatin and polycomb group protein association with the herpes simplex viral genome during establishment of latent infection.HSV carrying WT REST establishes latency but reactivates only if the synthesis of REST is suppressed.miR-H28 and miR-H29 expressed late in productive infection are exported and restrict HSV-1 replication and spread in recipient cellsThe herpes simplex virus host shutoff RNase degrades cellular and viral mRNAs made before infection but not viral mRNA made after infection.PML plays both inimical and beneficial roles in HSV-1 replicationModulation of reactivation of latent herpes simplex virus 1 in ganglionic organ cultures by p300/CBP and STAT3.Bovine herpesvirus 1 regulatory proteins bICP0 and VP16 are readily detected in trigeminal ganglionic neurons expressing the glucocorticoid receptor during the early stages of reactivation from latency.Activation of human herpesvirus replication by apoptosisStress-induced cellular transcription factors expressed in trigeminal ganglionic neurons stimulate the herpes simplex virus 1 ICP0 promoterMutations Inactivating Herpes Simplex Virus 1 MicroRNA miR-H2 Do Not Detectably Increase ICP0 Gene Expression in Infected Cultured Cells or Mouse Trigeminal Ganglia.The herpes simplex virus type 1 (HSV-1) latency-associated transcript (LAT) protects cells against cold-shock-induced apoptosis by maintaining phosphorylation of protein kinase B (AKT)The 3 facets of regulation of herpes simplex virus gene expression: A critical inquiry.Neurons versus herpes simplex virus: the innate immune interactions that contribute to a host-pathogen standoff
P2860
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P2860
HSV-1 gene expression from reactivated ganglia is disordered and concurrent with suppression of latency-associated transcript and miRNAs
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@ast
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@en
type
label
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@ast
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@en
prefLabel
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@ast
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@en
P2860
P356
P1476
HSV-1 gene expression from rea ...... sociated transcript and miRNAs
@en
P2093
P2860
P304
18820-18824
P356
10.1073/PNAS.1117203108
P407
P577
2011-11-07T00:00:00Z