Injury, sepsis, and the regulation of Toll-like receptor responses.
about
Impact of interleukin-10, soluble CD25 and interferon-γ on the prognosis and early diagnosis of bacteremic systemic inflammatory response syndrome: a prospective observational studyInsights into the regulation of TNF-alpha production in human mononuclear cells: the effects of non-specific phosphodiesterase inhibitionCurrent views on the mechanisms of immune responses to trauma and infectionDivergent adaptive and innate immunological responses are observed in humans following blunt trauma.Identification and interpretation of longitudinal gene expression changes in trauma.Salutary effects of 17beta-estradiol on Peyer's patch T cell functions following trauma-hemorrhage.IL-15 prevents apoptosis, reverses innate and adaptive immune dysfunction, and improves survival in sepsis.Claude H. Organ, Jr. memorial lecture: splanchnic hypoperfusion provokes acute lung injury via a 5-lipoxygenase-dependent mechanism.Modulation of acetylation: creating a pro-survival and anti-inflammatory phenotype in lethal hemorrhagic and septic shock.Heme oxygenase-1 protects against neutrophil-mediated intestinal damage by down-regulation of neutrophil p47phox and p67phox activity and O2- production in a two-hit model of alcohol intoxication and burn injury.Acute alcohol intoxication potentiates neutrophil-mediated intestinal tissue damage after burn injury.Inhibition of protein tyrosine phosphatases prevents mesenteric lymph node T-cell suppression following alcohol intoxication and burn injury.MyD88 and Src are differentially regulated in Kupffer cells of males and proestrus females following hypoxia.Trauma is dangerCecal ligation and puncture-induced impairment of innate immune function does not occur in the absence of caspase-1Clinical recovery from surgery correlates with single-cell immune signatures.Burn-induced alterations in toll-like receptor-mediated responses by bronchoalveolar lavage cells.Effects of Rhubarb combined with ulinastatin on T-cell subsets in sepsis rats.Acute acalculous cholecystitis-like phenotype in scavenger receptor A knock-out mice.Kupffer cells and their mediators: the culprits in producing distant organ damage after trauma-hemorrhage.Histone deacetylase inhibitor treatment attenuates MAP kinase pathway activation and pulmonary inflammation following hemorrhagic shock in a rodent model.Burn Injury Alters the Intestinal Microbiome and Increases Gut Permeability and Bacterial TranslocationSurface-micromachined microfiltration membranes for efficient isolation and functional immunophenotyping of subpopulations of immune cells.Effects of trauma-hemorrhage and IL-6 deficiency on splenic immune function in a murine trauma model.Immunosuppression in patients who die of sepsis and multiple organ failure.Period of irreversible therapeutic intervention during sepsis correlates with phase of innate immune dysfunction.Role of A(2A) adenosine receptors in regulation of opsonized E. coli-induced macrophage function.Innate immunity gene expression changes in critically ill patients with sepsis and disease-related malnutrition.Opioids and opiates: analgesia with cardiovascular, haemodynamic and immune implications in critical illness.Patient-specific Immune States before Surgery Are Strong Correlates of Surgical Recovery.Adenosine A2A receptor inactivation increases survival in polymicrobial sepsis.17Beta-estradiol downregulates Kupffer cell TLR4-dependent p38 MAPK pathway and normalizes inflammatory cytokine production following trauma-hemorrhage.Sex differences and estrogen modulation of the cellular immune response after injury.Creating a pro-survival and anti-inflammatory phenotype by modulation of acetylation in models of hemorrhagic and septic shock.Role of estrogen receptor subtypes in estrogen-induced organ-specific vasorelaxation after trauma-hemorrhage.Effect of interleukin-15 on depressed splenic dendritic cell functions following trauma-hemorrhageAlcohol, burn injury, and the intestine.17beta-Estradiol's salutary effects on splenic dendritic cell functions following trauma-hemorrhage are mediated via estrogen receptor-alpha.Toll-like receptor-4 antagonist eritoran tetrasodium for severe sepsis.The effects of estrogen on various organs: therapeutic approach for sepsis, trauma, and reperfusion injury. Part 1: central nervous system, lung, and heart.
P2860
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P2860
Injury, sepsis, and the regulation of Toll-like receptor responses.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Injury, sepsis, and the regulation of Toll-like receptor responses.
@ast
Injury, sepsis, and the regulation of Toll-like receptor responses.
@en
type
label
Injury, sepsis, and the regulation of Toll-like receptor responses.
@ast
Injury, sepsis, and the regulation of Toll-like receptor responses.
@en
prefLabel
Injury, sepsis, and the regulation of Toll-like receptor responses.
@ast
Injury, sepsis, and the regulation of Toll-like receptor responses.
@en
P2093
P2860
P356
P1476
Injury, sepsis, and the regulation of Toll-like receptor responses.
@en
P2093
James A Lederer
John A Mannick
Thomas J Murphy
P2860
P304
P356
10.1189/JLB.0503233
P577
2003-10-13T00:00:00Z