Targets of miR-200c mediate suppression of cell motility and anoikis resistance
about
MiRNAs and Other Epigenetic Changes as Biomarkers in Triple Negative Breast CancerRole of redoximiRs in fibrogenesisThe role of miRNAs in stress-responsive hepatic stellate cells during liver fibrosisBreast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticityMicroRNAs and triple negative breast cancerThe miR-200 and miR-221/222 microRNA families: opposing effects on epithelial identityEpithelial-mesenchymal transition: focus on metastatic cascade, alternative splicing, non-coding RNAs and modulating compoundsLinking metabolism and epigenetic regulation in development of hepatocellular carcinomaThe human PMR1 endonuclease stimulates cell motility by down regulating miR-200 family microRNAs.miR-200c Regulation of Metastases in Ovarian Cancer: Potential Role in Epithelial and Mesenchymal TransitionThe microRNA-200 family: small molecules with novel roles in cancer development, progression and therapyEvaluation of MicroRNAs Regulating Anoikis Pathways and Its Therapeutic PotentialmiR-200c sensitizes breast cancer cells to doxorubicin treatment by decreasing TrkB and Bmi1 expressionmiR-8 modulates cytoskeletal regulators to influence cell survival and epithelial organization in Drosophila wings.Dysregulation of uterine signaling pathways in progesterone receptor-Cre knockout of dicer.Re-expression of miR-200c suppresses proliferation, colony formation and in vivo tumor growth of murine claudin-low mammary tumor cells.The tetraindole SK228 reverses the epithelial-to-mesenchymal transition of breast cancer cells by up-regulating members of the miR-200 family.MicroRNA-200c and microRNA- 141 are regulated by a FOXP3-KAT2B axis and associated with tumor metastasis in breast cancer.Decreased levels of baseline and drug-induced tubulin polymerisation are hallmarks of resistance to taxanes in ovarian cancer cells and are associated with epithelial-to-mesenchymal transitionMicroRNA-200b suppresses arsenic-transformed cell migration by targeting protein kinase Cα and Wnt5b-protein kinase Cα positive feedback loop and inhibiting Rac1 activationmicroRNA alterations in ALDH positive mammary epithelial cells: a crucial contributing factor towards breast cancer risk reduction in case of early pregnancy.MicroRNA-200b targets protein kinase Cα and suppresses triple-negative breast cancer metastasisOverexpression of microRNA-200c predicts poor outcome in patients with PR-negative breast cancer.MicroRNA-200 family modulation in distinct breast cancer phenotypes.miR-200c targets a NF-κB up-regulated TrkB/NTF3 autocrine signaling loop to enhance anoikis sensitivity in triple negative breast cancerRegulation of trabecular meshwork cell contraction and intraocular pressure by miR-200c.Progestin suppression of miR-29 potentiates dedifferentiation of breast cancer cells via KLF4.Tumor growth in complex, evolving microenvironmental geometries: a diffuse domain approach.Reduced expression of miR-200 family members contributes to antiestrogen resistance in LY2 human breast cancer cellsNovel small RNA expression libraries uncover hsa-miR-30b and hsa-miR-30c as important factors in anoikis resistance.MUC1 regulates expression of multiple microRNAs involved in pancreatic tumor progression, including the miR-200c/141 cluster.Sequential Salinomycin Treatment Results in Resistance Formation through Clonal Selection of Epithelial-Like Tumor Cells.Characterization of genome-wide TFCP2 targets in hepatocellular carcinoma: implication of targets FN1 and TJP1 in metastasisMicroRNA-200c modulates the expression of MUC4 and MUC16 by directly targeting their coding sequences in human pancreatic cancer.A TrkB-STAT3-miR-204-5p regulatory circuitry controls proliferation and invasion of endometrial carcinoma cellsmiR-200c regulates IL8 expression by targeting IKBKB: a potential mediator of inflammation in leiomyoma pathogenesis.Progesterone downregulation of miR-141 contributes to expansion of stem-like breast cancer cells through maintenance of progesterone receptor and Stat5a.p53 in stem cellsβ-catenin/TCF4 complex induces the epithelial-to-mesenchymal transition (EMT)-activator ZEB1 to regulate tumor invasiveness.MicroRNA-200c represses migration and invasion of breast cancer cells by targeting actin-regulatory proteins FHOD1 and PPM1F.
P2860
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P2860
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@ast
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@en
type
label
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@ast
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@en
prefLabel
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@ast
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@en
P2093
P2860
P356
P1476
Targets of miR-200c mediate suppression of cell motility and anoikis resistance
@en
P2093
Dawn R Cochrane
Erin N Howe
Jennifer K Richer
P2860
P2888
P356
10.1186/BCR2867
P577
2011-04-18T00:00:00Z
P5875
P6179
1033017956