Intestinal Epithelial TLR-4 Activation Is Required for the Development of Acute Lung Injury after Trauma/Hemorrhagic Shock via the Release of HMGB1 from the Gut
about
Nutrition: A Primary Therapy in Pediatric Acute Respiratory Distress SyndromePaneth-cell-disruption-induced necrotizing enterocolitis in mice requires live bacteria and occurs independently of TLR4 signalingENDOPLASMIC RETICULUM STRESS IN SEPSIS.Blockade of Interplay between IL-17A and Endoplasmic Reticulum Stress Attenuates LPS-Induced Lung InjuryHigh Mobility Group Box Protein 1 (HMGB1): The Prototypical Endogenous Danger MoleculeToll-like receptor 4-mediated lymphocyte influx induces neonatal necrotizing enterocolitis.Novel strategies for targeting innate immune responses to influenza.Cold-inducible RNA-binding protein (CIRP) causes sepsis-associated acute lung injury via induction of endoplasmic reticulum stress.HMGB1, IL-1α, IL-33 and S100 proteins: dual-function alarminsThe haptoglobin beta subunit sequesters HMGB1 toxicity in sterile and infectious inflammation.Pulmonary Epithelial TLR4 Activation Leads to Lung Injury in Neonatal Necrotizing Enterocolitis.Ketamine effect on HMGB1 and TLR4 expression in rats with acute lung injury.Toll-Like Receptor 4 on both Myeloid Cells and Dendritic Cells Is Required for Systemic Inflammation and Organ Damage after Hemorrhagic Shock with Tissue Trauma in Mice.Attenuation of hemorrhage-associated lung injury by adjuvant treatment with C23, an oligopeptide derived from cold-inducible RNA-binding protein.Mitochondrial dysfunction in rat splenocytes following hemorrhagic shock.Protective Effect of Atazanavir Sulphate Against Pulmonary Fibrosis In Vivo and In Vitro.Immunothrombotic Activity of Damage-Associated Molecular Patterns and Extracellular Vesicles in Secondary Organ Failure Induced by Trauma and Sterile Insults.Toll-like receptor 4 is critical for the development of resection-associated hepatic steatosis.Innate immune responses to trauma.Trauma-Induced Damage-Associated Molecular Patterns-Mediated Remote Organ Injury and Immunosuppression in the Acutely Ill Patient.Fat composition in infant formula contributes to the severity of necrotising enterocolitisReduced peripheral blood miR-140 may be a biomarker for acute lung injury by targeting Toll-like receptor 4 (TLR4)
P2860
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P2860
Intestinal Epithelial TLR-4 Activation Is Required for the Development of Acute Lung Injury after Trauma/Hemorrhagic Shock via the Release of HMGB1 from the Gut
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@ast
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@en
type
label
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@ast
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@en
prefLabel
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@ast
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@en
P2093
P2860
P356
P1476
Intestinal Epithelial TLR-4 Ac ...... Release of HMGB1 from the Gut
@en
P2093
Charlotte Egan
Chhinder P Sodhi
David J Hackam
Hongpeng Jia
John Ozolek
Timothy R Billiar
Xiaorong Zhu
Yukihiro Yamaguchi
P2860
P304
P356
10.4049/JIMMUNOL.1402490
P407
P50
P577
2015-04-10T00:00:00Z