The reverse evolution from multicellularity to unicellularity during carcinogenesis.
about
Measuring intratumor heterogeneity by network entropy using RNA-seq data.Ancient genes establish stress-induced mutation as a hallmark of cancerCancer in the parasitic protozoans Trypanosoma brucei and Toxoplasma gondii.Evolutionary and Topological Properties of Genes and Community Structures in Human Gene Regulatory Networks.Different BCR/Abl protein suppression patterns as a converging trait of chronic myeloid leukemia cell adaptation to energy restriction.Evolutionary Origins of Cancer Driver Genes and Implications for Cancer Prognosis.Similarity in gene-regulatory networks suggests that cancer cells share characteristics of embryonic neural cells.Altered interactions between unicellular and multicellular genes drive hallmarks of transformation in a diverse range of solid tumors.Natural Selection in Cancer Biology: From Molecular Snowflakes to Trait Hallmarks.The dJ/dS Ratio Test Reveals Hundreds of Novel Putative Cancer Drivers.Free-living human cells reconfigure their chromosomes in the evolution back to uni-cellularity.Tumorigenesis as a process of gradual loss of original cell identity and gain of properties of neural precursor/progenitor cells.Mechanisms and clinical implications of tumor heterogeneity and convergence on recurrent phenotypes.Evolutionary and genetic features of drug targets.How the evolution of multicellularity set the stage for cancer.The Convergent Cancer Evolution toward a Single Cellular Destination.A Pan-Cancer Analysis of Enhancer Expression in Nearly 9000 Patient Samples.Phosphotyrosine signalling and the origin of animal multicellularity.Somatic mosaicism: on the road to cancer.Resistance to cancer chemotherapy as an atavism? (retrospective on DOI 10.1002/bies.201300170).Evolution of oncogenic signatures of mutation hotspots in tyrosine kinases supports the atavistic hypothesis of cancer.Does cancer strive to minimize the cost of gene expression?Culture shockExperimental Design, Population Dynamics, and Diversity in Microbial Experimental EvolutionBiosynthetic energy cost for amino acids decreases in cancer evolutionDecelerated DNA methylation age predicts poor prognosis of breast cancerViability Assessment Following Anticancer Treatment Requires Single-Cell Visualization
P2860
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P2860
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@ast
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@en
type
label
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@ast
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@en
prefLabel
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@ast
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@en
P2093
P2860
P356
P1476
The reverse evolution from multicellularity to unicellularity during carcinogenesis.
@en
P2093
P2507
P2860
P2888
P356
10.1038/NCOMMS7367
P407
P577
2015-03-09T00:00:00Z