Elevation of cellular BPDE uptake by human cells: a possible factor contributing to co-carcinogenicity by arsenite

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Inorganic arsenic inhibits the nucleotide excision repair pathway and reduces the expression of XPC

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Elevation of cellular BPDE uptake by human cells: a possible factor contributing to co-carcinogenicity by arsenite

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Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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P1476

Elevation of cellular BPDE upt ...... co-carcinogenicity by arsenite

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P2093

Hailin Wang

http://www.w3.org/2001/XMLSchema#string

Jane Lee

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Michael Weinfeld

http://www.w3.org/2001/XMLSchema#string

Shengwen Shen

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X Chris Le

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Xuejun Sun

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P2860

Effect of arsenic on benzo[a]pyrene DNA adduct levels in mouse skin and lung

The role of biomethylation in toxicity and carcinogenicity of arsenic: a research update.

Heat shock and arsenite increase expression of the multidrug resistance (MDR1) gene in human renal carcinoma cells

Chronic exposure to methylated arsenicals stimulates arsenic excretion pathways and induces arsenic tolerance in rat liver cells

Arsenite and its biomethylated metabolites interfere with the formation and repair of stable BPDE-induced DNA adducts in human cells and impair XPAzf and Fpg.

Nonenzymatic reduction of benzo(a)pyrene diol-epoxides to trihydroxypentahydrobenzo(a)pyrenes by reduced nicotinamide adenine dinucleotide phosphate.

Inducible repair of thymine glycol detected by an ultrasensitive assay for DNA damage.

Chronic arsenic-exposed human prostate epithelial cells exhibit stable arsenic tolerance: mechanistic implications of altered cellular glutathione and glutathione S-transferase.

Upregulation of glutathione-related genes and enzyme activities in cultured human cells by sublethal concentrations of inorganic arsenic.

Pharmacokinetics, metabolism, and carcinogenicity of arsenic.

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1832-1837

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scholarly article

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10.1289/EHP.9284

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114

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2006-12-01T00:00:00Z

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6615715

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17185271

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1764144

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Elevation of cellular BPDE uptake by human cells: a possible factor contributing to co-carcinogenicity by arsenite

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Elevation of cellular BPDE uptake by human cells: a possible factor contributing to co-carcinogenicity by arsenite

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P356

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P698

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P1476

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P478

P577

P5875

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