Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunction.
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Elucidating immune mechanisms causing hypertension during pregnancyAngiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyondPregnant rats treated with a high-fat/prooxidant Western diet with ANG II and TNF-α are resistant to elevations in blood pressure and renal oxidative stressThe role of inflammation in the pathology of preeclampsiaAdvances in the pathophysiology of pre-eclampsia and related podocyte injury.Autoantibody-mediated complement C3a receptor activation contributes to the pathogenesis of preeclampsia.Receptor-activating autoantibodies and disease: preeclampsia and beyond.Hypertension in response to CD4(+) T cells from reduced uterine perfusion pregnant rats is associated with activation of the endothelin-1 system.IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancy.Timing of ischemic insult alters fetal growth trajectory, maternal angiogenic balance, and markers of renal oxidative stress in the pregnant rat.Blockade of CD40 ligand for intercellular communication reduces hypertension, placental oxidative stress, and AT1-AA in response to adoptive transfer of CD4+ T lymphocytes from RUPP ratsAn increased population of regulatory T cells improves the pathophysiology of placental ischemia in a rat model of preeclampsia.Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia.Vitamin D supplementation improves pathophysiology in a rat model of preeclampsiaIL-10 supplementation increases Tregs and decreases hypertension in the RUPP rat model of preeclampsia.Mechanisms of Endothelial Dysfunction in Hypertensive Pregnancy and Preeclampsia.Endothelial dysfunction. An important mediator in the pathophysiology of hypertension during pre-eclampsia.Angiotensin II type 1 receptor autoantibody (AT1-AA)-mediated pregnancy hypertension.Placental Ischemia and Resultant Phenotype in Animal Models of Preeclampsia.Administration of interleukin-17 soluble receptor C suppresses TH17 cells, oxidative stress, and hypertension in response to placental ischemia during pregnancy.Tumor necrosis factor-alpha, interleukin-6, and interleukin-10 levels are altered in preeclampsia: a systematic review and meta-analysis.Vascular dysfunction in preeclampsia.A complicated role for the renin-angiotensin system during pregnancy: highlighting the importance of drug discovery.Proliferation of endogenous regulatory T cells improve the pathophysiology associated with placental ischaemia of pregnancy.Recent Advances in Immunity and Hypertension.Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy.Matrix Metalloproteinases in Normal Pregnancy and Preeclampsia.The agonistic autoantibodies to the angiotensin II type 1 receptor in pregnancies complicated by hypertensive disorders.
P2860
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P2860
Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunction.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@ast
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@en
type
label
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@ast
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@en
prefLabel
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@ast
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@en
P2093
P2860
P1433
P1476
Angiotensin II type 1 autoanti ...... renal endothelial dysfunction.
@en
P2093
Babbette B Lamarca
James N Martin
Justin Brewer
Lillian Ray
Marc R Parrish
Michael J Ryan
Porter Glover
P2860
P304
P356
10.1016/J.GENM.2011.04.003
P50
P577
2011-05-20T00:00:00Z