Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice.
about
Peroxisome proliferator activated receptors at the crossroad of obesity, diabetes, and pancreatic cancerRole of Notch signaling in regulating innate immunity and inflammation in health and diseaseGEMMs as preclinical models for testing pancreatic cancer therapiesCritical role of NF-κB in pancreatic cancerNotch signaling as a novel regulator of metabolismThe wound healing, chronic fibrosis, and cancer progression triadSignaling cross-talk in the resistance to HER family receptor targeted therapyAnti-pancreatic cancer deliverables from sea: first-hand evidence on the efficacy, molecular targets and mode of action for multifarious polyphenols from five different brown-algaeMolecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.Inflammation and Epithelial-Mesenchymal Transition in Pancreatic Ductal Adenocarcinoma: Fighting Against Multiple OpponentsDesign, synthesis, and biological evaluation of novel EF24 and EF31 analogs as potential IκB kinase β inhibitors for the treatment of pancreatic cancerNOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB.Nuclear receptors and pathogenesis of pancreatic cancer.Downstream of mutant KRAS, the transcription regulator YAP is essential for neoplastic progression to pancreatic ductal adenocarcinomaMutant KRas-Induced Mitochondrial Oxidative Stress in Acinar Cells Upregulates EGFR Signaling to Drive Formation of Pancreatic Precancerous Lesions.Peroxisome proliferator activated receptor-α/hypoxia inducible factor-1α interplay sustains carbonic anhydrase IX and apoliprotein E expression in breast cancer stem cells.Inhibition of oxidative stress-elicited AKT activation facilitates PPARγ agonist-mediated inhibition of stem cell character and tumor growth of liver cancer cellsRegeneration and repair of the exocrine pancreas.MiR-196a promotes pancreatic cancer progression by targeting nuclear factor kappa-B-inhibitor alphaConstitutive activation of IKK2/NF-κB impairs osteogenesis and skeletal development.Protein kinase D1 drives pancreatic acinar cell reprogramming and progression to intraepithelial neoplasiaThe role of Notch signaling in muscle progenitor cell depletion and the rapid onset of histopathology in muscular dystrophy.Inactivation of Ink4a/Arf leads to deregulated expression of miRNAs in K-Ras transgenic mouse model of pancreatic cancer.First quantitative high-throughput screen in zebrafish identifies novel pathways for increasing pancreatic β-cell mass.The acinar differentiation determinant PTF1A inhibits initiation of pancreatic ductal adenocarcinomaCXCR2 Inhibition Profoundly Suppresses Metastases and Augments Immunotherapy in Pancreatic Ductal Adenocarcinoma.De novo transcriptome sequencing and analysis of freshwater snail (Radix balthica) to discover genes and pathways affected by exposure to oxazepam.Toll-like receptor 7 regulates pancreatic carcinogenesis in mice and humans.Network insights on oxaliplatin anti-cancer mechanisms.Evidence of functional cross talk between the Notch and NF-κB pathways in nonneoplastic hyperproliferating colonic epithelium.Genetically engineered mouse models of pancreatic cancer.Targeting the NF-κB and mTOR pathways with a quinoxaline urea analog that inhibits IKKβ for pancreas cancer therapy.Notch Signaling Mediates Skeletal Muscle Atrophy in Cancer Cachexia Caused by Osteosarcoma.CD163 interacts with TWEAK to regulate tissue regeneration after ischaemic injuryMacrophages in pancreatic cancer: starting things off on the wrong track.Notch-induced transcription factors are predictive of survival and 5-fluorouracil response in colorectal cancer patients.The PRKD1 promoter is a target of the KRas-NF-κB pathway in pancreatic cancer.LTβR signalling preferentially accelerates oncogenic AKT-initiated liver tumoursNFκB signaling regulates embryonic and adult neurogenesis.Challenges and advances in mouse modeling for human pancreatic tumorigenesis and metastasis
P2860
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P2860
Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Crosstalk between the canonica ...... ic cancer progression in mice.
@ast
Crosstalk between the canonica ...... ic cancer progression in mice.
@en
type
label
Crosstalk between the canonica ...... ic cancer progression in mice.
@ast
Crosstalk between the canonica ...... ic cancer progression in mice.
@en
prefLabel
Crosstalk between the canonica ...... ic cancer progression in mice.
@ast
Crosstalk between the canonica ...... ic cancer progression in mice.
@en
P2093
P2860
P50
P356
P1476
Crosstalk between the canonica ...... ic cancer progression in mice.
@en
P2093
Eleni Maniati
Juliana B Candido
Maud Bossard
Nia Emami-Shahri
Sergei A Nedospasov
Thorsten Hagemann
P2860
P304
P356
10.1172/JCI45797
P407
P577
2011-11-07T00:00:00Z