Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice. - Wikidata - wikimedia - TriplyDB

Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice.

Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice.

http://www.wikidata.org/entity/Q35578568

about

Peroxisome proliferator activated receptors at the crossroad of obesity, diabetes, and pancreatic cancer Role of Notch signaling in regulating innate immunity and inflammation in health and disease GEMMs as preclinical models for testing pancreatic cancer therapies Critical role of NF-κB in pancreatic cancer Notch signaling as a novel regulator of metabolism The wound healing, chronic fibrosis, and cancer progression triad Signaling cross-talk in the resistance to HER family receptor targeted therapy Anti-pancreatic cancer deliverables from sea: first-hand evidence on the efficacy, molecular targets and mode of action for multifarious polyphenols from five different brown-algae Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.Inflammation and Epithelial-Mesenchymal Transition in Pancreatic Ductal Adenocarcinoma: Fighting Against Multiple Opponents Design, synthesis, and biological evaluation of novel EF24 and EF31 analogs as potential IκB kinase β inhibitors for the treatment of pancreatic cancer NOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB.Nuclear receptors and pathogenesis of pancreatic cancer.Downstream of mutant KRAS, the transcription regulator YAP is essential for neoplastic progression to pancreatic ductal adenocarcinoma Mutant KRas-Induced Mitochondrial Oxidative Stress in Acinar Cells Upregulates EGFR Signaling to Drive Formation of Pancreatic Precancerous Lesions.Peroxisome proliferator activated receptor-α/hypoxia inducible factor-1α interplay sustains carbonic anhydrase IX and apoliprotein E expression in breast cancer stem cells.Inhibition of oxidative stress-elicited AKT activation facilitates PPARγ agonist-mediated inhibition of stem cell character and tumor growth of liver cancer cells Regeneration and repair of the exocrine pancreas.MiR-196a promotes pancreatic cancer progression by targeting nuclear factor kappa-B-inhibitor alpha Constitutive activation of IKK2/NF-κB impairs osteogenesis and skeletal development.Protein kinase D1 drives pancreatic acinar cell reprogramming and progression to intraepithelial neoplasia The role of Notch signaling in muscle progenitor cell depletion and the rapid onset of histopathology in muscular dystrophy.Inactivation of Ink4a/Arf leads to deregulated expression of miRNAs in K-Ras transgenic mouse model of pancreatic cancer.First quantitative high-throughput screen in zebrafish identifies novel pathways for increasing pancreatic β-cell mass.The acinar differentiation determinant PTF1A inhibits initiation of pancreatic ductal adenocarcinoma CXCR2 Inhibition Profoundly Suppresses Metastases and Augments Immunotherapy in Pancreatic Ductal Adenocarcinoma.De novo transcriptome sequencing and analysis of freshwater snail (Radix balthica) to discover genes and pathways affected by exposure to oxazepam.Toll-like receptor 7 regulates pancreatic carcinogenesis in mice and humans.Network insights on oxaliplatin anti-cancer mechanisms.Evidence of functional cross talk between the Notch and NF-κB pathways in nonneoplastic hyperproliferating colonic epithelium.Genetically engineered mouse models of pancreatic cancer.Targeting the NF-κB and mTOR pathways with a quinoxaline urea analog that inhibits IKKβ for pancreas cancer therapy.Notch Signaling Mediates Skeletal Muscle Atrophy in Cancer Cachexia Caused by Osteosarcoma.CD163 interacts with TWEAK to regulate tissue regeneration after ischaemic injury Macrophages in pancreatic cancer: starting things off on the wrong track.Notch-induced transcription factors are predictive of survival and 5-fluorouracil response in colorectal cancer patients.The PRKD1 promoter is a target of the KRas-NF-κB pathway in pancreatic cancer.LTβR signalling preferentially accelerates oncogenic AKT-initiated liver tumours NFκB signaling regulates embryonic and adult neurogenesis.Challenges and advances in mouse modeling for human pancreatic tumorigenesis and metastasis

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P2860

Crosstalk between the canonical NF-κB and Notch signaling pathways inhibits Pparγ expression and promotes pancreatic cancer progression in mice.

http://www.wikidata.org/entity/Q35578568

description

2011 nî lūn-bûn

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2011年の論文

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2011年论文

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2011年论文

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name

Crosstalk between the canonica ...... ic cancer progression in mice.

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Crosstalk between the canonica ...... ic cancer progression in mice.

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Crosstalk between the canonica ...... ic cancer progression in mice.

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Crosstalk between the canonica ...... ic cancer progression in mice.

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Crosstalk between the canonica ...... ic cancer progression in mice.

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Crosstalk between the canonica ...... ic cancer progression in mice.

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Journal of Clinical Investigation

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Crosstalk between the canonica ...... ic cancer progression in mice.

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Eleni Maniati

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Juliana B Candido

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Maud Bossard

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Nia Emami-Shahri

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Sergei A Nedospasov

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Thorsten Hagemann

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P2860

Rel/NF-kappaB can trigger the Notch signaling pathway by inducing the expression of Jagged1, a ligand for Notch receptors

Inflammation and mitochondrial fatty acid beta-oxidation link obesity to early tumor promotion

Cancer-related inflammation

Immunity, Inflammation, and Cancer

Targeting RAS signalling pathways in cancer therapy

HES and HERP families: multiple effectors of the Notch signaling pathway

Dangerous liaisons: STAT3 and NF-kappaB collaboration and crosstalk in cancer

Signalling downstream of activated mammalian Notch

Notch signalling: a simple pathway becomes complex

Inducible gene targeting in mice

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12

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121

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Frances R Balkwill

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2011-11-07T00:00:00Z

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51772862

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