JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
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Immune System Involvement in the Pathogenesis of JC Virus Induced PML: What is Learned from Studies of Patients with Underlying Diseases and Therapies as Risk FactorsProgressive multi-focal leucoencephalopathy - driven from rarity to clinical mainstream by iatrogenic immunodeficiency.Progressive Multifocal Leukoencephalopathy in HIV-Uninfected Individuals.Persistence and pathogenesis of the neurotropic polyomavirus JCImmunological Markers for PML Prediction in MS Patients Treated with NatalizumabHematopoietic mobilization: Potential biomarker of response to natalizumab in multiple sclerosis.Lack of Major Histocompatibility Complex Class I Upregulation and Restrictive Infection by JC Virus Hamper Detection of Neurons by T Lymphocytes in the Central Nervous System.Brief Report: Role of Thymic Reconstitution in the Outcome of AIDS-Related PMLImmunotherapies for Neurological Manifestations in the Context of Systemic Autoimmunity.Prevalence of Antibodies Against JC Virus in Patients With Refractory Crohn's Disease and Effects of Natalizumab Therapy.Progressive Multifocal Leukoencephalopathy in Primary Immune Deficiencies: Stat1 Gain of Function and Review of the Literature.Immunology of progressive multifocal leukoencephalopathy.Natalizumab in relapsing-remitting multiple sclerosis.Cerebellar Involvement in an Immunocompetent Patient Presenting with Progressive Multifocal Leukoencephalopathy.JC virus reactivation in patients with autoimmune rheumatic diseases treated with rituximab.Monitoring the John Cunningham virus throughout natalizumab treatment in multiple sclerosis patients.Protective Humoral Immunity in the CNS Requires Peripheral CD19-Dependent Germinal Center Formation Following Coronavirus Encephalomyelitis.Natalizumab exerts a suppressive effect on surrogates of B cell function in blood and CSF.Detection and analysis of variants of JC polyomavirus in urine samples from HIV-1-infected patients in China's Zhejiang Province.JC polyomavirus expression and bell-shaped regulation of its SF2/ASF suppressor during the follow-up of multiple sclerosis patients treated with natalizumab.
P2860
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P2860
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@ast
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@en
type
label
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@ast
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@en
prefLabel
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@ast
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@en
P2093
P2860
P356
P1433
P1476
JC virus reactivation during prolonged natalizumab monotherapy for multiple sclerosis.
@en
P2093
Carolina Ionete
Evelyn Bord
Guy J Buckle
Igor J Koralnik
Maria K Houtchens
Marion C Stein
Maureen Donnelly
R Philip Kinkel
Spyridon Chalkias
Stephanie Batson
P2860
P304
P356
10.1002/ANA.24148
P577
2014-06-10T00:00:00Z