Evidence that cagA(+) Helicobacter pylori strains are disappearing more rapidly than cagA(-) strains. - Wikidata - wikimedia - TriplyDB

Evidence that cagA(+) Helicobacter pylori strains are disappearing more rapidly than cagA(-) strains.

Evidence that cagA(+) Helicobacter pylori strains are disappearing more rapidly than cagA(-) strains.

http://www.wikidata.org/entity/Q35594455

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Helicobacter pylori persistence: biology and disease Polymorphism in the Helicobacter pylori CagA and VacA toxins and disease Early-life family structure and microbially induced cancer risk Consequences of Helicobacter pylori infection in children.Asthma is inversely associated with Helicobacter pylori status in an urban population.Blood pressure and stature in Helicobacter pylori positive and negative persons.Longitudinal analysis of serological responses of adults to Helicobacter pylori antigens.Exposure to Helicobacter pylori infection in early childhood and the risk of allergic disease and atopic sensitization: a longitudinal birth cohort study.Etiology of pancreatic cancer, with a hypothesis concerning the role of N-nitroso compounds and excess gastric acidity.Specific serum immunoglobulin G to H pylori and CagA in healthy children and adults (south-east of Iran)East Asian genotypes of Helicobacter pylori strains in Amerindians provide evidence for its ancient human carriage.Helicobacter pylori colonization ameliorates glucose homeostasis in mice through a PPAR γ-dependent mechanism.Theodore E. Woodward Award: Global warming and the human stomach: microecology follows macroecology.Helicobacter pylori seropositivities and risk of pancreatic carcinoma Association between Helicobacter pylori and pancreatic cancer risk: a meta-analysis.Does Helicobacter pylori protect against asthma and allergy?Helicobacter pylori colonization is inversely associated with childhood asthma.Epidemiology and diagnosis of Helicobacter pylori infection.Concordance of Helicobacter pylori strains within families.Transient and persistent Helicobacter pylori colonization in Native American children.Role of childhood infection in the sequelae of H. pylori disease Helicobacter pylori and the birth cohort effect: evidence for stabilized colonization rates in childhood.Helicobacter pylori modulates host cell survival regulation through the serine-threonine kinase, 3-phosphoinositide dependent kinase 1 (PDK-1).Intergenerational reduction in Helicobacter pylori prevalence is similar between different ethnic groups living in a Western city Association between gastric Helicobacter pylori colonization and glycated hemoglobin levels.Plasticity of repetitive DNA sequences within a bacterial (Type IV) secretion system component.Serologic host response to Helicobacter pylori and Campylobacter jejuni in socially housed Rhesus macaques (Macaca mulatta).Helicobacter pylori and esophageal cancer risk: a meta-analysis.Helicobacter pylori induces beta3GnT5 in human gastric cell lines, modulating expression of the SabA ligand sialyl-Lewis x Helicobacter pylori in health and disease.Do you have a probiotic in your future?Ethnicity is a strong predictor for Helicobacter pylori infection in young women in a multi-ethnic European city Association between Helicobacter pylori and mortality in the NHANES III study.Helicobacter pylori infection affects immune responses following vaccination of typhoid-naive U.S. adults with attenuated Salmonella typhi oral vaccine CVD 908-htrA.The role of Helicobacter pylori infection in the development of allergic asthma.Characterization of the Cag pathogenicity island in Helicobacter pylori from naturally infected rhesus macaques.The prevalence of Helicobacter pylori infection in the Netherlands.Effects of Helicobacter pylori, geohelminth infection and selected commensal bacteria on the risk of allergic disease and sensitization in 3-year-old Ethiopian children.Identification of Helicobacter pylori Strain cagPAI+ and cagPAI− Antigens by IgG Antibodies from Sera of Experimentally Colonized Meriones unguiculatus (Mongolian gerbils)

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P2860

Evidence that cagA(+) Helicobacter pylori strains are disappearing more rapidly than cagA(-) strains.

http://www.wikidata.org/entity/Q35594455

description

2002 nî lūn-bûn

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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Evidence that cagA(+) Helicoba ...... rapidly than cagA(-) strains.

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P2860

Risk for gastric cancer in people with CagA positive or CagA negative Helicobacter pylori infection.

Variants of the 3' region of the cagA gene in Helicobacter pylori isolates from patients with different H. pylori-associated diseases

Distinct variants of Helicobacter pylori cagA are associated with vacA subtypes.

Colonization of Mexican patients by multiple Helicobacter pylori strains with different vacA and cagA genotypes.

Genotypic, clinical, and demographic characteristics of children infected with Helicobacter pylori.

Helicobacter pylori infection rates in relation to age and social class in a population of Welsh men.

Translocation of Helicobacter pylori CagA into gastric epithelial cells by type IV secretion.

The epidemiology of Helicobacter pylori infection.

Antigenic characterization of Helicobacter pylori strains from different parts of the world

Helicobacters are indigenous to the human stomach: duodenal ulceration is due to changes in gastric microecology in the modern era.

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Helicobacter pylori

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