Tumor necrosis factor-induced reversal of adipocytic phenotype of 3T3-L1 cells is preceded by a loss of nuclear CCAAT/enhancer binding protein (C/EBP).
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Identification of novel stress-induced genes downstream of chopCCAAT/enhancer-binding proteins: structure, function and regulationTumor necrosis factor alpha inhibits type I collagen synthesis through repressive CCAAT/enhancer-binding proteinsModulation of the transcriptional activity of peroxisome proliferator-activated receptor gamma by protein-protein interactions and post-translational modificationsCytokines suppress adipogenesis and PPAR-gamma function through the TAK1/TAB1/NIK cascadeA novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOPNrf2 protects against 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced oxidative injury and steatohepatitisStress-induced binding of the transcriptional factor CHOP to a novel DNA control elementCHOP-Dependent stress-inducible expression of a novel form of carbonic anhydrase VIProfiling of chicken adipose tissue gene expression by genome arrayPeroxisome proliferator-activated receptor-gamma: a versatile metabolic regulator.Adipose tissue-specific dysregulation of angiotensinogen by oxidative stress in obesity.Molecular genetics of chromosome translocations involving EWS and related family members.Inhibition of glyceroneogenesis by histone deacetylase 3 contributes to lipodystrophy in mice with adipose tissue inflammation.Analysis of transcription factor network underlying 3T3-L1 adipocyte differentiation.The expression of adipogenic genes is decreased in obesity and diabetes mellitusInducible translational regulation of the NF-IL6 transcription factor by respiratory syncytial virus infection in pulmonary epithelial cells.C/EBPbeta is a negative regulator of human papillomavirus type 11 in keratinocytes.Regulatory elements and transcription factors controlling basal and cytokine-induced expression of the gene encoding intercellular adhesion molecule 1.Adipose tissue and inflammatory bowel disease pathogenesisAdiponectin as an anti-fibrotic and anti-inflammatory adipokine in the liver.Tumor necrosis factor-induced c-myc expression in the absence of mitogenesis is associated with inhibition of adipocyte differentiation.A novel cis-acting element controlling the rat CYP2D5 gene and requiring cooperativity between C/EBP beta and an Sp1 factor.Transcriptional and post-translational regulation of adiponectin.C/EBP-related sites in addition to a STAT site are necessary for ciliary neurotrophic factor-leukemia inhibitory factor-dependent transcriptional activation by the vasoactive intestinal peptide cytokine response element.Insulin regulates transcription of the CCAAT/enhancer binding protein (C/EBP) alpha, beta, and delta genes in fully-differentiated 3T3-L1 adipocytes.Impaired cyclic AMP-dependent phosphorylation renders CREB a repressor of C/EBP-induced transcription of the somatostatin gene in an insulinoma cell line.Transcription of the rat glucagon gene by the cyclic AMP response element-binding protein CREB is modulated by adjacent CREB-associated proteins.Acute-phase response factor, a nuclear factor binding to acute-phase response elements, is rapidly activated by interleukin-6 at the posttranslational level.Alpha-lipoic acid inhibits adipocyte differentiation by regulating pro-adipogenic transcription factors via mitogen-activated protein kinase pathways.Cross-Talk between PPARgamma and Insulin Signaling and Modulation of Insulin Sensitivity.Control of adipocyte differentiation.Inhibition of adipogenesis by the stress-induced protein CHOP (Gadd153).Negative regulation of human fibrinogen gene expression by peroxisome proliferator-activated receptor alpha agonists via inhibition of CCAAT box/enhancer-binding protein beta.Regulation of adipogenesis by a transcriptional repressor that modulates MAPK activation.Specific inhibitors of p38 mitogen-activated protein kinase block 3T3-L1 adipogenesis.Adipocyte hormone-sensitive lipase: a major regulator of lipid metabolism.Plasminogen activator inhibitor-1 modulates adipocyte differentiation.GO6976 prevents TNF-alpha-induced suppression of adiponectin expression in 3T3-L1 adipocytes: putative involvement of protein kinase C.Opposing effects of tumour necrosis factor alpha and hyperosmolarity on Na+/myo-inositol co-transporter mRNA levels and myo-inositol accumulation by 3T3-L1 adipocytes.
P2860
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P2860
Tumor necrosis factor-induced reversal of adipocytic phenotype of 3T3-L1 cells is preceded by a loss of nuclear CCAAT/enhancer binding protein (C/EBP).
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@ast
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@en
type
label
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@ast
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@en
prefLabel
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@ast
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@en
P2093
P2860
P356
P1476
Tumor necrosis factor-induced ...... ancer binding protein (C/EBP).
@en
P2093
A R Brasier
J F Habener
R E McGehee
P2860
P304
P356
10.1172/JCI115566
P407
P50
P577
1992-01-01T00:00:00Z