Interleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammation
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Immune-responsive gene 1 protein links metabolism to immunity by catalyzing itaconic acid productionMicroRNA-146a provides feedback regulation of lyme arthritis but not carditis during infection with Borrelia burgdorferi.Different patterns of expression and of IL-10 modulation of inflammatory mediators from macrophages of Lyme disease-resistant and -susceptible mice.Borrelia burgdorferi elicited-IL-10 suppresses the production of inflammatory mediators, phagocytosis, and expression of co-stimulatory receptors by murine macrophages and/or dendritic cells.Tick sialostatins L and L2 differentially influence dendritic cell responses to Borrelia spirochetes.Flavonoid naringenin: a potential immunomodulator for Chlamydia trachomatis inflammation.Role of interleukin 6 in innate immunity to Mycobacterium tuberculosis infection.Suppression of IRG-1 Reduces Inflammatory Cell Infiltration and Lung Injury in Respiratory Syncytial Virus Infection by Reducing Production of Reactive Oxygen Species.Assessment of transcriptional activity of Borrelia burgdorferi and host cytokine genes during early and late infection in a mouse modelCerebrospinal fluid cytokines in Lyme neuroborreliosis.The Potential of Omics Technologies in Lyme Disease Biomarker Discovery and Early Detection.Spirochetal Lipoproteins and Immune Evasion.Anti-inflammatory effects of silver-polyvinyl pyrrolidone (Ag-PVP) nanoparticles in mouse macrophages infected with live Chlamydia trachomatis.Lyme disease: aetiopathogenesis, factors for disease development and control.The Cross-Talk between Spirochetal Lipoproteins and Immunity.CD4+ cell-derived interleukin-17 in a model of dysregulated, Borrelia-induced arthritis.Cutaneous Lyme borreliosis: Guideline of the German Dermatology Society.Th1 and Th17 but no Th2-related cytokine spectrum in the cerebrospinal fluid of children with Borrelia-related facial nerve palsy.Interleukin-10 (IL-10) inhibits Borrelia burgdorferi-induced IL-17 production and attenuates IL-17-mediated Lyme arthritis.Induction of IL-10 by Borrelia burgdorferi is regulated by the action of CD14-dependent p38-MAPK and cAMP-mediated chromatin remodeling.A multi-omic analysis reveals the regulatory role of CD180 during the response of macrophages to Borrelia burgdorferi.Leptospirosis Pathophysiology: Into the Storm of Cytokines.
P2860
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P2860
Interleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammation
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Interleukin-10 alters effector ...... late Lyme disease inflammation
@ast
Interleukin-10 alters effector ...... late Lyme disease inflammation
@en
type
label
Interleukin-10 alters effector ...... late Lyme disease inflammation
@ast
Interleukin-10 alters effector ...... late Lyme disease inflammation
@en
prefLabel
Interleukin-10 alters effector ...... late Lyme disease inflammation
@ast
Interleukin-10 alters effector ...... late Lyme disease inflammation
@en
P2093
P2860
P921
P356
P1476
Interleukin-10 alters effector ...... late Lyme disease inflammation
@en
P2093
Aarti Gautam
Deepak Kaushal
Lisa A Morici
Mario T Philipp
Saurabh Dixit
Vida A Dennis
P2860
P304
P356
10.1128/IAI.05451-11
P407
P577
2011-09-26T00:00:00Z