SagS contributes to the motile-sessile switch and acts in concert with BfiSR to enable Pseudomonas aeruginosa biofilm formation
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'Big things in small packages: the genetics of filamentous phage and effects on fitness of their host'BdlA, DipA and induced dispersion contribute to acute virulence and chronic persistence of Pseudomonas aeruginosaThe phosphodiesterase DipA (PA5017) is essential for Pseudomonas aeruginosa biofilm dispersionNO-induced biofilm dispersion in Pseudomonas aeruginosa is mediated by an MHYT domain-coupled phosphodiesteraseThe Pseudomonas aeruginosa diguanylate cyclase GcbA, a homolog of P. fluorescens GcbA, promotes initial attachment to surfaces, but not biofilm formation, via regulation of motilityThe diguanylate cyclase GcbA facilitates Pseudomonas aeruginosa biofilm dispersion by activating BdlABrlR from Pseudomonas aeruginosa is a c-di-GMP-responsive transcription factorElevated levels of the second messenger c-di-GMP contribute to antimicrobial resistance of Pseudomonas aeruginosa.A starvation-induced regulator, RovM, acts as a switch for planktonic/biofilm state transition in Yersinia pseudotuberculosis.Histidine-containing phosphotransfer protein-B (HptB) regulates swarming motility through partner-switching system in Pseudomonas aeruginosa PAO1 strain.Sticky situations: key components that control bacterial surface attachmentThe Diguanylate Cyclase HsbD Intersects with the HptB Regulatory Cascade to Control Pseudomonas aeruginosa Biofilm and MotilityAn Oxygen-Sensing Two-Component System in the Burkholderia cepacia Complex Regulates Biofilm, Intracellular Invasion, and Pathogenicity.Dispersion by Pseudomonas aeruginosa requires an unusual posttranslational modification of BdlA.Microcolony formation by the opportunistic pathogen Pseudomonas aeruginosa requires pyruvate and pyruvate fermentation.A dynamic and intricate regulatory network determines Pseudomonas aeruginosa virulence.Escaping the biofilm in more than one way: desorption, detachment or dispersion.Antimicrobial tolerance of Pseudomonas aeruginosa biofilms is activated during an early developmental stage and requires the two-component hybrid SagS.Advances in understanding PseudomonasBroth versus Surface-Grown Cells: Differential Regulation of RsmY/Z Small RNAs in Pseudomonas aeruginosa by the Gac/HptB System.Efficient surface modification of biomaterial to prevent biofilm formation and the attachment of microorganisms.Small RNAs and their role in biofilm formationPseudomonas aeruginosa biofilm infections: from molecular biofilm biology to new treatment possibilities.New mechanistic insights into the motile-to-sessile switch in various bacteria with particular emphasis on Bacillus subtilis and Pseudomonas aeruginosa: a review.Quick change: post-transcriptional regulation in Pseudomonas.Targeting c-di-GMP Signaling, Biofilm Formation, and Bacterial Motility with Small Molecules.Two-component systems required for virulence in Pseudomonas aeruginosa.Role of psl Genes in Antibiotic Tolerance of Adherent Pseudomonas aeruginosa.Pseudomonas aeruginosa Genome Evolution in Patients and under the Hospital Environment.Diguanylate cyclase NicD-based signalling mechanism of nutrient-induced dispersion by Pseudomonas aeruginosa.A Cyclic di-GMP-binding Adaptor Protein Interacts with Histidine Kinase to Regulate Two-component Signaling.Interconnection of post-transcriptional regulation: The RNA-binding protein Hfq is a novel target of the Lon protease in Pseudomonas aeruginosa.Pleiotropic effects of acyltransferases on various virulence-related phenotypes of Pseudomonas aeruginosa.PqsR-dependent and PqsR-independent regulation of motility and biofilm formation by PQS in Pseudomonas aeruginosa PAO1.Divide and conquer: the Pseudomonas aeruginosa two-component hybrid SagS enables biofilm formation and recalcitrance of biofilm cells to antimicrobial agents via distinct regulatory circuits.The ABC of biofilm drug tolerance: The MerR-like regulator BrlR is an activator of ABC transport systems, with PA1874-77 contributing to the tolerance by Pseudomonas aeruginosa biofilms to tobramycin.Susceptibility of Pseudomonas aeruginosa Dispersed Cells to Antimicrobial Agents Is Dependent on the Dispersion Cue and Class of the Antimicrobial Agent Used.Options and Limitations in Clinical Investigation of Bacterial Biofilms.High-throughput detection of RNA processing in bacteria.The Yin and Yang of SagS: Distinct Residues in the HmsP Domain of SagS Independently Regulate Biofilm Formation and Biofilm Drug Tolerance.
P2860
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P2860
SagS contributes to the motile-sessile switch and acts in concert with BfiSR to enable Pseudomonas aeruginosa biofilm formation
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
SagS contributes to the motile ...... s aeruginosa biofilm formation
@ast
SagS contributes to the motile ...... s aeruginosa biofilm formation
@en
type
label
SagS contributes to the motile ...... s aeruginosa biofilm formation
@ast
SagS contributes to the motile ...... s aeruginosa biofilm formation
@en
prefLabel
SagS contributes to the motile ...... s aeruginosa biofilm formation
@ast
SagS contributes to the motile ...... s aeruginosa biofilm formation
@en
P2860
P356
P1476
SagS contributes to the motile ...... s aeruginosa biofilm formation
@en
P2093
Karin Sauer
Olga E Petrova
P2860
P304
P356
10.1128/JB.00305-11
P407
P577
2011-09-23T00:00:00Z