Tumor necrosis factor-alpha-mediated decrease in glutathione increases the sensitivity of pulmonary vascular endothelial cells to H2O2
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Overexpression of gamma-glutamylcysteine synthetase suppresses tumor necrosis factor-induced apoptosis and activation of nuclear transcription factor-kappa B and activator protein-1Filovirus-induced endothelial leakage triggered by infected monocytes/macrophagesMolecular mechanism of decreased glutathione content in human immunodeficiency virus type 1 Tat-transgenic miceThe role of reactive oxygen and nitrogen species in the response of airway epithelium to particulatesEffects of tumor necrosis factor alpha on sin nombre virus infection in vitro.PI3K/Akt and mTOR/p70S6K pathways mediate neuroprotectin D1-induced retinal pigment epithelial cell survival during oxidative stress-induced apoptosis.Proteomic analysis of endothelial cold-adaptationEffect of bone marrow-derived mesenchymal stem cells on endotoxin-induced oxidation of plasma cysteine and glutathione in miceRole of TNFalpha in pulmonary pathophysiology.Chronic hypoxia promotes pulmonary artery endothelial cell proliferation through H2O2-induced 5-lipoxygenase.Human hsp27, Drosophila hsp27 and human alphaB-crystallin expression-mediated increase in glutathione is essential for the protective activity of these proteins against TNFalpha-induced cell deathInterleukin-6 counteracts therapy-induced cellular oxidative stress in multiple myeloma by up-regulating manganese superoxide dismutaseResults of and further prevention of hypoxic fetal brain damage by inhibition of xanthine oxidase enzyme with allopurinol.Influence of nutrients and cytokines on endothelial cell metabolism.Possible involvement of reactive oxygen species in D-galactosamine-induced sensitization against tumor necrosis factor-alpha-induced hepatocyte apoptosis.Neutrophil-derived Oxidants and Proteinases as Immunomodulatory Mediators in Inflammation.Mechanism for the increased permeability in endothelial monolayers induced by elastase.Pulmonary granulocyte influx and impaired alveolar macrophage adenylyl cyclase responsiveness in developing respiratory distress.Immunomodulatory role of phagocyte-derived chloramines involving lymphocyte glutathione.Brain vessels normally undergo cyclic activation and inactivation: evidence from tumor necrosis factor-alpha, heme oxygenase-1, and manganese superoxide dismutase immunostaining of vessels and perivascular brain cells.N-Acetylcysteine Attenuates the Development of Renal Fibrosis in Transgenic Mice with Dilated Cardiomyopathy.In-vitro uptake of vitamin A by Plasmodium falciparum.Mitochondrial thioredoxin-2 has a key role in determining tumor necrosis factor-alpha-induced reactive oxygen species generation, NF-kappaB activation, and apoptosis.Protein kinase C-alpha mediates endothelial barrier dysfunction induced by TNF-alpha.Ginsenoside Rg3 attenuated omethoate-induced lung injury in rats.The Role of Cytokines in the Production of Prostacyclin and Thromboxane in Human Mononuclear Cells
P2860
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P2860
Tumor necrosis factor-alpha-mediated decrease in glutathione increases the sensitivity of pulmonary vascular endothelial cells to H2O2
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@ast
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@en
type
label
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@ast
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@en
prefLabel
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@ast
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@en
P2093
P2860
P356
P1476
Tumor necrosis factor-alpha-me ...... ular endothelial cells to H2O2
@en
P2093
C A Partridge
P J Del Vecchio
P2860
P304
P356
10.1172/JCI115658
P407
P577
1992-03-01T00:00:00Z