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Exercise training improves systolic function in hypertensive myocardium

Exercise training improves systolic function in hypertensive myocardium

http://www.wikidata.org/entity/Q35601649

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Effects of hypertension and exercise on cardiac proteome remodelling Exercise performed around MLSS decreases systolic blood pressure and increases aerobic fitness in hypertensive rats.Heart failure with preserved ejection fraction: chronic low-intensity interval exercise training preserves myocardial O2 balance and diastolic function.Effect of Moderate Exercise on Mitochondrial Proteome in Heart Tissue of Spontaneous Hypertensive Rats.Is exercise really deleterious for the hypertensive heart?Effects of acute angiotensin II on ischemia reperfusion injury following myocardial infarction.Adverse cardiac remodelling in spontaneously hypertensive rats: acceleration by high aerobic exercise intensity.Monitoring chronic physical stress using biomarkers, performance protocols and mathematical functions to identify physiological adaptations in rats.Physical Exercise and Regulation of Intracellular Calcium in Cardiomyocytes of Hypertensive Rats Cardiac Effects of Exercise Training in Hypertension

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Exercise training improves systolic function in hypertensive myocardium

http://www.wikidata.org/entity/Q35601649

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

2011年论文

@zh

2011年论文

@zh-cn

name

Exercise training improves systolic function in hypertensive myocardium

@ast

Exercise training improves systolic function in hypertensive myocardium

@en

type

label

Exercise training improves systolic function in hypertensive myocardium

@ast

Exercise training improves systolic function in hypertensive myocardium

@en

prefLabel

Exercise training improves systolic function in hypertensive myocardium

@ast

Exercise training improves systolic function in hypertensive myocardium

@en

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JAPPLPHYSIOL.00292.2011

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Journal of Applied Physiology

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Exercise training improves systolic function in hypertensive myocardium

@en

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Abdelkarim Sabri

http://www.w3.org/2001/XMLSchema#string

Brian F Renna

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Canhua Xiao

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Joseph R Libonati

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Scott M Macdonnell

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P2860

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Ca2+-induced apoptosis through calcineurin dephosphorylation of BAD

The Seventh Report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure: the JNC 7 report

A conserved XIAP-interaction motif in caspase-9 and Smac/DIABLO regulates caspase activity and apoptosis

Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

Regulation of cell death protease caspase-9 by phosphorylation

IAP proteins: blocking the road to death's door

Akt/Protein kinase B inhibits cell death by preventing the release of cytochrome c from mitochondria

Calcineurin/NFAT coupling participates in pathological, but not physiological, cardiac hypertrophy.

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scholarly article

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10.1152/JAPPLPHYSIOL.00292.2011

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