Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia. - Wikidata - wikimedia - TriplyDB

Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia.

Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia.

http://www.wikidata.org/entity/Q35606725

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Opioids and Viral Infections: A Double-Edged Sword Interactions of Opioids and HIV Infection in the Pathogenesis of Chronic Pain Synergistic cooperation between methamphetamine and HIV-1 gsp120 through the P13K/Akt pathway induces IL-6 but not IL-8 expression in astrocytes Morphine enhances HIV-1SF162-mediated neuron death and delays recovery of injured neurites HIV-1 alters neural and glial progenitor cell dynamics in the central nervous system: coordinated response to opiates during maturation.Interactive HIV-1 Tat and morphine-induced synaptodendritic injury is triggered through focal disruptions in Na⁺ influx, mitochondrial instability, and Ca²⁺ overload.Rat nucleus accumbens core astrocytes modulate reward and the motivation to self-administer ethanol after abstinence HIV transactivator of transcription enhances methamphetamine-induced Parkinson's-like behavior in the rats.Ibudilast (AV411), and its AV1013 analog, reduce HIV-1 replication and neuronal death induced by HIV-1 and morphine Interactions of HIV and drugs of abuse: the importance of glia, neural progenitors, and host genetic factors.Bivalent ligands targeting chemokine receptor dimerization: molecular design and functional studies.Sensitization of enteric neurons to morphine by HIV-1 Tat protein.Opiate addiction therapies and HIV-1 Tat: interactive effects on glial [Ca²⁺]i, oxyradical and neuroinflammatory chemokine production and correlative neurotoxicity.Fractalkine/CX3CL1 protects striatal neurons from synergistic morphine and HIV-1 Tat-induced dendritic losses and death Differing roles of autophagy in HIV-associated neurocognitive impairment and encephalitis with implications for morphine co-exposure Morphine and gp120 toxic interactions in striatal neurons are dependent on HIV-1 strain.Oligodendrocytes Are Targets of HIV-1 Tat: NMDA and AMPA Receptor-Mediated Effects on Survival and Development.Morphine efficacy is altered in conditional HIV-1 Tat transgenic mice.Opiate drug use and the pathophysiology of neuroAIDS.Morphine Tolerance and Physical Dependence Are Altered in Conditional HIV-1 Tat Transgenic Mice.Mechanism(s) involved in opioid drug abuse modulation of HAND Morphine exposure during HIV encephalitis in SCID mice.A Bivalent Ligand Targeting the Putative Mu Opioid Receptor and Chemokine Receptor CCR5 Heterodimers: Binding Affinity versus Functional Activities.5α-reduced progestogens ameliorate mood-related behavioral pathology, neurotoxicity, and microgliosis associated with exposure to HIV-1 Tat.Differential expression and HIV-1 regulation of μ-opioid receptor splice variants across human central nervous system cell types.HIV-1 Tat Inhibits Autotaxin Lysophospholipase D Activity and Modulates Oligodendrocyte Differentiation Inhibition of GABAergic Neurotransmission by HIV-1 Tat and Opioid Treatment in the Striatum Involves μ-Opioid Receptors.Ligand-gated purinergic receptors regulate HIV-1 Tat and morphine related neurotoxicity in primary mouse striatal neuron-glia co-cultures.Productive infection of human neural progenitor cells by R5 tropic HIV-1: opiate co-exposure heightens infectivity and functional vulnerability.Neuronal toxicity in HIV CNS disease Development of in vivo drug-induced neurotoxicity models.Atg5- and Atg7-dependent autophagy in dopaminergic neurons regulates cellular and behavioral responses to morphine.GSK3β-activation is a point of convergence for HIV-1 and opiate-mediated interactive neurotoxicity Importance of Autophagy in Mediating Human Immunodeficiency Virus (HIV) and Morphine-Induced Metabolic Dysfunction and Inflammation in Human Astrocytes.Endocannabinoids exert CB1 receptor-mediated neuroprotective effects in models of neuronal damage induced by HIV-1 Tat protein.Exploration of bivalent ligands targeting putative mu opioid receptor and chemokine receptor CCR5 dimerization.Microglia and drug-induced plasticity in reward-related neuronal circuits.Central HIV-1 Tat exposure elevates anxiety and fear conditioned responses of male mice concurrent with altered mu-opioid receptor-mediated G-protein activation and β-arrestin 2 activity in the forebrain.The effect of various morphine weaning regimens on the sequelae of opioid tolerance involving physical dependency, anxiety and hippocampus cell neurodegeneration in rats.Long-term morphine delivery via slow release morphine pellets or osmotic pumps: Plasma concentration, analgesia, and naloxone-precipitated withdrawal.

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Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia.

http://www.wikidata.org/entity/Q35606725

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Morphine potentiates neurodege ...... ioid receptor-expressing glia.

@ast

Morphine potentiates neurodege ...... ioid receptor-expressing glia.

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type

label

Morphine potentiates neurodege ...... ioid receptor-expressing glia.

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Morphine potentiates neurodege ...... ioid receptor-expressing glia.

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prefLabel

Morphine potentiates neurodege ...... ioid receptor-expressing glia.

@ast

Morphine potentiates neurodege ...... ioid receptor-expressing glia.

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Morphine potentiates neurodege ...... ioid receptor-expressing glia.

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Kurt F Hauser

http://www.w3.org/2001/XMLSchema#string

Nazira El-Hage

http://www.w3.org/2001/XMLSchema#string

Pamela E Knapp

http://www.w3.org/2001/XMLSchema#string

Sandra P Welch

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Shiping Zou

http://www.w3.org/2001/XMLSchema#string

Yun-Kyung Hahn

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P2860

The Tat protein of human immunodeficiency virus type 1, a growth factor for AIDS Kaposi sarcoma and cytokine-activated vascular cells, induces adhesion of the same cell types by using integrin receptors recognizing the RGD amino acid sequence

Expression of CX3CR1 chemokine receptors on neurons and their role in neuronal survival

Profile of neurologic disorders associated with HIV/AIDS from Bangalore, south India (1989-96)

Neurological manifestations of HIV disease

Interaction of HIV-1 Tat protein with heparin. Role of the backbone structure, sulfation, and size

Effects of human immunodeficiency virus type 1 on astrocyte gene expression and function: potential role in neuropathogenesis

Apoptotic death of striatal neurons induced by human immunodeficiency virus-1 Tat and gp120: Differential involvement of caspase-3 and endonuclease G

HIV and antiretroviral therapy in the brain: neuronal injury and repair

Synergistic neurotoxicity of opioids and human immunodeficiency virus-1 Tat protein in striatal neurons in vitro

Opioid system diversity in developing neurons, astroglia, and oligodendroglia in the subventricular zone and striatum: impact on gliogenesis in vivo

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3616-3631

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scholarly article

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10.1093/BRAIN/AWR281

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P407

P433

Pt 12

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P478

134

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P577

2011-11-18T00:00:00Z

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51816778

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22102648

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neurodegeneration

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3235561

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