PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells - Wikidata - wikimedia - TriplyDB

PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

http://www.wikidata.org/entity/Q35608757

about

Pax-8-PPAR-γ fusion protein in thyroid carcinoma Characterization of thyroid cancer cell lines in murine orthotopic and intracardiac metastasis models.Diabetes and thyroid cancer risk: literature review.Anti- and Protumorigenic Effects of PPARγ in Lung Cancer Progression: A Double-Edged Sword Efatutazone, an oral PPAR-γ agonist, in combination with paclitaxel in anaplastic thyroid cancer: results of a multicenter phase 1 trial.Targeted inhibition of Src kinase with dasatinib blocks thyroid cancer growth and metastasis.PPAR-γ Agonists As Antineoplastic Agents in Cancers with Dysregulated IGF Axis.Thioredoxin interacting protein (TXNIP) is a novel tumor suppressor in thyroid cancer.The thyroid cancer PAX8-PPARG fusion protein activates Wnt/TCF-responsive cells that have a transformed phenotype.FAK Expression, Not Kinase Activity, Is a Key Mediator of Thyroid Tumorigenesis and Protumorigenic Processes.Nuclear factor κB-dependent regulation of angiogenesis, and metastasis in an in vivo model of thyroid cancer is associated with secreted interleukin-8.Familial Cancer Variant Prioritization Pipeline version 2 (FCVPPv2) applied to a papillary thyroid cancer family

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P2860

PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

http://www.wikidata.org/entity/Q35608757

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells

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Bryan R Haugen

http://www.w3.org/2001/XMLSchema#string

Deborah L Fretwell

http://www.w3.org/2001/XMLSchema#string

Kevin T Bauerle

http://www.w3.org/2001/XMLSchema#string

Laura A Pike

http://www.w3.org/2001/XMLSchema#string

Qiong Zhou

http://www.w3.org/2001/XMLSchema#string

Vibha Sharma

http://www.w3.org/2001/XMLSchema#string

William M Wood

http://www.w3.org/2001/XMLSchema#string

P2860

Reactivation of suppressed RhoB is a critical step for the inhibition of anaplastic thyroid cancer growth

Expression of the G2-M checkpoint regulators cyclin B1 and cdc2 in nonmalignant and malignant human breast lesions: immunocytochemical and quantitative image analyses

Cyclin A in cell cycle control and cancer

Troglitazone, the peroxisome proliferator-activated receptor-gamma agonist, induces antiproliferation and redifferentiation in human thyroid cancer cell lines

The PPARs: from orphan receptors to drug discovery

Inhibition of cell motility by troglitazone in human ovarian carcinoma cell line.

Management of undifferentiated thyroid cancer.

The roles of PPARs in adipocyte differentiation.

Terminal differentiation of human liposarcoma cells induced by ligands for peroxisome proliferator-activated receptor gamma and the retinoid X receptor.

The Putative PAX8/PPARγ Fusion Oncoprotein Exhibits Partial Tumor Suppressor Activity through Up-Regulation of Micro-RNA-122 and Dominant-Negative PPARγ Activity

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171765

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scholarly article

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10.1155/2011/171765

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2011

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Rebecca Schweppe

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2011-12-12T00:00:00Z

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51920850

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22194735

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3236353

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