Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
about
NF-κB induces miR-148a to sustain TGF-β/Smad signaling activation in glioblastoma.Developmental Coordination of Gene Expression between Synaptic Partners During GABAergic Circuit Assembly in Cerebellar Cortex.The QKI-5 and QKI-6 RNA binding proteins regulate the expression of microRNA 7 in glial cells.Oligodendroglial defects during quakingviable cerebellar development.Gene Expression of Quaking in Sporadic Alzheimer's Disease Patients is Both Upregulated and Related to Expression Levels of Genes Involved in Amyloid Plaque and Neurofibrillary Tangle Formation.
P2860
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
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name
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@ast
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@en
type
label
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@ast
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@en
prefLabel
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@ast
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@en
P2860
P356
P1433
P1476
Loss of p53 in quaking viable mice leads to Purkinje cell defects and reduced survival.
@en
P2093
Christina Gavino
Stéphane Richard
P2860
P2888
P356
10.1038/SREP00084
P407
P577
2011-09-06T00:00:00Z
P5875
P6179
1019974397