Glutathione preconditioning ameliorates mitochondria dysfunction during warm pulmonary ischemia-reperfusion injury

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2012 nî lūn-bûn

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name

Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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P1476

Glutathione preconditioning am ...... ry ischemia-reperfusion injury

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P2093

Bernhard Gohrbandt

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Bhanu Sinha

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Christoph Otto

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Daniel Walter

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Ivan Aleksic

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Rainer G Leyh

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Sebastian-Patrick Sommer

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Stefanie Sommer

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P2860

Involvement of mitochondrial permeability transition, glutathione status, pentose phosphate pathway and oxidative damage in the protective effect of fasting on the ischaemic-reperfused rat heart

Mitochondrial depolarization underlies delay in permeability transition by preconditioning with isoflurane: roles of ROS and Ca2+.

Glutathione peroxidase 1 protects mitochondria against hypoxia/reoxygenation damage in mouse hearts.

Mechanism of superoxide and hydrogen peroxide formation by fumarate reductase, succinate dehydrogenase, and aspartate oxidase.

Mitochondria as decision-makers in cell death.

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

Cardiac arrhythmias induced by glutathione oxidation can be inhibited by preventing mitochondrial depolarization.

Glutathione redox status in mitochondria and cytoplasm differentially and sequentially activates apoptosis cascade in dopamine-melanin-treated SH-SY5Y cells.

Role of calcium and cyclophilin D in the regulation of mitochondrial permeabilization induced by glutathione depletion.

Glutathione improves the function of porcine pulmonary grafts stored for twenty-four hours in low-potassium dextran solution.

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140-8; discussion 148

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scholarly article

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10.1016/J.EJCTS.2011.02.081

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2012-01-01T00:00:00Z

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P5875

51150435

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P698

21596579

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P921

reperfusion injury

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3241131

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