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Caveolin-1, cellular senescence and age-related diseases.

Caveolin-1, cellular senescence and age-related diseases.

http://www.wikidata.org/entity/Q35626889

about

Redox mechanisms in age-related lung fibrosis ROS, Cell Senescence, and Novel Molecular Mechanisms in Aging and Age-Related Diseases Increased Oxidative and Nitrative Stress Accelerates Aging of the Retinal Vasculature in the Diabetic Retina The Role of Caveolin 1 in HIV Infection and Pathogenesis.Berberine suppresses gero-conversion from cell cycle arrest to senescence.Affinity-Guided Design of Caveolin-1 Ligands for Deoligomerization.Cyclooxygenase-2 inhibitors modulate skin aging in a catalytic activity-independent manner Biochemical markers of aging for longitudinal studies in humans.Increased caveolin-1 in intervertebral disc degeneration facilitates repair Effects of Aging and Hypercholesterolemia on Oxidative Stress and DNA Damage in Bone Marrow Mononuclear Cells in Apolipoprotein E-deficient Mice.Loss of Epithelial Membrane Protein 2 Aggravates Podocyte Injury via Upregulation of Caveolin-1.Blood CD33(+)HLA-DR(-) myeloid-derived suppressor cells are increased with age and a history of cancer Loss of caveolin-1 expression in knock-in mouse model of Huntington's disease suppresses pathophysiology in vivo.Caveolin and TGF-β entanglements.Altered TGF-β signaling in fetal fibroblasts: what is known about the underlying mechanisms?The role of caveolae in the pathophysiology of lung diseases.The Natural Polyphenol Epigallocatechin Gallate Protects Intervertebral Disc Cells from Oxidative Stress.Caveolin-1 is involved in high glucose accelerated human glomerular mesangial cell senescence.Differential responses of choroidal melanocytes and uveal melanoma cells to low oxygen conditions.Adverse Effects of Diabetes Mellitus on the Skeleton of Aging Mice.Caveolin‑1 regulates oxidative stress‑induced senescence in nucleus pulposus cells primarily via the p53/p21 signaling pathway in vitro.Inflammatory cytokines induce caveolin-1/β-catenin signalling in rat nucleus pulposus cell apoptosis through the p38 MAPK pathway.Induction of a Senescence-Like Phenotype in Cultured Human Fetal Microglia During HIV-1 Infection.Prediction and characterization of human ageing-related proteins by using machine learning.Role of Adipose Tissue Endothelial ADAM17 in Age-Related Coronary Microvascular Dysfunction.Caveolin-1 down-regulation is required for Wnt5a-Frizzled 2 signalling in Ha-RasV12 -induced cell transformation.Role of the Inflammation-Autophagy-Senescence Integrative Network in Osteoarthritis.

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P2860

Caveolin-1, cellular senescence and age-related diseases.

http://www.wikidata.org/entity/Q35626889

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Caveolin-1, cellular senescence and age-related diseases.

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Caveolin-1, cellular senescence and age-related diseases.

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Caveolin-1, cellular senescence and age-related diseases.

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Caveolin-1, cellular senescence and age-related diseases.

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Caveolin-1, cellular senescence and age-related diseases.

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Caveolin-1, cellular senescence and age-related diseases.

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J.MAD.2011.11.001

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Mechanisms of Ageing and Development

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Caveolin-1, cellular senescence and age-related diseases.

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P2093

Daniela Volonte

http://www.w3.org/2001/XMLSchema#string

Elena Stoppani

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Ferruccio Galbiati

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Huafei Zou

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P2860

Inhibition of thioredoxin reductase 1 by caveolin 1 promotes stress-induced premature senescence

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Extension of life-span by introduction of telomerase into normal human cells

Thioredoxin reductase

DNA damage signaling and p53-dependent senescence after prolonged beta-interferon stimulation

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Autophagy in cancer associated fibroblasts promotes tumor cell survival: Role of hypoxia, HIF1 induction and NFκB activation in the tumor stromal microenvironment

Oxidative stress in cancer associated fibroblasts drives tumor-stroma co-evolution: A new paradigm for understanding tumor metabolism, the field effect and genomic instability in cancer cells

Senescence of activated stellate cells limits liver fibrosis

The matricellular protein CCN1 induces fibroblast senescence and restricts fibrosis in cutaneous wound healing

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533-542

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scholarly article

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10.1016/J.MAD.2011.11.001

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11-12

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132

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2011-11-12T00:00:00Z

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51815094

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22100852

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3243775

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