Substance P mediates antiapoptotic responses in human colonocytes by Akt activation - Wikidata - wikimedia - TriplyDB

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

http://www.wikidata.org/entity/Q35629395

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Substance P-mediated expression of the pro-angiogenic factor CCN1 modulates the course of colitis Biological and Pharmacological Aspects of the NK1-Receptor Substance P is a mechanoresponsive, autocrine regulator of human tenocyte proliferation A novel role for villin in intestinal epithelial cell survival and homeostasis Substance P reduces TNF-α-induced apoptosis in human tenocytes through NK-1 receptor stimulation Substance P modulates colitis-associated fibrosis Protective effects of Lactobacillus plantarum against epithelial barrier dysfunction of human colon cell line NCM460.Insulin-like growth factor-1 receptor transactivation modulates the inflammatory and proliferative responses of neurotensin in human colonic epithelial cells.Loss of desmocollin-2 confers a tumorigenic phenotype to colonic epithelial cells through activation of Akt/β-catenin signaling MicroRNA-124 regulates STAT3 expression and is down-regulated in colon tissues of pediatric patients with ulcerative colitis.Protective Effect of Neuropeptide Substance P on Bone Marrow Mesenchymal Stem Cells against Apoptosis Induced by Serum Deprivation Serum and colonic mucosal immune markers in irritable bowel syndrome Substance P Inhibits Hyperosmotic Stress-Induced Apoptosis in Corneal Epithelial Cells through the Mechanism of Akt Activation and Reactive Oxygen Species Scavenging via the Neurokinin-1 Receptor Substance P induces cardioprotection in ischemia-reperfusion via activation of AKT.Substance P Induces HO-1 Expression in RAW 264.7 Cells Promoting Switch towards M2-Like Macrophages Substance P mediates pro-inflammatory cytokine release form mesenteric adipocytes in Inflammatory Bowel Disease patients Neurokinin-1 receptor directly mediates glioma cell migration by up-regulation of matrix metalloproteinase-2 (MMP-2) and membrane type 1-matrix metalloproteinase (MT1-MMP).High expression of substance P and its receptor neurokinin-1 receptor in colorectal cancer is associated with tumor progression and prognosis.Effect of a neurokinin-1 receptor antagonist in a rat model of colitis-associated colon cancer The antimicrobial peptide cathelicidin modulates Clostridium difficile-associated colitis and toxin A-mediated enteritis in mice.Transforming Growth Factor Beta 1 Modulates the Functional Expression of the Neurokinin-1 Receptor in Human Keratocytes.A constitutively active form of neurokinin 1 receptor and neurokinin 1 receptor-mediated apoptosis in glioblastomas Substance P promotes expansion of human mesenteric preadipocytes through proliferative and antiapoptotic pathways.Akt-mediated anti-apoptotic effects of substance P in Anti-Fas-induced apoptosis of human tenocytes.Effects of alanyl-glutamine supplementation on the small intestinal mucosa barrier in weaned piglets.Tachykinins and their receptors: contributions to physiological control and the mechanisms of disease.Reviews for immune privilege in the year 2010: immune privilege and infection.Immunolocalization of Substance P and NK-1 Receptor in ADIPOSE Stem Cells.β-Arrestin 1 has an essential role in neurokinin-1 receptor-mediated glioblastoma cell proliferation and G2/M phase transition.The fast track to canonical Wnt signaling in MC3T3-E1 cells protected by substance P against serum deprivation-induced apoptosis.Cathelicidin signaling via the Toll-like receptor protects against colitis in mice.PanIN Neuroendocrine Cells Promote Tumorigenesis via Neuronal Cross-talk.Substance P induces CCN1 expression via histone deacetylase activity in human colonic epithelial cells.Overexpression of membrane metalloendopeptidase inhibits substance P stimulation of cholangiocarcinoma growth.Sensory neuron regulation of gastrointestinal inflammation and bacterial host defence.Cigarette smoke synergizes lipopolysaccharide-induced interleukin-1β and tumor necrosis factor-α secretion from macrophages via substance P-mediated nuclear factor-κB activation.Identification of a novel substance P (SP)-neurokinin-1 receptor (NK-1R) microRNA-221-5p inflammatory network in human colonic epithelial cells.Role of G protein-coupled receptors-microRNA interactions in gastrointestinal pathophysiology.MicroRNA-31-3p Is Involved in Substance P (SP)-Associated Inflammation in Human Colonic Epithelial Cells and Experimental Colitis.Substance P Enhances Keratocyte Migration and Neutrophil Recruitment through Interleukin-8.

P2860

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P2860

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

http://www.wikidata.org/entity/Q35629395

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

@wuu

2007年论文

@zh

2007年论文

@zh-cn

name

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@ast

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@en

type

label

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@ast

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@en

prefLabel

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@ast

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@en

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PNAS.0610664104

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Substance P mediates antiapoptotic responses in human colonocytes by Akt activation

@en

P2093

Charalabos Pothoulakis

http://www.w3.org/2001/XMLSchema#string

Dezheng Zhao

http://www.w3.org/2001/XMLSchema#string

Hon-Wai Koon

http://www.w3.org/2001/XMLSchema#string

Mary P Moyer

http://www.w3.org/2001/XMLSchema#string

Yanai Zhan

http://www.w3.org/2001/XMLSchema#string

P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

An Akt/beta-arrestin 2/PP2A signaling complex mediates dopaminergic neurotransmission and behavior

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

The alpha v beta 3 integrin "vitronectin receptor"

Mechanisms and functions of cell death

Regulation of cell death protease caspase-9 by phosphorylation

Direct regulation of the Akt proto-oncogene product by phosphatidylinositol-3,4-bisphosphate

The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase

Protein kinase B (c-Akt) in phosphatidylinositol-3-OH kinase signal transduction

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2013-2018

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scholarly article

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10.1073/PNAS.0610664104

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6

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104

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6540765

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17264209

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1794289

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