Dysfunctional B-cell activation in cirrhosis resulting from hepatitis C infection associated with disappearance of CD27-positive B-cell population.
about
Primary Sjögren's syndrome is characterized by distinct phenotypic and transcriptional profiles of IgD+ unswitched memory B cellsB lymphocytes in human subcutaneous adipose crown-like structuresEnhanced activation of memory, but not naïve, B cells in chronic hepatitis C virus-infected patients with cryoglobulinemia and advanced liver fibrosisSubversion of the B-cell compartment during parasitic, bacterial, and viral infectionsToll-like receptors in liver fibrosis: cellular crosstalk and mechanismsChronic inflammation, immune escape, and oncogenesis in the liver: a unique neighborhood for novel intersections.Peripheral CD27-CD21- B-cells represent an exhausted lymphocyte population in hepatitis C cirrhosisImmune function biomarker QuantiFERON-monitor is associated with infection risk in cirrhotic patients.Risk factors and outcome of bacterial infections in cirrhosisSpontaneous and therapeutic immune responses in hepatocellular carcinoma: implications for current and future immunotherapies.Immune dysfunction in cirrhosis.Immunotherapy in the treatment and prevention of infection in acute-on-chronic liver failure.Reactivation of Hepatitis C Virus and Its Clinical Outcomes in Patients Treated with Systemic Chemotherapy or Immunosuppressive Therapy.Immune Dysfunction in Cirrhosis.Analysis of HBsAg using high-sensitivity HBsAg assays in hepatitis B virus carriers in whom HBsAg seroclearance was confirmed by conventional assays.Endotoxemia contributes to CD27+ memory B-cell apoptosis via enhanced sensitivity to Fas ligation in patients with Cirrhosis.Intestinal barrier dysfunction in cirrhosis: Current concepts in pathophysiology and clinical implicationsEnvironmental peer pressure: CD4+ T cell help in tolerance and transplantation.Increased serum C-reactive protein and decreased urinary aquaporin 2 levels are predictive of the efficacy of tolvaptan in patients with liver cirrhosis.Advanced cirrhosis drives enhanced B-cell differentiation resulting in hyperglobulinemia.B-cell-associated immune profiles in patients with decompensated cirrhosis.Immunopathogenesis of Hepatitis C Virus Infection.
P2860
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P2860
Dysfunctional B-cell activation in cirrhosis resulting from hepatitis C infection associated with disappearance of CD27-positive B-cell population.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Dysfunctional B-cell activatio ...... 27-positive B-cell population.
@ast
Dysfunctional B-cell activatio ...... 27-positive B-cell population.
@en
type
label
Dysfunctional B-cell activatio ...... 27-positive B-cell population.
@ast
Dysfunctional B-cell activatio ...... 27-positive B-cell population.
@en
prefLabel
Dysfunctional B-cell activatio ...... 27-positive B-cell population.
@ast
Dysfunctional B-cell activatio ...... 27-positive B-cell population.
@en
P2093
P2860
P356
P1433
P1476
Dysfunctional B-cell activatio ...... D27-positive B-cell population
@en
P2093
Erica Carpenter
Hiroyoshi Doi
Kyong-Mi Chang
Robert H Vonderheide
Tara K Iyer
P2860
P304
P356
10.1002/HEP.24689
P407
P577
2012-01-19T00:00:00Z