Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunity. - Wikidata - wikimedia - TriplyDB

Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunity.

Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunity.

http://www.wikidata.org/entity/Q35634065

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The origin and role of innate lymphoid cells in the lung Inflammasome, IL-1 and inflammation in ozone-induced lung injury Type 2 innate lymphoid cells: at the cross-roads in allergic asthma Maintenance of Immune Homeostasis through ILC/T Cell Interactions Linking acute infection to chronic lung disease. The role of IL-33-expressing epithelial progenitor cells Development and function of group 2 innate lymphoid cells The role of type 2 innate lymphoid cells in asthma Innate lymphocyte cells in asthma phenotypes Innate lymphoid cells in the airways Group 2 innate lymphoid cells in the lung Animal models of COPD: What do they tell us?The Role of Lymphocytes in Radiotherapy-Induced Adverse Late Effects in the Lung When Insult Is Added to Injury: Cross Talk between ILCs and Intestinal Epithelium in IBD Apoptosis and engulfment by bronchial epithelial cells. Implications for allergic airway inflammation.Group 2 innate lymphoid cell production of IL-5 is regulated by NKT cells during influenza virus infection Characterization and Quantification of Innate Lymphoid Cell Subsets in Human Lung.Dysregulation of type 2 innate lymphoid cells and TH2 cells impairs pollutant-induced allergic airway responses.Emerging Endotypes of Chronic Rhinosinusitis and Its Application to Precision Medicine.Invariant natural killer T cells recognize a fungal glycosphingolipid that can induce airway hyperreactivity IL-33 mediates multi-walled carbon nanotube (MWCNT)-induced airway hyper-reactivity via the mobilization of innate helper cells in the lung.Aligning mouse models of asthma to human endotypes of disease.Respiratory viral infection, epithelial cytokines, and innate lymphoid cells in asthma exacerbations.Chronic low dose chlorine exposure aggravates allergic inflammation and airway hyperresponsiveness and activates inflammasome pathway.IL-17 in severe asthma. Where do we stand?IL-33 promotes airway remodeling in pediatric patients with severe steroid-resistant asthma.Innate immunity is a key factor for the resolution of inflammation in asthma ICOS:ICOS-ligand interaction is required for type 2 innate lymphoid cell function, homeostasis, and induction of airway hyperreactivity.Group 2 innate lymphoid cell proportions are diminished in young helminth infected children and restored by curative anti-helminthic treatment.Nasal sensitization with ragweed pollen induces local-allergic-rhinitis-like symptoms in mice TNF-α Producing Innate Lymphoid Cells (ILCs) Are Increased in Active Celiac Disease and Contribute to Promote Intestinal Atrophy in Mice.Persistence of asthma requires multiple feedback circuits involving type 2 innate lymphoid cells and IL-33.Porphyromonas gingivalis Gingipain-Dependently Enhances IL-33 Production in Human Gingival Epithelial Cells.Innate lymphoid cells: critical regulators of allergic inflammation and tissue repair in the lung.Aspirin-Exacerbated Respiratory Disease Involves a Cysteinyl Leukotriene-Driven IL-33-Mediated Mast Cell Activation Pathway Sterile inflammation - do innate lymphoid cell subsets play a role?STAT6 regulates natural helper cell proliferation during lung inflammation initiated by Alternaria.Pulmonary Epithelial Cell-Derived Cytokine TGF-β1 Is a Critical Cofactor for Enhanced Innate Lymphoid Cell Function.Obesity enhances Th2 inflammatory response via natural killer T cells in a murine model of allergic asthma.Group 2 innate lymphoid cells mediate ozone-induced airway inflammation and hyperresponsiveness in mice.CD4(+) T-cell subsets and host defense in the lung

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Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunity.

http://www.wikidata.org/entity/Q35634065

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J.JACI.2011.10.036

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The Journal of Allergy and Clinical Immunology

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Dale T Umetsu

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Dirk E Smith

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Hye Young Kim

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Hyun-Hee Lee

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James B Rottman

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Paul B Savage

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Ponpan Matangkasombut

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Rosemarie H DeKruyff

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Srividya Subramanian

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Ya-Jen Chang

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P2860

IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

Systemically dispersed innate IL-13-expressing cells in type 2 immunity

Nuocytes represent a new innate effector leukocyte that mediates type-2 immunity

IL25 elicits a multipotent progenitor cell population that promotes T(H)2 cytokine responses

A novel IL-1 family cytokine, IL-33, potently activates human eosinophils

Resolution of airway inflammation and hyperreactivity after in vivo transfer of CD4+CD25+ regulatory T cells is interleukin 10 dependent.

Interleukin-13: central mediator of allergic asthma

A large-scale, consortium-based genomewide association study of asthma

IL-33 mediates inflammatory responses in human lung tissue cells

Sequence variants affecting eosinophil numbers associate with asthma and myocardial infarction

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216-27.e1-6

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10.1016/J.JACI.2011.10.036

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1

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Andrew N. J. McKenzie

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2011-11-25T00:00:00Z

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51836385

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22119406

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3246069

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