Critical role of dipeptidyl peptidase I in neutrophil recruitment during the development of experimental abdominal aortic aneurysms. - Wikidata - wikimedia - TriplyDB

Critical role of dipeptidyl peptidase I in neutrophil recruitment during the development of experimental abdominal aortic aneurysms.

Critical role of dipeptidyl peptidase I in neutrophil recruitment during the development of experimental abdominal aortic aneurysms.

http://www.wikidata.org/entity/Q35635554

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Cystatin F is a cathepsin C-directed protease inhibitor regulated by proteolysis The Abdominal Aortic Aneurysm and Intraluminal Thrombus: Current Concepts of Development and Treatment Cystatin C deficiency promotes inflammation in angiotensin II-induced abdominal aortic aneurisms in atherosclerotic mice.Mast cell proteases as pharmacological targets Animal models of cardiovascular diseases Novel aspects of the pathogenesis of aneurysms of the abdominal aorta in humans.Leukocyte cathepsin C deficiency attenuates atherosclerotic lesion progression by selective tuning of innate and adaptive immune responses.Cysteinyl cathepsins and mast cell proteases in the pathogenesis and therapeutics of cardiovascular diseases.Anti-mouse properdin TSR 5/6 monoclonal antibodies block complement alternative pathway-dependent pathogenesis.Tenascin-C: a novel mediator of hepatic ischemia and reperfusion injury Antibody directs properdin-dependent activation of the complement alternative pathway in a mouse model of abdominal aortic aneurysm Neutrophil serine proteases promote IL-1β generation and injury in necrotizing crescentic glomerulonephritis.Cathepsin L activity is essential to elastase perfusion-induced abdominal aortic aneurysms in mice Cysteine Protease Cathepsins in Atherosclerosis and Abdominal Aortic Aneurysm.Functional alterations of myeloid cell subsets in hyperlipidaemia: relevance for atherosclerosis.Mast cells modulate the pathogenesis of elastase-induced abdominal aortic aneurysms in mice Novel mechanisms of abdominal aortic aneurysms Anti-complement activity of the Ixodes scapularis salivary protein Salp20.Novel mechanism of aortic aneurysm development in mice associated with smoking and leukocytes.Animal models of human granulocyte diseases.Asthma Associates With Human Abdominal Aortic Aneurysm and Rupture.Cysteine proteases: destruction ability versus immunomodulation capacity in immune cells.Biochemomechanics of intraluminal thrombus in abdominal aortic aneurysms.Dipeptidyl peptidase I-dependent neutrophil recruitment modulates the inflammatory response to Sendai virus infection.Role of complement cascade in abdominal aortic aneurysms Cysteine cathepsin S as an immunomodulatory target: present and future trends.Long-lasting tissue inflammatory processes trigger autoimmune responses to extracellular matrix molecules.Fibrinogen-specific antibody induces abdominal aortic aneurysm in mice through complement lectin pathway activation.DPPI may activate KLK4 during enamel formation.Critical role of mast cell chymase in mouse abdominal aortic aneurysm formation.Cathepsins mediate tumor metastasis.Complement-dependent neutrophil recruitment is critical for the development of elastase-induced abdominal aortic aneurysm Disinhibition of Cathepsin C Caused by Cystatin F Deficiency Aggravates the Demyelination in a Cuprizone Model Mast cell chymase and tryptase as targets for cardiovascular and metabolic diseases.Inhibitors of cathepsin C (dipeptidyl peptidase I).Diagnostic and therapeutic strategies for small abdominal aortic aneurysms.Neutrophils in local and systemic antibody-dependent inflammatory and anaphylactic reactions.Inhibitors of cathepsin G: a patent review (2005 to present).The pathophysiologic basis of abdominal aortic aneurysm progression: a critical appraisal.Cellular stress amplifies TLR3/4-induced CXCL1/2 gene transcription in mononuclear phagocytes via RIPK1.

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P2860

Critical role of dipeptidyl peptidase I in neutrophil recruitment during the development of experimental abdominal aortic aneurysms.

http://www.wikidata.org/entity/Q35635554

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Critical role of dipeptidyl pe ...... al abdominal aortic aneurysms.

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Christine T N Pham

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Dongli Mao

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Michel A Bartoli

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Monica B Pagano

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Pamela M Simmons

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Robert W Thompson

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Terri L Ennis

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P2860

Targeted gene disruption of matrix metalloproteinase-9 (gelatinase B) suppresses development of experimental abdominal aortic aneurysms

Detection and management of small aortic aneurysms

Neutrophil serine proteases: specific regulators of inflammation

Serine protease cathepsin G regulates adhesion-dependent neutrophil effector functions by modulating integrin clustering.

Abdominal aortic aneurysms: basic mechanisms and clinical implications.

Dipeptidyl peptidase I is required for the processing and activation of granzymes A and B in vivo.

Production and localization of 92-kilodalton gelatinase in abdominal aortic aneurysms. An elastolytic metalloproteinase expressed by aneurysm-infiltrating macrophages.

Matrix metalloproteinase-2: the forgotten enzyme in aneurysm pathogenesis.

Dipeptidyl peptidase I activates neutrophil-derived serine proteases and regulates the development of acute experimental arthritis.

Proinflammatory cytokines induce proteinase 3 as membrane-bound and secretory forms in human oral epithelial cells and antibodies to proteinase 3 activate the cells through protease-activated receptor-2.

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