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Reduced expression of Cx43 attenuates ventricular remodeling after myocardial infarction via impaired TGF-beta signalingHelix-loop-helix protein p8, a transcriptional regulator required for cardiomyocyte hypertrophy and cardiac fibroblast matrix metalloprotease inductionTranslating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulatesCell-cell contact activation of fibroblasts increases the expression of matrix metalloproteinasesDonepezil, anti-Alzheimer's disease drug, prevents cardiac rupture during acute phase of myocardial infarction in miceMatrix metalloproteinase-28 deletion amplifies inflammatory and extracellular matrix responses to cardiac agingToll-like receptor 4 modulates myocardial ischaemia-reperfusion injury: Role of matrix metalloproteinases.Intramyocardial fibroblast myocyte communication.A conceptual cellular interaction model of left ventricular remodelling post-MI: dynamic network with exit-entry competition strategyEffects of novel semiselective matrix metalloproteinase inhibitors on ex vivo cardiac structure-function.Zinc-binding groups modulate selective inhibition of MMPs.Macrophage roles following myocardial infarctionHemodialysis removes uremic toxins that alter the biological actions of endothelial cellsInvestigating chelating sulfonamides and their use in metalloproteinase inhibitorsArrhythmogenic properties of dismantling cadherin-mediated adhesion in murine hearts.Bone marrow-derived CXCR4+ cells mobilized by macrophage colony-stimulating factor participate in the reduction of infarct area and improvement of cardiac remodeling after myocardial infarction in miceCD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling.Matrix metalloproteinase (MMP)-9: a proximal biomarker for cardiac remodeling and a distal biomarker for inflammation.Matrix metalloproteinases and myocardial infarctionMMPs as therapeutic targets--still a viable option?Blockade of NF-kappaB using IkappaB alpha dominant-negative mice ameliorates cardiac hypertrophy in myotrophin-overexpressed transgenic mice.Effects of age on plasma matrix metalloproteinases (MMPs) and tissue inhibitor of metalloproteinases (TIMPs)Extracellular matrix turnover and signaling during cardiac remodeling following MI: causes and consequences.The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction.Renal studies provide an insight into cardiac extracellular matrix remodeling during health and disease.Intracardiac renin-angiotensin system and myocardial repair/remodeling following infarction.Dynamic interactions between the cellular components of the heart and the extracellular matrix.Surgical approaches to left ventricular reconstruction: a matter of perspective.Intracellular signaling of cardiac fibroblasts.Loss or inhibition of uPA or MMP-9 attenuates LV remodeling and dysfunction after acute pressure overload in mice.Management of cardiac fibrosis in diabetic rats; the role of peroxisome proliferator activated receptor gamma (PPAR-gamma) and calcium channel blockers (CCBs)Proteomic analysis identifies in vivo candidate matrix metalloproteinase-9 substrates in the left ventricle post-myocardial infarction.Methanethiol Binding Strengths and Deprotonation Energies in Zn(II)-Imidazole Complexes from M05-2X and MP2 Theories: Coordination Number and Geometry Influences Relevant to Zinc Enzymes.Angiotensinase C mRNA and Protein Downregulations Are Involved in Ethanol-Deteriorated Left Ventricular Systolic Dysfunction in Spontaneously Hypertensive Rats.The giant danio (D. aequipinnatus) as a model of cardiac remodeling and regenerationEffects of doxycycline on cx43 distribution and cardiac arrhythmia susceptibility of rats after myocardial infarction.ACE inhibitors to block MMP-9 activity: new functions for old inhibitors.A murine model of alcoholic cardiomyopathy: a role for zinc and metallothionein in fibrosis.Cardiac matrix metalloproteinase-2 expression independently induces marked ventricular remodeling and systolic dysfunction.Identifying the regulative role of NF-κB binding sites within promoter region of human matrix metalloproteinase 9 (mmp-9) by TNF-α induction.
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
MMP induction and inhibition in myocardial infarction.
@ast
MMP induction and inhibition in myocardial infarction.
@en
type
label
MMP induction and inhibition in myocardial infarction.
@ast
MMP induction and inhibition in myocardial infarction.
@en
prefLabel
MMP induction and inhibition in myocardial infarction.
@ast
MMP induction and inhibition in myocardial infarction.
@en
P1476
MMP induction and inhibition in myocardial infarction.
@en
P2093
Merry L Lindsey
P356
10.1023/B:HREV.0000011390.44039.B7
P577
2004-01-01T00:00:00Z
P6179
1006725134