STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophy. - Wikidata - wikimedia - TriplyDB

STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophy.

STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophy.

http://www.wikidata.org/entity/Q35637186

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STIM and Orai1 Variants in Store-Operated Calcium Entry Store-operated Ca2+ entry in muscle physiology and diseases Store-operated calcium entry and diabetic complications miR-185 plays an anti-hypertrophic role in the heart via multiple targets in the calcium-signaling pathways Gastrodin Inhibits Store-Operated Ca2+ Entry and Alleviates Cardiac Hypertrophy.Compensatory and decompensatory alterations in cardiomyocyte Ca2+ dynamics in hearts with diastolic dysfunction following aortic banding.Protein O-GlcNAcylation and cardiovascular (patho)physiology.Altered myocardial calcium cycling and energetics in heart failure--a rational approach for disease treatment.Identification of Stim1 as a candidate gene for exaggerated sympathetic response to stress in the stroke-prone spontaneously hypertensive rat.Polycystin-1 Is a Cardiomyocyte Mechanosensor That Governs L-Type Ca2+ Channel Protein Stability STIM1 enhances SR Ca2+ content through binding phospholamban in rat ventricular myocytes.STIM1-Ca2+ signaling modulates automaticity of the mouse sinoatrial node.STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy Regression of pathological cardiac hypertrophy: signaling pathways and therapeutic targets.Store-operated Ca2+ entry supports contractile function in hearts of hibernators.Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytes Targeted STIM deletion impairs calcium homeostasis, NFAT activation, and growth of smooth muscle.Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling.Altered sarcoplasmic reticulum calcium cycling--targets for heart failure therapy.STIM1-dependent Ca(2+) microdomains are required for myofilament remodeling and signaling in the heart.Transient Receptor Potential Canonical (TRPC)/Orai1-dependent Store-operated Ca2+ Channels: NEW TARGETS OF ALDOSTERONE IN CARDIOMYOCYTES.STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function.ROS and SOCE: recent advances and controversies in the regulation of STIM and Orai.STIM/Orai signalling complexes in vascular smooth muscle.Roles for the sarco-/endoplasmic reticulum in cardiac myocyte contraction, protein synthesis, and protein quality control.The other side of cardiac Ca(2+) signaling: transcriptional control.What role for store-operated Ca²⁺ entry in muscle?The regulation of STIM1 by phosphorylation.Cardioprotection in ischaemia-reperfusion injury: novel mechanisms and clinical translation.Changing calcium: CRAC channel (STIM and Orai) expression, splicing, and posttranslational modifiers.Calcium and Excitation-Contraction Coupling in the Heart The Impact of Vitamin D3 Supplementation on Mechanisms of Cell Calcium Signaling in Chronic Kidney Disease.Essential Role of Smooth Muscle STIM1 in Hypertension and Cardiovascular Dysfunction.Long-term administration of pyridostigmine attenuates pressure overload-induced cardiac hypertrophy by inhibiting calcineurin signalling.Emergence of Orai3 activity during cardiac hypertrophy.Ca(2+) signaling in T-cell subsets with a focus on the role of cav1 channels: possible implications in therapeutics Stromal interaction molecule 1 haploinsufficiency causes maladaptive response to pressure overload.Suppression of Stim1 reduced intracellular calcium concentration and attenuated hypoxia/reoxygenation induced apoptosis in H9C2 cells.A proteolytic fragment of histone deacetylase 4 protects the heart from failure by regulating the hexosamine biosynthetic pathway.

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P2860

STIM1-dependent store-operated Ca²⁺ entry is required for pathological cardiac hypertrophy.

http://www.wikidata.org/entity/Q35637186

description

2011 nî lūn-bûn

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2011年の論文

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name

STIM1-dependent store-operated ...... hological cardiac hypertrophy.

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STIM1-dependent store-operated ...... hological cardiac hypertrophy.

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STIM1-dependent store-operated ...... hological cardiac hypertrophy.

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STIM1-dependent store-operated ...... hological cardiac hypertrophy.

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STIM1-dependent store-operated ...... hological cardiac hypertrophy.

@ast

STIM1-dependent store-operated ...... hological cardiac hypertrophy.

@en

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J.YJMCC.2011.11.003

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Journal of Molecular and Cellular Cardiology

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STIM1-dependent store-operated ...... hological cardiac hypertrophy.

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Andrey Rakalin

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Berdymammet Hojayev

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Beverly A Rothermel

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Joseph A Hill

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Nan Jiang

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Samvit Tandan

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Thomas G Gillette

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Xiang Luo

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Zhao V Wang

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P2860

STIM1 is a Ca2+ sensor that activates CRAC channels and migrates from the Ca2+ store to the plasma membrane

STIM1 has a plasma membrane role in the activation of store-operated Ca(2+) channels

Genome-wide RNAi screen of Ca(2+) influx identifies genes that regulate Ca(2+) release-activated Ca(2+) channel activity

STIM1, an essential and conserved component of store-operated Ca2+ channel function

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Calcium fluxes involved in control of cardiac myocyte contraction

STIM1 carboxyl-terminus activates native SOC, I(crac) and TRPC1 channels

A small molecular activator of cardiac hypertrophy uncovered in a chemical screen for modifiers of the calcineurin signaling pathway

Aberrant localization of intracellular organelles, Ca2+ signaling, and exocytosis in Mist1 null mice

Heart disease and stroke statistics--2011 update: a report from the American Heart Association

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2011-11-13T00:00:00Z

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51821691

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22108056

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3247164

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