Mouse mutants reveal that putative protein interaction sites in the p53 proline-rich domain are dispensable for tumor suppression
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Prolyl isomerase Pin1 as a molecular switch to determine the fate of phosphoproteinsProlyl isomerase Pin1 in cancerDeacetylation of p53 induces autophagy by suppressing Bmf expressionp53 Amino-terminus region (1-125) stabilizes and restores heat denatured p53 wild phenotype.Posttranslational modification of p53: cooperative integrators of functionp53 at a glance.In vivo analysis of p53 tumor suppressor function using genetically engineered mouse modelsThe Naked Mole Rat Genome Resource: facilitating analyses of cancer and longevity-related adaptations.A genetic variant of p53 restricts the mucous secretory phenotype by regulating SPDEF and Bcl-2 expression.The relationship of TP53 R72P polymorphism to disease outcome and TP53 mutation in myelodysplastic syndromesPeptidyl Prolyl Isomerase PIN1 Directly Binds to and Stabilizes Hypoxia-Inducible Factor-1α.MDM2 and MDM4: p53 regulators as targets in anticancer therapy.Structure of tumor suppressor p53 and its intrinsically disordered N-terminal transactivation domain.p53 polymorphisms: cancer implications.The tumor suppressor p53: from structures to drug discovery.Peptidyl-prolyl cis-trans isomerase Pin1 in ageing, cancer and Alzheimer disease.Illuminating p53 function in cancer with genetically engineered mouse models.Insights into wild-type and mutant p53 functions provided by genetically engineered mice.Genetic Modifiers of the p53 Pathway.Does control of mutant p53 by Mdm2 complicate cancer therapy?Activation of p53 following ionizing radiation, but not other stressors, is dependent on the proline-rich domain (PRD).Chromatin remodeling protein MORC2 promotes breast cancer invasion and metastasis through a PRD domain-mediated interaction with CTNND1.The proline rich domain of p53 is dispensable for MGMT-dependent DNA repair and cell survival following alkylation damage.
P2860
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P2860
Mouse mutants reveal that putative protein interaction sites in the p53 proline-rich domain are dispensable for tumor suppression
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
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2006年论文
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2006年论文
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name
Mouse mutants reveal that puta ...... pensable for tumor suppression
@ast
Mouse mutants reveal that puta ...... pensable for tumor suppression
@en
type
label
Mouse mutants reveal that puta ...... pensable for tumor suppression
@ast
Mouse mutants reveal that puta ...... pensable for tumor suppression
@en
prefLabel
Mouse mutants reveal that puta ...... pensable for tumor suppression
@ast
Mouse mutants reveal that puta ...... pensable for tumor suppression
@en
P2093
P2860
P356
P1476
Mouse mutants reveal that puta ...... pensable for tumor suppression
@en
P2093
Chung-Wen Liu
Crystal J Lee
Franck Toledo
Geoffrey M Wahl
Kurt A Krummel
Luo-Wei Rodewald
P2860
P304
P356
10.1128/MCB.00999-06
P407
P577
2006-12-11T00:00:00Z