Nicotinic cholinergic mechanisms causing elevated dopamine release and abnormal locomotor behavior. - Wikidata - wikimedia - TriplyDB

Nicotinic cholinergic mechanisms causing elevated dopamine release and abnormal locomotor behavior.

Nicotinic cholinergic mechanisms causing elevated dopamine release and abnormal locomotor behavior.

http://www.wikidata.org/entity/Q35645412

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Crucial roles of the CHRNB3-CHRNA6 gene cluster on chromosome 8 in nicotine dependence: update and subjects for future research Insights into the neurobiology of the nicotinic cholinergic system and nicotine addiction from mice expressing nicotinic receptors harboring gain-of-function mutations Nicotine-Cadmium Interaction Alters Exploratory Motor Function and Increased Anxiety in Adult Male Mice.Functional upregulation of Ca(2+)-activated K(+) channels in the development of substantia nigra dopamine neurons.Nicotine and ethanol cooperate to enhance ventral tegmental area AMPA receptor function via α6-containing nicotinic receptors Nicotinic acetylcholine receptors containing α6 subunits contribute to alcohol reward-related behaviours.α6-Containing nicotinic acetylcholine receptors in midbrain dopamine neurons are poised to govern dopamine-mediated behaviors and synaptic plasticity α6* nicotinic acetylcholine receptor expression and function in a visual salience circuit.Nicotine Dependence Reveals Distinct Responses from Neurons and Their Resident Nicotinic Receptors in Medial Habenula.Local application of drugs to study nicotinic acetylcholine receptor function in mouse brain slices.A role for α4(non-α6)* nicotinic acetylcholine receptors in motor behavior Cortical Control of Striatal Dopamine Transmission via Striatal Cholinergic Interneurons α4α6β2* nicotinic acetylcholine receptor activation on ventral tegmental area dopamine neurons is sufficient to stimulate a depolarizing conductance and enhance surface AMPA receptor function Enhanced synthesis and release of dopamine in transgenic mice with gain-of-function α6* nAChRs.Nicotine Addiction and Psychiatric Disorders.Altered nicotine reward-associated behavior following α4 nAChR subunit deletion in ventral midbrain.B6eGFPChAT mice overexpressing the vesicular acetylcholine transporter exhibit spontaneous hypoactivity and enhanced exploration in novel environments.Deletion of lynx1 reduces the function of α6* nicotinic receptors.α6β2 nicotinic acetylcholine receptors influence locomotor activity and ethanol consumption.α6β2 subunit containing nicotinic acetylcholine receptors exert opposing actions on rapid dopamine signaling in the nucleus accumbens of rats with high-versus low-response to novelty.

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Nicotinic cholinergic mechanisms causing elevated dopamine release and abnormal locomotor behavior.

http://www.wikidata.org/entity/Q35645412

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Nicotinic cholinergic mechanis ...... d abnormal locomotor behavior.

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Nicotinic cholinergic mechanis ...... d abnormal locomotor behavior.

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Nicotinic cholinergic mechanis ...... d abnormal locomotor behavior.

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Nicotinic cholinergic mechanis ...... d abnormal locomotor behavior.

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P2860

Hyperlocomotion and indifference to cocaine and amphetamine in mice lacking the dopamine transporter

In vivo activation of midbrain dopamine neurons via sensitized, high-affinity alpha 6 nicotinic acetylcholine receptors

Crucial role of alpha4 and alpha6 nicotinic acetylcholine receptor subunits from ventral tegmental area in systemic nicotine self-administration

Dopamine reward circuitry: two projection systems from the ventral midbrain to the nucleus accumbens-olfactory tubercle complex.

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

A new alpha-conotoxin which targets alpha3beta2 nicotinic acetylcholine receptors

Analyzing real-time PCR data by the comparative C(T) method

Subunit composition of functional nicotinic receptors in dopaminergic neurons investigated with knock-out mice

Distribution and pharmacology of alpha 6-containing nicotinic acetylcholine receptors analyzed with mutant mice

Point mutant mice with hypersensitive alpha 4 nicotinic receptors show dopaminergic deficits and increased anxiety.

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